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2. Abstracts of selected papers presented at the 33rd annual meeting of the japanese society of gastroenterology

Author

Fujii, Daigo, Suyama, Masafumi, Sugiyama, Masanori, Atomi, Yutaka, Inui, Kazuo, Nakazawa, Saburo, Nakajima, Kimihiro, Katoh, Hiroyuki, Kaneda, Makoto, Ogura, Yoshifumi, Eto, Toshifumi, Tsunoda, Tsukasa, Uchimura, Masayuki, Narita, Kazuyuki, Sugioka, Atsushi, Tsuzuki, Toshiharu, Kanemitsu, Keiichiro, Tashiro, Seiki, Yoshikawa, Tatsuya, Hanyu, Fujio, Kondo, Satoshi, Nimura, Yuji, Eriguchi, Naofumi, Nakayama, Toshimichi, Mitake, Masahiro, Naitoh, Yasuo, Isawa, T., Okamoto, A., Miyazaki, Hiroshi, Ogata, Yoshirou, Kuno, Nobuyoshi, Kurimoto, Kumiko, Noguchi, Tohru, Makuuchi, Masatoshi, Ikari, Takaaki, Ariyama, Joe, Takao, Sonshin, Shimazu, Hisaaki, Kobari, Masao, Matsuno, Seiki, Tanigawa, K., Mizumoto, R., Nakasako, T., Hanyu, F., Saito, Naoyuki, Hiraoka, Takehisa, Ishikawa, Osamu, Imaoka, Shingi, Miyazaki, Naoyuki, Saitoh, Yoichi, Harada, Akio, Takagi, Hiroshi, Kawai, Y., Yamamoto, S., Nakanishi, Ryo, Beppu, Tomoe, Okushiba, Shunichi, Katoh, Hiroyuki, Sakamoto, Hiroaki, Obara, Katsutoshi, Toba, Masahito, Onda, Masahiko, Azuma, Masayoshi, Kashiwagi, Toru, Sumino, Michihiro, Toyonaga, Atsushi, Hagiwara, Masaru, Sakai, Masahiro, Kokubu, Shigehiro, Shibata, Hisao, Miyoshi, Hirofumi, Orino, Shinya, Yoshida, Tomoharu, Itoh, Tadahiko, Murashima, Naoya, Matsumura, Masahiko, Tsujii, Tadasu, Murayama, Takashi, Tameda, Yukihiko, Matsutani, Shoichi, Ishii, Hiroshi, Mitarai, Yoshinobu, Kobayashi, Michio, Nishida, Hitoshi, Kawada, Taiji, Matsuda, Hiroko, Kawasaki, Tsunehisa, Arakawa, Tetsuo, Kobayashi, Kenzo, Ota, Shinichi, Terano, Akira, Nakamura, Masahiko, Oda, Masaya, Tari, Akira, Yamamoto, Goso, Saito, Eiichi, Matsuo, Yutaka, Mizuno, Motowo, Tomoda, Jun, Asada, Shuji, Takiuchi, Hiroya, Mogi, Mizuhiro, Kitajima, Masaki, Nagano, Kouichi, Sato, Nobuhiro, Naito, Yuji, Yoshikawa, Toshikazu, Asaka, Masahiro, Sato, Yasuo, Iwafuchi, Mitsuya, Watanabe, Hidenobu, Yoshida, Yukio, Kihira, Ken, Kashiwagi, Hideyuki, Aoki, Teruaki, Nishikawa, Masahiro, Suzuki, Takashi, Matsui, Toshiyuki, Iida, Mitsuo, Katoh, H., Munakata, A., Itsuno, Minoru, Makiyama, Kazuya, Hibi, Toshifumi, Kitahora, Tetsuji, Igarashi, Masahiro, Katsumata, Tomoe, Satomi, Masamichi, Yamamura, Makoto, Hirata, Ichiro, Slezak, Premysl, Kubota, Yoshiro, Muto, Tetsuichiro, Morise, Kimitomo, Kusugami, Kazuo, Sugita, Akira, Fukushima, Tsuneo, Okada, Mitsuo, Sakurai, Toshihiro, Iida, Mitsuo, Kawasaki, Atsushi, Hiwatashi, Nobuo, Yamazaki, Hideo, Oriishi, Tetsuharu, Sasaki, Ei, Takazoe, Masakazu, Inoue, Noboru, Funayama, Y., and Matsuno, S.

Published
1993
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3. Helicobacter pylori Infection and Gastroduodenal Disease : a Comparison of Endoscopic Findings, Histology, and Urease Test Data

Author

Haruma, Ken, Okamoto, Shiro, Sumii, Koji, Yoshihara, Masaharu, Tari, Akira, Teshima, Hitosihi, Shimamoto, Takehiro, Takehara, Yoshihiko, Tanaka, Shinji, Yamamoto, Goso, Teixeira, Claudio Rolim, Sumii, Masaharu, Kishimoto, Shinya, Kajiyama, Goro, and Wagner, Siegfried

Subjects
Peptic ulcer, Gastritis, Gastric carcinoma, digestive system diseases, Helicobacter pylori infection
Abstract

To determine the prevalence and significance of Helicobacter pylori (H. pylori) infection, biopsies of the antral mucosa were obtained from 139 patients and 43 asymptomatic volunteers. The specimens were examined by hematoxylin-eosin staining and the urease test. The detection rate of H. pylori by histologic examination was 91.3% in patients with duodenal ulcer, 75.0% in those with combined duodenal and gastric ulcer, 63.6% in those with gastric ulcer, 22.9% in those with gastric carcinoma, 36.4% in those with gastric adenoma, 14.3% in those with gastric hyperplastic polyp, and 51.7% in those with gastritis, and the respective percentages detected by the urease test were 91.3%, 75.0%, 54.5%, 28.6%, 27.3%, 14.3%, and 44.8%. H. pylori was also detected in 10/43 (23.3%) asymptomatic healthy volunteers by histology and the urease test. The prevalence of H. pylori was significantly higher in the patients than in the asymptomatic healthy volunteers (p < 0.05). H. pylori was detected in 62.9% of patients with endoscopic erosive gastritis and in 97.9% of those with histologically proven chronic active gastritis. The urease test was positive in 77/82 patients who were histologically positive for the organism (sensitivity: 93.9%), and it was negative in 98/100 patients who were negative by histology (specificity: 98.0%). Thus, there was over 90% agreement between the urease test and histology. Our investigations showed that H. pylori was closely related to peptic ulcers and antral gastritis, and that the urease test provides a simple, rapid and accurate diagnosis of H. pylori infection.

Published
1992

4. Expression of Antral Gastrin and Somatostatin mRNA in Helicobacter pylori-infected Subjects.

Author

Sumii, Masaharu, Sumii, Koji, Tari, Akira, Kawaguchi, Hiroyuki, Yamamoto, Goso, Takehara, Yoshihiko, Fukino, Yoichi, Kamiyasu, Toshiki, Hamada, Masanori, Tsuda, Toshitaka, Yoshihara, Masaharu, Haruma, Ken, and Kajiyama, Goro

Subjects
GENE expression, GASTRIN, SOMATOSTATIN, HELICOBACTER pylori, MUCOUS membranes
Abstract

Objectives: We investigated the gene expression of antral gastrin and somatostatin in Helicobacter pylori-infected subjects. Twelve asymptomatic men with normal endoscopic findings participated in this study, four of whom were infected with H. pylori. Methods: Biopsy specimens of the gastric mucosa were obtained after subjects had fasted overnight. Serum gastrin levels, antral gastrin and somatostatin content, and antral G- and D-cell densities also were measured. Results: Fasting serum gastrin levels were significantly higher in H. pylori-positive subjects than in H. pylori-negative subjects. There were no differences between groups in antral gastrin content, G-cell density, or gastrin mRNA levels. Antral somatostatin content, D-cell density, somatostatin mRNA levels, and the somatostatin mRNA:D-cell density ratio were significantly decreased in H. pylori-positive subjects. Conclusions: Our results suggest that in addition to reduced antral D-cell density, a suppression of somatostatin synthesis in D-cells may have contributed to the decrease in antral somatostatin in H. pylori-infected subjects. [ABSTRACT FROM AUTHOR]

Published
1994

5. Ultrasonographic Assessment of Inflammatory Bowel Disease.

Author

Hata, Jiro, Haruma, Ken, Suenaga, Kenji, Yoshihara, Masaharu, Yamamoto, Goso, Tanaka, Shinji, Shimamoto, Takehiro, Sumii, Koji, and Kajiyama, Goro

Subjects
ULTRASONIC imaging, CROHN'S disease, ULCERATIVE colitis, INTESTINAL diseases, INFLAMMATORY bowel diseases
Abstract

Ultrasound examinations were performed in 36 patients with Crohn's disease 28 with ulcerative colitis and 50 with no bowel disease. The pathological findings were classified into three types and compared with the radiographic and/or colonoscopic findings. Crohn's disease and ulcerative colitis could be detected by ultrasonography with a sensitivity of 86% and 89%, respectively. The ultrasonographic features correlated with the radiographic/colonoscopic findings and with disease activity, but did not help much in making a differential diagnosis, although the location of the pathologic changes was helpful to some extent. In conclusion, ultrasonography can serve as a useful alternative diagnostic procedure that permits us to obtain information about transmural changes in inflammatory bowel disease. [ABSTRACT FROM AUTHOR]

Published
1992

6. Famotidine, a histamine-2-receptor antagonist, inhibits the increase in rat gastric H+/K+-ATPase mRNA induced by intravenous infusion of gastrin 17 and histamine

Author

Yamamoto, Goso, Tari, Akira, Sumii, Koji, Sumii, Masaharu, Haruma, Ken, and Kajiyama, Goro

Abstract

We examined the effects of gastrin and histamine on rat gastric H+/K+-ATPase, the enzyme responsible for H+ secretion, gene expressionin vivo. Gastrin 17 (G 17) or histamine dihydrochloride (histamine) was continuously infused through the femoral vein of anesthetized rats. Gastric H+/K+-ATPase mRNA levels were measured using northern blot analysis. Infusion of G 17 and histamine increased the H+/K+-ATPase mRNA level significantly compared with basal control level or vehicle control level (P<0.01). However, pretreatment with famotidine, a potent histamine-2 (H2)-receptor antagonist, inhibited the increase of rat gastric H+/K+-ATPase mRNA following G 17 and histamine infusion. These findings indicate that both histamine and G 17 increase expression of H+/K+-ATPase mRNA by activating H2 receptor on the parietal cell.

Published
1995
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7. Helicobacter pylori Infection and Gastroduodenal Disease : a Comparison of Endoscopic Findings, Histology, and Urease Test Data

Author

Haruma, Ken, Okamoto, Shiro, Sumii, Koji, Yoshihara, Masaharu, Tari, Akira, Teshima, Hitosihi, Shimamoto, Takehiro, Takehara, Yoshihiko, Tanaka, Shinji, Yamamoto, Goso, Teixeira, Claudio Rolim, Sumii, Masaharu, Kishimoto, Shinya, Kajiyama, Goro, Wagner, Siegfried, Haruma, Ken, Okamoto, Shiro, Sumii, Koji, Yoshihara, Masaharu, Tari, Akira, Teshima, Hitosihi, Shimamoto, Takehiro, Takehara, Yoshihiko, Tanaka, Shinji, Yamamoto, Goso, Teixeira, Claudio Rolim, Sumii, Masaharu, Kishimoto, Shinya, Kajiyama, Goro, and Wagner, Siegfried

Abstract

To determine the prevalence and significance of Helicobacter pylori (H. pylori) infection, biopsies of the antral mucosa were obtained from 139 patients and 43 asymptomatic volunteers. The specimens were examined by hematoxylin-eosin staining and the urease test. The detection rate of H. pylori by histologic examination was 91.3% in patients with duodenal ulcer, 75.0% in those with combined duodenal and gastric ulcer, 63.6% in those with gastric ulcer, 22.9% in those with gastric carcinoma, 36.4% in those with gastric adenoma, 14.3% in those with gastric hyperplastic polyp, and 51.7% in those with gastritis, and the respective percentages detected by the urease test were 91.3%, 75.0%, 54.5%, 28.6%, 27.3%, 14.3%, and 44.8%. H. pylori was also detected in 10/43 (23.3%) asymptomatic healthy volunteers by histology and the urease test. The prevalence of H. pylori was significantly higher in the patients than in the asymptomatic healthy volunteers (p < 0.05). H. pylori was detected in 62.9% of patients with endoscopic erosive gastritis and in 97.9% of those with histologically proven chronic active gastritis. The urease test was positive in 77/82 patients who were histologically positive for the organism (sensitivity: 93.9%), and it was negative in 98/100 patients who were negative by histology (specificity: 98.0%). Thus, there was over 90% agreement between the urease test and histology. Our investigations showed that H. pylori was closely related to peptic ulcers and antral gastritis, and that the urease test provides a simple, rapid and accurate diagnosis of H. pylori infection.

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