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403 results on '"Yamada, Yasutaka"'

1. Implications of unconventional histological subtypes on magnetic resonance imaging and oncological outcomes in patients who have undergone radical prostatectomy

Author

Kurokawa, Koichiro, Yamada, Yasutaka, Sakamoto, Shinichi, Horikoshi, Takuro, Sato, Kodai, Nanba, Sakie, Kubota, Yoshihiro, Kanesaka, Manato, Fujimoto, Ayumi, Takeuchi, Nobuyoshi, Shibata, Hiroki, Sazuka, Tomokazu, Imamura, Yusuke, Tsuzuki, Toyonori, Uno, Takashi, and Ichikawa, Tomohiko

Published
2024
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4. Notch signaling suppresses neuroendocrine differentiation and alters the immune microenvironment in advanced prostate cancer

Author

Ku, Sheng-Yu, Wang, Yanqing, Garcia, Maria Mica, Yamada, Yasutaka, Mizuno, Kei, Long, Mark D., Rosario, Spencer, Chinnam, Meenalakshmi, Assaad, Majd Al, Puca, Loredana, Kim, Min Jin, Bakht, Martin K., Venkadakrishnan, Varadha Balaji, Robinson, Brian D., Acosta, Andres M., Wadosky, Kristine M., Mosquera, Juan Miguel, Goodrich, David W., and Beltran, Himisha

Subjects
Physiological aspects, Health aspects, Cell receptors -- Health aspects, Neuroendocrinology -- Health aspects, Cancer research, Immune system -- Health aspects, Prostate cancer -- Physiological aspects, Cellular signal transduction -- Health aspects, Oncology, Experimental, Cancer -- Research
Abstract

Introduction Phenotypic plasticity is a hallmark of cancer (1-3). In advanced prostate cancer, up to 15%-20% of patients develop resistance to androgen receptor-directed (AR-directed) therapies through conversion from an androgen-driven [...], Notch signaling can have either an oncogenic or tumor-suppressive function in cancer depending on the cancer type and cellular context. While Notch can be oncogenic in early prostate cancer, we identified significant downregulation of the Notch pathway during prostate cancer progression from adenocarcinoma to neuroendocrine (NE) prostate cancer, where it functions as a tumor suppressor. Activation of Notch in NE and Rb1/Trp53-deficient prostate cancer models led to phenotypic conversion toward a more indolent, non-NE state with glandular features and expression of luminal lineage markers. This was accompanied by upregulation of MHC and type I IFN and immune cell infiltration. Overall, these data support Notch signaling as a suppressor of NE differentiation in advanced prostate cancer and provide insights into how Notch signaling influences lineage plasticity and the tumor microenvironment (TME).

Published
2024
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6. Landscape of prostate-specific membrane antigen heterogeneity and regulation in AR-positive and AR-negative metastatic prostate cancer

Author

Bakht, Martin K., Yamada, Yasutaka, Ku, Sheng-Yu, Venkadakrishnan, Varadha Balaji, Korsen, Joshua A., Kalidindi, Teja M., Mizuno, Kei, Ahn, Shin Hye, Seo, Ji-Heui, Garcia, Maria Mica, Khani, Francesca, Elemento, Olivier, Long, Henry W., Chaglassian, Alain, Pillarsetty, Nagavarakishore, Lewis, Jason S., Freedman, Matthew, Belanger, Anthony P., Nguyen, Quang-De, and Beltran, Himisha

Published
2023
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7. Integrated analysis of tertiary lymphoid structures in relation to tumor-infiltrating lymphocytes and patient survival in pancreatic ductal adenocarcinoma

Author

Tanaka, Takeshi, Masuda, Atsuhiro, Inoue, Jun, Hamada, Tsuyoshi, Ikegawa, Takuya, Toyama, Hirochika, Sofue, Keitaro, Shiomi, Hideyuki, Sakai, Arata, Kobayashi, Takashi, Tanaka, Shunta, Nakano, Ryota, Yamada, Yasutaka, Ashina, Shigeto, Tsujimae, Masahiro, Yamakawa, Kohei, Abe, Shohei, Gonda, Masanori, Masuda, Shigeto, Inomata, Noriko, Uemura, Hisahiro, Kohashi, Shinya, Nagao, Kae, Kanzawa, Maki, Itoh, Tomoo, Ueda, Yoshihide, Fukumoto, Takumi, and Kodama, Yuzo

Published
2023
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8. Comprehensive Analysis of Molecular Biologic Characteristics of Pancreatic Ductal Adenocarcinoma Concomitant with Intraductal Papillary Mucinous Neoplasm

Author

Tsujimae, Masahiro, Masuda, Atsuhiro, Ikegawa, Takuya, Tanaka, Takeshi, Inoue, Jun, Toyama, Hirochika, Sofue, Keitaro, Uemura, Hisahiro, Kohashi, Shinya, Inomata, Noriko, Nagao, Kae, Masuda, Shigeto, Abe, Shohei, Gonda, Masanori, Yamakawa, Kohei, Ashina, Shigeto, Yamada, Yasutaka, Tanaka, Shunta, Nakano, Ryota, Sakai, Arata, Kobayashi, Takashi, Shiomi, Hideyuki, Kanzawa, Maki, Itoh, Tomoo, Fukumoto, Takumi, Ueda, Yoshihide, and Kodama, Yuzo

Published
2022
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9. Advantage of endoscopic papillectomy for ampullary tumors as an alternative treatment for pancreatoduodenectomy

Author

Abe, Shohei, Sakai, Arata, Masuda, Atsuhiro, Miki, Mika, Harada, Yoshiyuki, Nagao, Kae, Inomata, Noriko, Kohashi, Shinya, Uemura, Hisahiro, Masuda, Shigeto, Ashina, Shigeto, Gonda, Masanori, Yamakawa, Kohei, Tsujimae, Masahiro, Yamada, Yasutaka, Tanaka, Takeshi, Kobayashi, Takashi, Nakano, Ryota, Shiomi, Hideyuki, Tsugawa, Daisuke, Yanagimoto, Hiroaki, Ajiki, Tetsuo, Kanzawa, Maki, Fukumoto, Takumi, Itoh, Tomoo, and Kodama, Yuzo

Published
2022
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10. Recent advances in basic research on prostate cancer: Where we are heading?

Author

Obinata, Daisuke, Yamada, Yasutaka, Sumiyoshi, Takayuki, Tanegashima, Tokiyoshi, Watanabe, Ryuta, Kobayashi, Hiroaki, Ito, Daisuke, and Urabe, Fumihiko

Subjects
*ANDROGEN receptors, *ANDROGEN deprivation therapy, *GENOMICS, *PROSTATE biopsy, *TUMOR microenvironment, *CIRCULATING tumor DNA, *PROSTATE cancer
Abstract

In the over 80 years since androgens were found to play a pivotal role in prostate cancer (PCa) progression, androgen deprivation therapy (ADT) has been a cornerstone in treating advanced PCa. Castration‐resistant PCa persists, however, with some of these tumors evolving to androgen receptor (AR)‐independent forms like neuroendocrine PCa. The development of novel diagnostic and therapeutic approaches to PCa is therefore crucial. This review provides an overview of recent basic research in PCa, focusing on two main areas: PCa cells and their tumor microenvironments. The first section describes current knowledge on the intricate mechanisms of AR signaling pathways, emphasizing the roles of coactivators and chromatin state alterations in gene regulation. Genomic analyses have revealed recurrent mutations and copy number alterations critical for precision medicine. Liquid biopsy has become a promising tool for real‐time tumor monitoring, identifying genetic alterations in circulating‐tumor DNA or extracellular vesicles. The second section describes the tumor microenvironment of PCa, highlighting its immunosuppressive landscape and the potential of combining ADT with immunotherapy. Advanced techniques, including single‐cell RNA sequencing and spatial transcriptomics offer insights into cellular heterogeneity and interactions within the tumor microenvironment, paving the way for novel therapeutic strategies. Integration of these diverse research areas will provide a comprehensive understanding of the current state and future directions of PCa research, underscoring the importance of personalized medicine and the dynamic nature of cancer treatment strategies. [ABSTRACT FROM AUTHOR]

Published
2024
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14. Acute pancreatitis in intraductal papillary mucinous neoplasms correlates with pancreatic volume and epithelial subtypes

Author

Tanaka, Takeshi, Masuda, Atsuhiro, Sofue, Keitaro, Toyama, Hirochika, Shiomi, Hideyuki, Sakai, Arata, Kobayashi, Takashi, Tanaka, Shunta, Nakano, Ryota, Yamada, Yasutaka, Ashina, Shigeto, Tsujimae, Masahiro, Yamakawa, Kohei, Abe, Shohei, Gonda, Masanori, Masuda, Shigeto, Inomata, Noriko, Uemura, Hisahiro, Kohashi, Shinya, Nagao, Kae, Kanzawa, Maki, Itoh, Tomoo, Fukumoto, Takumi, and Kodama, Yuzo

Published
2021
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15. The LAT1 inhibitor JPH203 suppresses the growth of castration‐resistant prostate cancer through a CD24‐mediated mechanism.

Author

Saito, Shinpei, Ando, Keisuke, Sakamoto, Shinichi, Xu, Minhui, Yamada, Yasutaka, Rii, Junryo, Kanaoka, Sanji, Wei, Jiaxing, Zhao, Xue, Pae, Sangjon, Kanesaka, Manato, Goto, Yusuke, Sazuka, Tomokazu, Imamura, Yusuke, Reien, Yoshie, Hamaguchi‐Suzuki, Norie, Saito, Shota, Hirayama, Yuri, Hashimoto, Hirofumi, and Kanai, Yoshikatsu

Abstract

L‐type amino acid transporter 1 (LAT1) is specifically expressed in many malignancies, contributes to the transport of essential amino acids, such as leucine, and regulates the mammalian target of rapamycin (mTOR) signaling pathway. We investigated the expression profile and functional role of LAT1 in prostate cancer using JPH203, a specific inhibitor of LAT1. LAT1 was highly expressed in castration‐resistant prostate cancer (CRPC) cells, including C4‐2 and PC‐3 cells, but its expression level was low in castration‐sensitive LNCaP cells. JPH203 significantly inhibited [14C] leucine uptake in CRPC cells but had no effect in LNCaP cells. JPH203 inhibited the proliferation, migration, and invasion of CRPC cells but not of LNCaP cells. In C4‐2 cells, Cluster of differentiation (CD) 24 was identified by RNA sequencing as a novel downstream target of JPH203. CD24 was downregulated in a JPH203 concentration‐dependent manner and suppressed activation of the Wnt/β‐catenin signaling pathway. Furthermore, an in vivo study showed that JPH203 inhibited the proliferation of C4‐2 cells in a castration environment. The results of this study indicate that JPH203 may exert its antitumor effect in CRPC cells via mTOR and CD24. [ABSTRACT FROM AUTHOR]

Published
2024
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16. Testosterone bounce predicts favorable prognoses for prostate cancer patients treated with degarelix

Author

Kamada, Shuhei, primary, Sakamoto, Shinichi, additional, Kinoshita, Ryo, additional, Zhao, Xue, additional, Kamasako, Tomohiko, additional, Yamase, Ryosuke, additional, Junryo, Rii, additional, Saito, Shinpei, additional, Sangjon, Pae, additional, Takei, Akinori, additional, Yamada, Yasutaka, additional, Goto, Yusuke, additional, Imamura, Yusuke, additional, Iguchi, Taro, additional, Mizokami, Atsushi, additional, Suzuki, Hiroyoshi, additional, Akakura, Koichiro, additional, and Ichikawa, Tomohiko, additional

Published
2024
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17. Copy Number Gain in Androgen Receptors Predicts the Poor Prognosis in Japanese Castration-resistant Prostate Cancer

Author

SAKAMOTO, SHINICHI, primary, ANDO, KEISUKE, additional, PAE, SANGJON, additional, ZHAO, XUE, additional, SAKAI, KAZUKO, additional, SATO, KODAI, additional, SAITO, SHINPEI, additional, YAMADA, YASUTAKA, additional, RII, JUNRYO, additional, GOTO, YUSUKE, additional, SAZUKA, TOMOKAZU, additional, IMAMURA, YUSUKE, additional, ANZAI, NAOHIKO, additional, AKAKURA, KOICHIRO, additional, NISHIO, KAZUTO, additional, and ICHIKAWA, TOMOHIKO, additional

Published
2024
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18. Implications of unconventional histological subtypes on magnetic resonance imaging and oncological outcomes in patients who have undergone radical prostatectomy

Author

Kurokawa, Koichiro, primary, Yamada, Yasutaka, additional, Sakamoto, Shinichi, additional, Horikoshi, Takuro, additional, Sato, Kodai, additional, Namba, Sakie, additional, Kubota, Yoshihiro, additional, Kanesaka, Manato, additional, Fujimoto, Ayumi, additional, Takeuchi, Nobuyoshi, additional, Shibata, Hiroki, additional, Sazuka, Tomokazu, additional, Imamura, Yusuke, additional, Tsuzuki, Toyonori, additional, Uno, Takashi, additional, and Ichikawa, Tomohiko, additional

Published
2024
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19. L‐type amino acid transporter 1 inhibitor JPH203 prevents the growth of cabazitaxel‐resistant prostate cancer by inhibiting cyclin‐dependent kinase activity

Author

Rii, Junryo, primary, Sakamoto, Shinichi, additional, Mizokami, Atsushi, additional, Xu, Minhui, additional, Fujimoto, Ayumi, additional, Saito, Shinpei, additional, Koike, Hidekazu, additional, Tamura, Takaaki, additional, Arai, Takayuki, additional, Yamada, Yasutaka, additional, Goto, Yusuke, additional, Sazuka, Tomokazu, additional, Imamura, Yusuke, additional, Suzuki, Kazuhiro, additional, Kanai, Yoshikatsu, additional, Anzai, Naohiko, additional, and Ichikawa, Tomohiko, additional

Published
2024
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20. ASO Visual Abstract: Comprehensive Analysis of Molecular Biological Characteristics of Pancreatic Ductal Adenocarcinoma Concomitant with Intraductal Papillary Mucinous Neoplasm

Author

Tsujimae, Masahiro, Masuda, Atsuhiro, Ikegawa, Takuya, Tanaka, Takeshi, Inoue, Jun, Toyama, Hirochika, Sofue, Keitaro, Uemura, Hisahiro, Kohashi, Shinya, Inomata, Noriko, Nagao, Kae, Masuda, Shigeto, Abe, Shohei, Gonda, Masanori, Yamakawa, Kohei, Ashina, Shigeto, Yamada, Yasutaka, Tanaka, Shunta, Nakano, Ryota, Sakai, Arata, Kobayashi, Takashi, Shiomi, Hideyuki, Kanzawa, Maki, Itoh, Tomoo, Fukumoto, Takumi, Ueda, Yoshihide, and Kodama, Yuzo

Published
2022
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21. A case of high-grade pancreatic intraepithelial neoplasia diagnosed based on focal pancreatic parenchymal atrophy after acute pancreatitis

Author

Inomata, Noriko, Kobayashi, Takashi, Masuda, Atsuhiro, Masuda, Shigeto, Ashina, Shigeto, Gonda, Masanori, Abe, Shohei, Yamakawa, Kohei, Tsujimae, Masahiro, Tanaka, Takeshi, Yamada, Yasutaka, Tanaka, Shunta, Kakihara, Maya, Nakano, Ryota, Ikegawa, Takuya, Sakai, Arata, Shiomi, Hideyuki, Kannzawa, Maki, Toyama, Hirochika, Itoh, Tomoo, Fukumoto, Takumi, and Kodama, Yuzo

Published
2020
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23. Evaluation of efficacy of pancreatic juice cytology for risk classification according to international consensus guidelines in patients with intraductal papillary mucinous neoplasm; a retrospective study

Author

Yamakawa, Kohei, Masuda, Atsuhiro, Nakagawa, Takashi, Shiomi, Hideyuki, Toyama, Hirochika, Takenaka, Mamoru, Sakai, Arata, Kobayashi, Takashi, Tsujimae, Masahiro, Ashina, Shigeto, Yamada, Yasutaka, Tanaka, Takeshi, Tanaka, Shunta, Nakano, Ryota, Sato, Yu, Ikegawa, Takuya, Kurosawa, Manabu, Fujigaki, Seiji, Kutsumi, Hiromu, Itoh, Tomoo, Fukumoto, Takumi, and Kodama, Yuzo

Published
2019
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24. Preoperative PI-RADS v2.1 Scoring System Improves Risk Classification in Patients Undergoing Radical Prostatectomy

Author

FUKUI, YUDAI, primary, YAMADA, YASUTAKA, additional, SAKAMOTO, SHINICHI, additional, HORIKOSHI, TAKURO, additional, ZHAO, XUE, additional, SATO, KODAI, additional, NANBA, SAKIE, additional, KUBOTA, YOSHIHIRO, additional, KANESAKA, MANATO, additional, FUJIMOTO, AYUMI, additional, SHIBATA, HIROKI, additional, GOTO, YUSUKE, additional, SAZUKA, TOMOKAZU, additional, IMAMURA, YUSUKE, additional, UNO, TAKASHI, additional, and ICHIKAWA, TOMOHIKO, additional

Published
2023
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26. Metabolic Syndrome Accelerates the Age-Related Increase of Intraductal Papillary Mucinous Neoplasm of the Pancreas

Author

Tanaka, Shunta, primary, Tsujimae, Masahiro, additional, Masuda, Atsuhiro, additional, Inoue, Jun, additional, Inomata, Noriko, additional, Uemura, Hisahiro, additional, Kohashi, Shinya, additional, Nagao, Kae, additional, Masuda, Shigeto, additional, Abe, Shohei, additional, Gonda, Masanori, additional, Yamakawa, Kohei, additional, Ashina, Shigeto, additional, Nakano, Ryota, additional, Tanaka, Takeshi, additional, Yamada, Yasutaka, additional, Sakai, Arata, additional, Kobayashi, Takashi, additional, Shiomi, Hideyuki, additional, Fujita, Koichi, additional, Anami, Takahiro, additional, Fujita, Tsuyoshi, additional, Watanabe, Akihiko, additional, and Kodama, Yuzo, additional

Published
2023
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31. Progression of Recurrent Pancreatitis to Chronic Pancreatitis within 3 Years due to SPINK1 Mutation IVS3+2T>C

Author

Horitani, Susumu, Tsujimae, Masahiro, Sakai, Arata, Masuda, Atsuhiro, Nagao, Kae, Kohashi, Shinya, Inomata, Noriko, Uemura, Hisahiro, Masuda, Shigeto, Gonda, Masanori, Abe, Shohei, Yamakawa, Kohei, Ashina, Shigeto, Yamada, Yasutaka, Tanaka, Takeshi, Nakano, Ryota, Kobayashi, Takashi, Shiomi, Hideyuki, and Kodama, Yuzo

Subjects
Recurrent pancreatitis, SPINK1 mutation, Chronic pancreatitis
Abstract

When the etiology of pancreatitis cannot be determined despite sufficient investigation, recurrence and progression to chronic pancreatitis often involve genetic mutations. Herein, we describe a case of recurrent pancreatitis with the IVS3+2T>C mutation in the serine protease inhibitor Kazal type 1 (SPINK1) gene that progressed to chronic pancreatitis in only 3 years. A 35-year-old man was referred to our hospital, where he was diagnosed with mild pancreatitis and was treated conservatively. However, the patient experienced recurrent episodes of pancreatitis, which progressed to become chronic pancreatitis with a pancreatic calcification 1 year later. After 3 years, the patient developed pancreatic duct stenosis and required a pancreatic duct stent placement. Regarding the cause of chronic pancreatitis, alcohol abuse was ruled out based on history taking. Considering the course of treatment, autoimmune pancreatitis and obstructive pancreatitis, such as pancreatic divisum, were also ruled out. Finally, a germline genetic test was performed to determine the etiology of pancreatitis, which revealed the IVS3+2T>C mutation in SPINK1. This case shows the importance of genetic testing in patients with idiopathic pancreatitis to determine their etiology and is a rare incident that can report the progression of the disease from acute to chronic pancreatitis.

Published
2023

36. Expression of L-type amino acid transporter 1 as a molecular target for prognostic and therapeutic indicators in bladder carcinoma

Author

Maimaiti, Maihulan, Sakamoto, Shinichi, Yamada, Yasutaka, Sugiura, Masahiro, Rii, Junryo, Takeuchi, Nobuyoshi, Imamura, Yusuke, Furihata, Tomomi, Ando, Keisuke, Higuchi, Kosuke, Xu, Minhui, Sazuka, Tomokazu, Nakamura, Kazuyoshi, Kaneda, Atsushi, Kanai, Yoshikatsu, Kyprianou, Natasha, Ikehara, Yuzuru, Anzai, Naohiko, and Ichikawa, Tomohiko

Published
2020
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37. A prospective randomized study of multimodal analgesia combined with single injection transversus abdominis plane block versus epidural analgesia against postoperative pain after laparoscopic colon cancer surgery.

Author

Kitagawa, Hiroshi, Manabe, Tatsuya, Yamada, Yasutaka, Sato, Hirofumi, Takesue, Shin, Hiraki, Masatsugu, Kawaguchi, Atsushi, Sakaguchi, Yoshiro, and Noshiro, Hirokazu

Subjects
TRANSVERSUS abdominis muscle, COMBINED modality therapy, COLON cancer, EPIDURAL analgesia, POSTOPERATIVE pain treatment, ANALGESIA, ONCOLOGIC surgery
Abstract

Purpose: Transversus abdominis plane (TAP) block is a safe, effective, and promising analgesic procedure, but TAP block only cannot overcome postoperative pain. We conducted a prospective randomized study to evaluate postoperative pain control using multimodal analgesia (MA) combined with a single injection TAP block compared with epidural analgesia (EA) after laparoscopic colon cancer surgery. Methods: Sixty-seven patients scheduled for elective laparoscopic colon cancer surgery were enrolled in this study and randomized into EA and MA groups. The primary endpoint was the frequency of additional analgesic use until postoperative day (POD) 2. The VAS score, blood pressure, time to bowel movement, time to mobilization, postoperative complications, and length of hospital stay were also compared between the two groups. Results: Sixty-four patients (EA group, n = 33; MA group, n = 31) were analyzed. The patient characteristics did not differ markedly between the two groups. The frequency of additional analgesic use was significantly lower in the MA group than in the EA group (P < 0.001), whereas the VAS score did not differ markedly between the two groups. The postoperative blood pressure on the day of surgery was significantly lower in the MA group than in the EA group (P = 0.016), whereas urinary retention was significantly higher in the EA group than in the MA group (P < 0.001). Conclusion: MA combined with a single injection TAP block after laparoscopic colon cancer surgery may be comparable to EA in terms of analgesia and superior to EA in terms of urinary retention. [ABSTRACT FROM AUTHOR]

Published
2023
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45. GPR43 Suppresses Intestinal Tumor Growth by Modification of the Mammalian Target of Rapamycin Complex 1 Activity in Apc(Min/+) Mice

Author

Kong, Lingling, Hoshi, Namiko, Sui, Yunlong, Yamada, Yasutaka, Yoshida, Ryutaro, Ooi, Makoto, Tian, Zibin, Kimura, Ikuo, and Kodama, Yuzo

Subjects
FFAR2, Short-chain fatty acids, Mammalian target of rapamycin, G protein-coupled receptor 43, Colon cancer
Abstract

Objective: G protein-coupled receptor 43 (GPR43), a receptor for short-chain fatty acids, plays a role in suppressing tumor growth; however, the detailed underlying mechanism needs to be comprehensively elucidated. In this study, we investigated the role of GPR43 in inhibiting tumor growth using ApcMin/+, a murine model of intestinal tumors. Materials and Methods: Using GPR43−/− ApcMin/+ and GPR43+/− ApcMin/+ mice, the number of tumors was analyzed at the end of the experimental period. Immunohistochemistry, quantitative polymerase chain reaction, and Western blotting were performed to analyze cellular proliferation and proliferation-associated signal pathways. Results: Our results revealed that GPR43 deficiency resulted in increased tumor numbers in ApcMin/+ mice. Ki67 was highly expressed in GPR43−/− mice (p > 0.05). Increased expression levels of proinflammatory cytokines, including interleukin-6 and tumor necrosis factor-α, and amino acid transporters were not observed in GPR43-deficient mice compared to GPR43-sufficient mice. Furthermore, GPR43-deficient tumor tissues showed enhanced mammalian target of rapamycin-mediated phosphorylated ribosomal protein S6 kinase beta-1 (p > 0.05) and phosphorylated eukaryotic translation initiation factor 4E-binding protein 1 (p > 0.05), but not Akt (protein kinase B) phosphorylation (p = 0.7088). Conclusion: Collectively, GPR43 affords protection against tumor growth at least partly through inhibition of the mammalian target of rapamycin complex 1 pathway.

Published
2022

49. Tumor localization by Prostate Imaging and Reporting and Data System (PI-RADS) version 2.1 predicts the prognosis of prostate cancer after radical prostatectomy

Author

Fujimoto, Ayumi, primary, Sakamoto, Shinichi, additional, Horikoshi, Takuro, additional, Zhao, Xue, additional, Yamada, Yasutaka, additional, Rii, Junryo, additional, Takeuchi, Nobuyoshi, additional, Imamura, Yusuke, additional, Sazuka, Tomokazu, additional, Matsusaka, Keisuke, additional, Ikeda, Junichiro, additional, and Ichikawa, Tomohiko, additional

Published
2023
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50. Tumor Location and a Tumor Volume over 2.8 cc Predict the Prognosis for Japanese Localized Prostate Cancer

Author

Baba, Haruki, primary, Sakamoto, Shinichi, additional, Zhao, Xue, additional, Yamada, Yasutaka, additional, Rii, Junryo, additional, Fujimoto, Ayumi, additional, Kanesaka, Manato, additional, Takeuchi, Nobuyoshi, additional, Sazuka, Tomokazu, additional, Imamura, Yusuke, additional, Akakura, Koichiro, additional, and Ichikawa, Tomohiko, additional

Published
2022
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