Search

Your search keyword '"Ye, Qiurong"' showing total 23 results
Start Over Author "Ye, Qiurong"
23 results on '"Ye, Qiurong"'

6. Parentage assignment and parental contribution analysis in large yellow croaker Larimichthys crocea using microsatellite markers

Author

Xiande LIU, Guangtai ZHAO, Zhiyong WANG, Mingyi CAI, Hua YE, Qiurong WANG

Subjects
Larimichthys crocea, Parental contribution, Parentage assignment, Microsatellite marker, Zoology, QL1-991
Abstract

The large yellow croaker Larimichthys crocea is one of the most important fish species in China. To estimate the reproductive success of breeders, three independent full-factorial crosses were created and the fins of breeders and progenies were sampled for microsatellite analysis. Out of 959 offspring from three sets, 99.6% were assigned to their parents using 6–7 microsatellite markers. In all crosses, some parent pairs produced a large number of offspring and some parent pairs did not produce any offspring. The contributions of male or female parents were unequal, ranging from 1.0–89.3% across the three sets. The loss of putative Ne was 69.6% in set 1, 31.2% in set 2 and 57.6% in set 3. These results suggest that the unequal contribution of parents is universal in artificial breeding of L. crocea, especially in a small population, and this should be taken into account in hatcheries or when releasing animals for resource enhancement [Current Zoology 58 (2): 244-249, 2012].

Published
2012

13. Lactoferrin deficiency induces a pro-metastatic tumor microenvironment through recruiting myeloid-derived suppressor cells in mice

Author

Wei, Lingyu, primary, Zhang, Xuemei, additional, Wang, Jia, additional, Ye, Qiurong, additional, Zheng, Xiang, additional, Peng, Qiu, additional, Zheng, Ying, additional, Liu, Peishan, additional, Zhang, Xiaoyue, additional, Li, Zhengshuo, additional, Liu, Can, additional, Yan, Qun, additional, Li, Guiyuan, additional, and Ma, Jian, additional

Published
2019
Full Text
View/download PDF

14. Suppression Colitis and Colitis-Associated Colon Cancer by Anti-S100a9 Antibody in Mice

Author

Zhang, Xuemei, primary, Wei, Lingyu, additional, Wang, Jing, additional, Qin, Zailong, additional, Wang, Jia, additional, Lu, Yuanjun, additional, Zheng, Xiang, additional, Peng, Qiu, additional, Ye, Qiurong, additional, Ai, Feiyan, additional, Liu, Peishan, additional, Wang, Siwen, additional, Li, Guiyuan, additional, Shen, Shourong, additional, and Ma, Jian, additional

Published
2017
Full Text
View/download PDF

15. Abnormal expression of p-ATM/CHK2 in nasal extranodal NK/T cell lymphoma, nasal type, is correlated with poor prognosis

Author

Ye, Qiurong, Chen, Huiling, Wen, Zonghua, Guo, Wenwen, Huang, Yongta, and Mo, Xianglan

Abstract

AimsThe aim of this study is to investigate the expression profiles of cell cycle related proteins in nasal extranodal NK/T cell lymphoma, nasal type (ENKTCL).MethodsThe expression profiles of cell cycle related proteins were assessed with a cell cycle antibody array and validated by immunohistochemistry. Correlations between the expression levels of proteins and clinical outcomes of patients with nasal ENKTCL were evaluated.ResultsThe expression of full length ataxia telangiectasia mutated (ATM) in nasal ENKTCL significantly decreased compared with that in nasal benign lymphoid proliferative disease (NBLPD), but the expression levels of p-ATM, CHK2 and RAD51 significantly increased in nasal ENKTCL compared with that in NBLPD. Kaplan-Meier analysis showed that the expression levels of p-ATM and CHK2 in nasal ENKTCL were inversely related to overall survival (p=0.011 and p=0.025, respectively).ConclusionAbnormalities in the ATM pathway may play a crucial role in the oncogenesis and chemoradiotherapy resistance of nasal ENKTCL.

Published
2021
Full Text
View/download PDF

16. Epstein-Barr Virus miR-BART6-3p Inhibits the RIG-I Pathway

Author

Lu, Yuanjun, primary, Qin, Zailong, additional, Wang, Jia, additional, Zheng, Xiang, additional, Lu, Jianhong, additional, Zhang, Xuemei, additional, Wei, Lingyu, additional, Peng, Qiu, additional, Zheng, Ying, additional, Ou, Chunlin, additional, Ye, Qiurong, additional, Xiong, Wei, additional, Li, Guiyuan, additional, Fu, Yuxin, additional, Yan, Qun, additional, and Ma, Jian, additional

Published
2017
Full Text
View/download PDF

20. Lactoferrin Deficiency Promotes Colitis-Associated Colorectal Dysplasia in Mice

Author

Ye, Qiurong, primary, Zheng, Ying, additional, Fan, Songqing, additional, Qin, Zailong, additional, Li, Nan, additional, Tang, Anliu, additional, Ai, Feiyan, additional, Zhang, Xuemei, additional, Bian, Yanhui, additional, Dang, Wei, additional, Huang, Jing, additional, Zhou, Ming, additional, Zhou, Yanhong, additional, Xiong, Wei, additional, Yan, Qun, additional, Ma, Jian, additional, and Li, Guiyuan, additional

Published
2014
Full Text
View/download PDF

21. MicroRNAs Provide Feedback Regulation of Epithelial-Mesenchymal Transition Induced by Growth Factors.

Author

Qin, Zailong, He, Wei, Tang, Jingqun, Ye, Qiurong, Dang, Wei, Lu, Yuanjun, Wang, Jia, Li, Guiyuan, Yan, Qun, and Ma, Jian

Subjects
MICRORNA, EPITHELIAL cells, MESENCHYMAL stem cells, GROWTH factors, GENE expression, CELL differentiation, APOPTOSIS
Abstract

As regulators in gene expression, microRNAs take part in most biological processes including cell differentiation, apoptosis, cell cycle, and epithelial-to-mesenchymal transition (EMT). In order to evaluate their roles in EMT process, microRNA expression profile changes induced by EGF or TGF-β treatment on nasopharyngeal carcinoma cell HK-1 were analyzed, and miR-21, miR-148a, miR-505, and miR-1207-5p were found to be upregulated in growth factors-induced EMT process. miR-21 is already known as an oncogenic miRNA to promote metastasis, however, the exact functions of other three miRNAs in EMT are unclear. To our surprise, we found that miR-148a, miR-505, and miR-1207-5p can suppress EMT and metastasis phenotypes in HK-1 cells both in vitro and in vivo, which may relate to their inhibition on EMT and Wnt signaling molecules. MiRNAs confer robustness to biological processes by posttranscriptional repression of key transcriptional programs that are related to previous developmental stages or to alternative cell fates. Our findings indicate that miRNA feedback circuit is tuned to respond to growth factors-induced EMT, and we suggested a new negative feedback loop which may be an important element of the EMT process and confer biological robustness. J. Cell. Physiol. 230: 120-129, 2016. © 2015 Wiley Periodicals, Inc. [ABSTRACT FROM AUTHOR]

Published
2016
Full Text
View/download PDF

22. Inhibition of Epstein-Barr Virus Infection by Lactoferrin

Author

Zheng, Ying, primary, Zhang, Wenling, additional, Ye, Qiurong, additional, Zhou, Yanhong, additional, Xiong, Wei, additional, He, Wei, additional, Deng, Min, additional, Zhou, Ming, additional, Guo, Xiaofang, additional, Chen, Pan, additional, Fan, Songqing, additional, Liu, Xiaoping, additional, Wang, Zhen, additional, Li, Xiaoling, additional, Ma, Jian, additional, and Li, Guiyuan, additional

Published
2012
Full Text
View/download PDF

Catalog

Books, media, physical & digital resources
See catalog results