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1. A nationwide questionnaire study evaluated kidney injury associated with Beni-koji tablets in Japan

Author

Ono, Masami, Yonekura, Yuriko, Nishizawa, Yuki, Azuma, Masahiro, Aoki, Ryousuke, Inagaki, Koji, Nagawa, Daiki, Miyoshi, Ken-ichi, Kamikawa, Yasutaka, Kasagi, Yuri, Kishi, Seiji, Motegi, Shinsuke, Suzuki, Hitoshi, Okada, Shioko, Nomura, Kazufumi, Ito, Jun, Itoh, Tomoaki, Saka, Yosuke, Ebihara, Itaru, Hamano, Takayuki, Ogi, Makoto, Imasawa, Toshiyuki, Shibuya, Ken, Yoshikawa, Kazuhiro, Ushimaru, Shu, Fujii, Hideki, Sato, Koji, Hayashi, Kaori, Habuka, Masato, Tojo, Akihiro, Katori, Hideyuki, Torii, Akiko, Miyazaki, Reina, Kitashoji, Emi, Tokura, Takehiko, Nemoto, Yoshikazu, Baba, Masahito, Kudo, Risshi, Saito, Atsushi, Fujimura, Ryuta, Araki, Takashi, Yoshida, Hiraku, Miyazaki, Takanori, Osaki, Yosuke, Nishio, Saori, Utsunomiya, Yasunori, Nakanishi, Shohei, Doi, Toshiki, Sato, Mitsuhiro, Miyabe, Yoei, Yoshida, Hideaki, Sofue, Tadashi, Izumi, Masaaki, Hayashi, Matsuhiko, Aoyama, Togo, Osaki, Keisuke, Iwatani, Ryuuji, Iehara, Noriyuki, Mikoto, Matsui, Tsukamoto, Tatsuo, Nishitani, Yoshiharu, Konishi, Yoshio, Shihomi, Maeda, Kawano, Yuki, Maruyama, Keisuke, Nakamori, Aya, Ishikawa, Eiji, Osawa, Norihisa, Komeda, Masami, Hasegawa, Ritsuko, Shiratori-Aso, Satoka, Takahashi, Sachiko, Deguchi, Hiroyuki, Aoki, Toshiyuki, Kobayashi, Shuzo, Ichikawa, Daisuke, Yokoyama, Tomoko, Nagae, Tetsuya, Sakai, Takayuki, Shibata, Shigeru, Ino, Jun, Matsuura, Motokazu, China, Rieko, Okada, Tomonari, Ohara, Maiko, Kinuno, Hiroyuki, Toda, Takayuki, Nukui, Ikuo, Oshikawa, Jin, Shinzawa, Maki, Matsui, Isao, Doi, Yohei, Matsumoto, Ayumi, Takahashi, Atsushi, Nangaku, Masaomi, and Isaka, Yoshitaka

Published
2024
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2. Interventional nephrology: current status and clinical impact in Japan

Author

Ikeda, Masato, Terawaki, Hiroyuki, Kanda, Eiichiro, Furuya, Maiko, Tanno, Yudo, Nakao, Masatsugu, Maruyama, Yukio, Maeda, Masutaka, Higuchi, Chieko, Sakurada, Tsutomu, Kaneko, Tomohiro, Io, Hiroaki, Hashimoto, Koji, Ueda, Atsushi, Hirano, Keita, Washida, Naoki, Yoshida, Hiraku, Yoshikawa, Kazuhiro, Taniyama, Yoshihiro, Harada, Kenji, Matsuo, Nanae, Okido, Ichiro, and Yokoo, Takashi

Published
2018
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5. A nationwide questionnaire study evaluated kidney injury associated with Beni-koji tablets in Japan

Author

Shinzawa, Maki, Matsui, Isao, Doi, Yohei, Matsumoto, Ayumi, Takahashi, Atsushi, Nangaku, Masaomi, Isaka, Yoshitaka, Ono, Masami, Yonekura, Yuriko, Nishizawa, Yuki, Azuma, Masahiro, Aoki, Ryousuke, Inagaki, Koji, Nagawa, Daiki, Miyoshi, Ken-ichi, Kamikawa, Yasutaka, Kasagi, Yuri, Kishi, Seiji, Motegi, Shinsuke, Suzuki, Hitoshi, Okada, Shioko, Nomura, Kazufumi, Ito, Jun, Itoh, Tomoaki, Saka, Yosuke, Ebihara, Itaru, Hamano, Takayuki, Ogi, Makoto, Imasawa, Toshiyuki, Shibuya, Ken, Yoshikawa, Kazuhiro, Ushimaru, Shu, Fujii, Hideki, Sato, Koji, Hayashi, Kaori, Habuka, Masato, Tojo, Akihiro, Katori, Hideyuki, Torii, Akiko, Miyazaki, Reina, Kitashoji, Emi, Tokura, Takehiko, Nemoto, Yoshikazu, Baba, Masahito, Kudo, Risshi, Saito, Atsushi, Fujimura, Ryuta, Araki, Takashi, Yoshida, Hiraku, Miyazaki, Takanori, Osaki, Yosuke, Nishio, Saori, Utsunomiya, Yasunori, Nakanishi, Shohei, Doi, Toshiki, Sato, Mitsuhiro, Miyabe, Yoei, Yoshida, Hideaki, Sofue, Tadashi, Izumi, Masaaki, Hayashi, Matsuhiko, Aoyama, Togo, Osaki, Keisuke, Iwatani, Ryuuji, Iehara, Noriyuki, Mikoto, Matsui, Tsukamoto, Tatsuo, Nishitani, Yoshiharu, Konishi, Yoshio, Shihomi, Maeda, Kawano, Yuki, Maruyama, Keisuke, Nakamori, Aya, Ishikawa, Eiji, Osawa, Norihisa, Komeda, Masami, Hasegawa, Ritsuko, Shiratori-Aso, Satoka, Takahashi, Sachiko, Deguchi, Hiroyuki, Aoki, Toshiyuki, Kobayashi, Shuzo, Ichikawa, Daisuke, Yokoyama, Tomoko, Nagae, Tetsuya, Sakai, Takayuki, Shibata, Shigeru, Ino, Jun, Matsuura, Motokazu, China, Rieko, Okada, Tomonari, Ohara, Maiko, Kinuno, Hiroyuki, Toda, Takayuki, Nukui, Ikuo, and Oshikawa, Jin

Abstract

Red yeast rice, traditionally used in Asian cuisine and increasingly marketed as a dietary supplement for cholesterol management, has recently been linked to kidney dysfunction in Japan. In late 2023 to early 2024, multiple cases involving specific Beni-koji (red yeast rice) tablets from three different Beni-koji preparations, prompted a safety reevaluation. Although citrinin, a known nephrotoxin of red yeast rice, was not produced by the implicated strains, new safety concerns emerged. Here, we aimed to investigate the clinical, laboratory, and pathological features of affected patients with a two-phase nationwide survey of Japanese nephrologists. The initial survey captured clinical presentations, while the follow-up survey tracked changes in kidney function and gathered pathological data. Statistical analyses included trend assessments across estimated glomerular filtration rate (eGFR) categories and mixed-effects models for eGFR trajectories. Of 192 patients, 94.1% presented with low eGFR (under 60 ml/min/1.73m2). Laboratory findings revealed characteristics of Fanconi syndrome, including hypokalemia, hypophosphatemia, hypouricemia, glycosuria, and metabolic acidosis. Creatine kinase levels were not elevated suggesting no rhabdomyolysis related kidney injury. Kidney biopsies showed predominant tubulointerstitial changes, with 50% exhibiting tubulointerstitial nephritis and 32% showing tubular necrosis. Glomerular changes were less prominent. Following product discontinuation and treatment, Fanconi syndrome-related parameters improved significantly. However, 87% of patients still had eGFR under 60 ml/min/1.73m2at the last observation. Our findings underscore the need for long-term follow-up of affected individuals and highlight the importance of rigorous safety evaluations for dietary supplements. Further research is necessary to establish definitive causal relationships and long-term outcomes.

Published
2025
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11. Suzaku Wide-band All-sky Monitor – 10 Years history

Author

Ohno, Masanori, primary, Fukazawa, Yasushi, additional, Kawano, Takafumi, additional, Yamaoka, Kazutaka, additional, Tashiro, Makoto S., additional, Yasuda, Tetsuya, additional, Enomoto, Junichi, additional, Matsuoka, Shunsuke, additional, Nagayoshi, Tsutomu, additional, Nakaya, Souhei, additional, Takeda, Sawako, additional, Terada, Yukikatsu, additional, Yabe, Seiya, additional, Hurley, Kevin, additional, Krimm, Hans A., additional, Lien, Amy Y., additional, Ohmori, Norisuke, additional, Kinoshita, Ryuuji, additional, Nishioka, Yusuke, additional, Yamauchi, Makoto, additional, Yoshida, Hiraku, additional, Sugita, Satoshi, additional, Urata, Yuji, additional, Hanabata, Yoshitaka, additional, Iwakiri, Wataru, additional, Kokubun, Motohide, additional, Makishima, Kazuo, additional, Nakagawa, Yujin, additional, Nakazawa, Kazuhiro, additional, and Sakamoto, Takanori, additional

Published
2019
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12. Suzaku Wide-band All-sky Monitor (WAM) observations of GRBs and SGRs

Author

Hurley, Kevin, Krimm, Hans A., Lien, Amy Y., Yoshida, Hiraku, Yamaoka, Kazutaka, Ohno, Masanori, Tashiro, Makoto S., Ohmori, Norisuke, Sugita, Satoshi, Urata, Yuji, Yasuda, Tetsuya, Enomoto, Junichi, Fujinuma, Takeshi, Fukazawa, Yasushi, Hanabata, Yoshitaka, Iwakiri, Wataru, Kawano, Takafumi, Kinoshita, Ryuuji, Kokubun, Motohide, Makishima, Kazuo, Matsuoka, Shunsuke, Nagayoshi, Tsutomu, Nakagawa, Yujin, Nakaya, Souhei, Nakazawa, Kazuhiro, Nishioka, Yusuke, Sakamoto, Takanori, Takahashi, Tadayuki, Takeda, Sawako, Terada, Yukikatsu, Yabe, Seiya, and Yamauchi, Makoto

Subjects
Physics, instrumentation: detectors, 010308 nuclear & particles physics, media_common.quotation_subject, gamma-ray burst: general, Astronomy, Astronomy and Astrophysics, methods: data analysis, 01 natural sciences, Space and Planetary Science, Sky, 0103 physical sciences, Wide band, methods: observational, 010303 astronomy & astrophysics, media_common
Abstract

著者人数: 32名, Accepted: 2017-03-14, 資料番号: SA1170064000

Published
2017
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13. Interventional nephrology: current status and clinical impact in Japan

Author

Ikeda, Masato, primary, Terawaki, Hiroyuki, additional, Kanda, Eiichiro, additional, Furuya, Maiko, additional, Tanno, Yudo, additional, Nakao, Masatsugu, additional, Maruyama, Yukio, additional, Maeda, Masutaka, additional, Higuchi, Chieko, additional, Sakurada, Tsutomu, additional, Kaneko, Tomohiro, additional, Io, Hiroaki, additional, Hashimoto, Koji, additional, Ueda, Atsushi, additional, Hirano, Keita, additional, Washida, Naoki, additional, Yoshida, Hiraku, additional, Yoshikawa, Kazuhiro, additional, Taniyama, Yoshihiro, additional, Harada, Kenji, additional, Matsuo, Nanae, additional, Okido, Ichiro, additional, and Yokoo, Takashi, additional

Published
2017
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14. Suzaku Wide-band All-sky Monitor (WAM) observations of GRBs and SGRs

Author

Yamaoka, Kazutaka, primary, Ohno, Masanori, additional, Tashiro, Makoto S., additional, Hurley, Kevin, additional, Krimm, Hans A., additional, Lien, Amy Y., additional, Ohmori, Norisuke, additional, Sugita, Satoshi, additional, Urata, Yuji, additional, Yasuda, Tetsuya, additional, Enomoto, Junichi, additional, Fujinuma, Takeshi, additional, Fukazawa, Yasushi, additional, Hanabata, Yoshitaka, additional, Iwakiri, Wataru, additional, Kawano, Takafumi, additional, Kinoshita, Ryuuji, additional, Kokubun, Motohide, additional, Makishima, Kazuo, additional, Matsuoka, Shunsuke, additional, Nagayoshi, Tsutomu, additional, Nakagawa, Yujin, additional, Nakaya, Souhei, additional, Nakazawa, Kazuhiro, additional, Nishioka, Yusuke, additional, Sakamoto, Takanori, additional, Takahashi, Tadayuki, additional, Takeda, Sawako, additional, Terada, Yukikatsu, additional, Yabe, Seiya, additional, Yamauchi, Makoto, additional, and Yoshida, Hiraku, additional

Published
2017
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15. Suzaku Wide-band All-sky Monitor (WAM) observations of GRBs and SGRs

Author

Yamaoka, Kazutaka, Ohno, Masanori, Tashiro, Makoto S., Hurley, Kevin, Krimm, Hans A., Lien, Amy Y., Ohmori, Norisuke, Sugita, Satoshi, Urata, Yuji, Yasuda, Tetsuya, Enomoto, Junichi, Fujinuma, Takeshi, Fukazawa, Yasushi, Hanabata, Yoshitaka, Iwakiri, Wataru, Kawano, Takafumi, Kinoshita, Ryuuji, Kokubun, Motohide, Makishima, Kazuo, Matsuoka, Shunsuke, Nagayoshi, Tsutomu, Nakagawa, Yujin, Nakaya, Souhei, Nakazawa, Kazuhiro, Nishioka, Yusuke, Sakamoto, Takanori, Takahashi, Tadayuki, Takeda, Sawako, Terada, Yukikatsu, Yabe, Seiya, Yamauchi, Makoto, Yoshida, Hiraku, 山岡, 和貴, 大野, 雅功, 田代, 信, 大森, 法輔, 杉田, 聡司, 浦田, 裕次, 安田, 哲也, 榎本, 淳一, 藤沼, 洸, 深沢, 泰司, 花畑, 義隆, 岩切, 渉, 河野, 貴文, 木下, 龍治, 国分, 紀秀, 牧島, 一夫, 松岡, 俊介, 永吉, 勤, 中川, 友進, 中谷, 創平, 中澤, 知洋, 西岡, 祐介, 坂本, 貴紀, 高橋, 忠幸, 武田, 佐和子, 寺田, 幸功, 矢部, 聖也, 山内, 誠, Yamaoka, Kazutaka, Ohno, Masanori, Tashiro, Makoto S., Hurley, Kevin, Krimm, Hans A., Lien, Amy Y., Ohmori, Norisuke, Sugita, Satoshi, Urata, Yuji, Yasuda, Tetsuya, Enomoto, Junichi, Fujinuma, Takeshi, Fukazawa, Yasushi, Hanabata, Yoshitaka, Iwakiri, Wataru, Kawano, Takafumi, Kinoshita, Ryuuji, Kokubun, Motohide, Makishima, Kazuo, Matsuoka, Shunsuke, Nagayoshi, Tsutomu, Nakagawa, Yujin, Nakaya, Souhei, Nakazawa, Kazuhiro, Nishioka, Yusuke, Sakamoto, Takanori, Takahashi, Tadayuki, Takeda, Sawako, Terada, Yukikatsu, Yabe, Seiya, Yamauchi, Makoto, Yoshida, Hiraku, 山岡, 和貴, 大野, 雅功, 田代, 信, 大森, 法輔, 杉田, 聡司, 浦田, 裕次, 安田, 哲也, 榎本, 淳一, 藤沼, 洸, 深沢, 泰司, 花畑, 義隆, 岩切, 渉, 河野, 貴文, 木下, 龍治, 国分, 紀秀, 牧島, 一夫, 松岡, 俊介, 永吉, 勤, 中川, 友進, 中谷, 創平, 中澤, 知洋, 西岡, 祐介, 坂本, 貴紀, 高橋, 忠幸, 武田, 佐和子, 寺田, 幸功, 矢部, 聖也, and 山内, 誠

Abstract

著者人数: 32名, Accepted: 2017-03-14

Published
2017

16. A Case of Concurrent MPO-/PR3-Negative ANCA-Associated Glomerulonephritis and Membranous Glomerulopathy

Author

Nakada, Yasuyuki, Tsuboi, Nobuo, Takahashi, Yasuto, Yoshida, Hiraku, Hara, Yoriko, Okonogi, Hideo, Kawamura, Tetsuya, Arimura, Yoshihiro, and Yokoo, Takashi

Subjects
Article Subject, cardiovascular diseases, urologic and male genital diseases
Abstract

We report a case in which antineutrophil cytoplasmic antibody- (ANCA-) associated glomerulonephritis and membranous glomerulopathy (MGN) were detected concurrently. The patient showed rapidly progressive renal deterioration. A renal biopsy showed crescentic glomerulonephritis, together with marked thickening and spike and bubbling formations in the glomerular basement membranes. Indirect immunofluorescence examination of the patient’s neutrophils showed a perinuclear pattern. Enzyme-linked immunosorbent assays revealed that the ANCA in this case did not target myeloperoxidase (MPO) or proteinase 3 (PR3) but bactericidal-/permeability-increasing protein, elastase, and lysosome. The relationship between these two etiologically distinct entities, MPO-/PR3-negative ANCA-associated glomerulonephritis and MGN, remains unclear.

Published
2015
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22. Successful Treatment of Adult IgA Nephropathy with Nephrotic-Level Proteinuria by Combination Therapy Including Long-Term Coadministration of Mizoribine

Author

Okonogi, Hideo, primary, Kawamura, Tetsuya, additional, Matsuo, Nanae, additional, Takahashi, Yasuto, additional, Yamamoto, Izumi, additional, Yoshida, Hiraku, additional, Koike, Kentaro, additional, Yokoo, Takashi, additional, Joh, Kensuke, additional, Utsunomiya, Yasunori, additional, Hanaoka, Kazushige, additional, and Hosoya, Tatsuo, additional

Published
2012
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27. Most Patients With Coronary Artery Calcification Have No Coronary Artery Stenosis and Hyperphosphatemia Should be Important in Reevaluating the K/DOQI Guidelines

Author

Yokoyama, Keitaro, primary, Nishioka, Makiko, additional, Sakuma, Toru, additional, Yoshida, Satoru, additional, Iida, Rinako, additional, Yoshida, Hiraku, additional, Hara, Shino, additional, Ohkido, Ichiro, additional, Katoh, Naohiko, additional, Yamamoto, Hiroyasu, additional, Shigematsu, Takashi, additional, Nakayama, Massaki, additional, Sherif, Ali Mohamed, additional, Fukuda, Kunihiko, additional, and Hosoya, Tatsuo, additional

Published
2006
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30. Preventive Strategies for Vascular Calcification in Patients with Chronic Kidney Disease.

Author

Shigematsu T, Sonou T, Ohya M, Yokoyama K, Yoshida H, Yokoo T, Okuda K, Masumoto AR, Iwashita Y, Iseki K, Kunimoto S, and Yamano Y

Subjects
Atherosclerosis, Humans, Hyperparathyroidism, Secondary drug therapy, Hyperparathyroidism, Secondary surgery, Hyperphosphatemia drug therapy, Hyperphosphatemia therapy, Renal Dialysis, Vascular Calcification etiology, Renal Insufficiency, Chronic complications, Vascular Calcification prevention & control
Abstract

Background: Vascular calcification is significant because of the close association between the degree of vascular calcification and cardiovascular mortality in chronic kidney disease (CKD) patients., Summary: There are 2 types of vascular calcification in CKD patients. One is endothelial vascular calcification, a common type of vascular calcification. Another is medial vascular calcification, a specific type that is common in CKD patients. The former is mainly associated with atherosclerosis due to hyperlipidemia, especially hypercholesterolemia. The latter CKD-specific type is called Moenckeberg's arteriosclerosis. A known risk factor for this type of vascular calcification is hyperphosphatemia. In this review article, we mainly discuss a preventive strategy for Moenckeberg type vascular calcification in CKD, primarily involving the treatment of hyperphosphatemia. Several possible modalities are considered. However, at present, dietary restriction of phosphate is not recommended so as to avoid malnutrition in CKD patients. The first consideration is the enhancement of phosphate removal by renal replacement therapy in dialysis patients. Various phosphate binder therapies can be beneficial and effective. Surgical and pharmacological parathyroidectomies are also useful for treating secondary hyperparathyroidism. Good quality bone provides a good pool of calcium and phosphate. Thus, bone protection is another option for preventing vascular calcification. Several therapeutic agents have been developed to manage osteoporosis. These trial agents may be reasonably effective in impeding the progression of vascular calcification in CKD patients. Key Messages: We should make full use of several modalities so as to completely prevent vascular calcification., (© 2017 S. Karger AG, Basel.)

Published
2017
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31. [Mechanism, imaging technique and therapy for vascular calcification in patients of chronic kidney disease].

Author

Yoshida H and Yokoyama K

Subjects
Humans, Vascular Diseases diagnosis, Vascular Diseases drug therapy, Calcinosis, Kidney Failure, Chronic complications, Vascular Diseases etiology
Abstract

Vascular calcification has been reported to influence mortality and complications of cardiovascular diseases in patients with chronic kidney disease. Once vascular calcification was thought to the result from passive precipitation of calcium and phosphate, it now appears that end result of phenotypic change of vascular smooth muscle cells (VSMC) into osteoblast-like cells. A variety of imaging technique are available to visualize vascular calcification, including X-ray, vascular ultrasound, electron beam computed tomography (EBCT) and multidetector-row computed tomography (MDCT) . Especially, MDCT with contrast medium that can detect not only coronary calcification but also stenosis is useful and noninvasive methods for screening of coronary artery disease. Through greater understanding of both the mechanism and clinical consequence of vascular calcification, future therapeutic strategies may be more effectively designed and applied.

Published
2007
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32. [Renal osteodystrophy (ROD)].

Author

Yoshida H and Shigematsu T

Subjects
Aluminum isolation & purification, Calcitriol administration & dosage, Ethanol administration & dosage, Glomerular Filtration Rate, Humans, Hypocalcemia etiology, Injections, Intralesional, Parathyroid Hormone metabolism, Phosphorus blood, Phosphorus isolation & purification, Vitamin D Deficiency etiology, Chronic Kidney Disease-Mineral and Bone Disorder classification, Chronic Kidney Disease-Mineral and Bone Disorder diagnosis, Chronic Kidney Disease-Mineral and Bone Disorder etiology, Chronic Kidney Disease-Mineral and Bone Disorder therapy
Published
2006

33. [Molecular biology in regulation of kidney functions: PTH (parathyroid hormone)].

Author

Iida R, Yoshida H, Hara S, Ookido I, and Shigematsu T

Subjects
25-Hydroxyvitamin D3 1-alpha-Hydroxylase metabolism, Animals, Calcium metabolism, Calcium physiology, Carrier Proteins physiology, Homeodomain Proteins physiology, Humans, Kidney Tubules metabolism, Morphogenesis genetics, Parathyroid Glands embryology, Phosphorus metabolism, Polycomb Repressive Complex 1, Receptor, Parathyroid Hormone, Type 1 physiology, Signal Transduction physiology, Kidney physiology, Parathyroid Hormone biosynthesis, Parathyroid Hormone metabolism, Parathyroid Hormone physiology
Published
2006

34. [The role of phosphatonin in phosphate metabolism].

Author

Hara S, Iida R, Yoshida H, Ohkido I, and Shigematsu T

Subjects
Fibroblast Growth Factor-23, Humans, PHEX Phosphate Regulating Neutral Endopeptidase, Peptide Fragments physiology, Proto-Oncogene Proteins physiology, Fibroblast Growth Factors physiology, Phosphates metabolism, Proteins physiology
Abstract

PTH and active vitamin D are well known as classical phosphate regulating hormones. However, presence of some phosphaturic factors is assumed from investigations about TIO, XLH, ADHR. Those diseases cause hypophsophatemia, hyperphosphaturia, low vitamin D and rickets/ostepmalacia. FGF-23, which has been detected from TIO tumors, can induce hypophosphatemia by direct inhibition on phosphate reabsorption and by suppressing 1,25 (OH)(2)D(3) production through the inhibition of 25-hydroxyvitaminD 1alpha-hydroxylase, in the kidney. We have still other phosphatonin candidates such as MEPE, FRP4, etc. The role of these substances are not clear yet. Future investigations are required to clarify their roles in phosphate metabolism.

Published
2005
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