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1. ACECode: A Reinforcement Learning Framework for Aligning Code Efficiency and Correctness in Code Language Models

2. Variational learning of integrated quantum photonic circuits

3. PTM4Tag+: Tag Recommendation of Stack Overflow Posts with Pre-trained Models

4. Variational Quantum Circuit Decoupling

5. Proteogenomic analysis of human cerebrospinal fluid identifies neurologically relevant regulation and implicates causal proteins for Alzheimer’s disease

6. Genetic architecture of cerebrospinal fluid and brain metabolite levels and the genetic colocalization of metabolites with human traits

7. Dimension reduction in quantum sampling of stochastic processes

8. APIDocBooster: An Extract-Then-Abstract Framework Leveraging Large Language Models for Augmenting API Documentation

11. Proteo-genomics of soluble TREM2 in cerebrospinal fluid provides novel insights and identifies novel modulators for Alzheimer’s disease

12. Multi-Granularity Detector for Vulnerability Fixes

13. Implementing quantum dimensionality reduction for non-Markovian stochastic simulation

14. PTM4Tag: Sharpening Tag Recommendation of Stack Overflow Posts with Pre-trained Models

15. Efficient Search of Live-Coding Screencasts from Online Videos

16. Aspect-Based API Review Classification: How Far Can Pre-Trained Transformer Model Go?

17. CSF proteomics identifies early changes in autosomal dominant Alzheimer’s disease

18. Provable superior accuracy in machine learned quantum models

20. Measures of distinguishability between stochastic processes

21. Extreme dimensionality reduction with quantum modelling

22. Matrix Product States for Quantum Stochastic Modelling

23. Variational Learning of Integrated Quantum Photonic Circuits via Genetic Algorithm.

24. Genetic proxies for predicting plasma protein levels reveal TIMP2 role in human cognitive performance.

26. Genomic atlas of the proteome from brain, CSF and plasma prioritizes proteins implicated in neurological disorders

29. A 3′UTR Insertion Is a Candidate Causal Variant at the TMEM106B Locus Associated With Increased Risk for FTLD-TDP

31. Proteome Wide Association Studies of LRRK2 variants identify novel causal and druggable proteins for Parkinson’s disease

34. The TMEM106B FTLD-protective variant, rs1990621, is also associated with increased neuronal proportion

36. Functional genomic analyses uncover APOE-mediated regulation of brain and cerebrospinal fluid beta-amyloid levels in Parkinson disease

38. A 3’UTR Insertion Is a Candidate Causal Variant at theTMEM106BLocus Associated with Increased Risk for FTLD-TDP

39. Proteo-genomics of soluble TREM2 in cerebrospinal fluid provides novel insights and identifies novel modulators for Alzheimer’s disease

40. Unique genetic architecture of CSF and brain metabolites pinpoints the novel targets for the traits of human wellness

41. Proteogenomic analysis of human cerebrospinal fluid identifies neurologically relevant regulation and informs causal proteins for Alzheimer’s disease

42. Large‐scale proteome and metabolome analysis of CSF implicates altered glucose and carbon metabolism and succinylcarnitine in Alzheimer's disease.

44. Genetic analyses of the plasma proteome and metabolome from the same cohort pinpoint AD risk associated molecular phenotypes.

46. Multi-Granularity Detector for Vulnerability Fixes

47. Proteomic clusters underlie heterogeneity in preclinical Alzheimer’s disease progression

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