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186 results on '"Yadav, Vinita"'

3. Informalized Workforce of Women Domestic Workers: Case of Bengaluru Metropolitan Region

Author

Yadav, Vinita, Jacob, Sherin, Dahiya, Bharat, Series Editor, Kirby, Andrew, Editorial Board Member, Friedberg, Erhard, Editorial Board Member, Singh, Rana P. B., Editorial Board Member, Yu, Kongjian, Editorial Board Member, El Sioufi, Mohamed, Editorial Board Member, Campbell, Tim, Editorial Board Member, Hayashi, Yoshitsugu, Editorial Board Member, Bai, Xuemei, Editorial Board Member, Haase, Dagmar, Editorial Board Member, Arimah, Ben C., Editorial Board Member, and Cirella, Giuseppe T., editor

Published
2023
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4. Adaptive Capacity Analysis of Flood Prone Regions in Bihar, India

Author

Yadav, Vinita, Ibrar, Zeeshan, Dahiya, Bharat, Series Editor, Kirby, Andrew, Editorial Board Member, Friedberg, Erhard, Editorial Board Member, Singh, Rana P. B., Editorial Board Member, Yu, Kongjian, Editorial Board Member, El Sioufi, Mohamed, Editorial Board Member, Campbell, Tim, Editorial Board Member, Hayashi, Yoshitsugu, Editorial Board Member, Bai, Xuemei, Editorial Board Member, Haase, Dagmar, Editorial Board Member, Arimah, Ben C., Editorial Board Member, de Pascale, Francesco, editor, De Pietro, Orlando, editor, Farabollini, Piero, editor, Lugeri, Francesca Romana, editor, and Mercatanti, Leonardo, editor

Published
2023
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8. Assessing the State of Homeless People to Plan Inclusive Smart Regions

Author

Yadav, Vinita, Farnaz Arefian, Fatemeh, Editorial Board Member, Batty, Michael, Editorial Board Member, Davoudi, Simin, Editorial Board Member, DeVerteuil, Geoffrey, Editorial Board Member, Kirby, Andrew, Editorial Board Member, Kropf, Karl, Editorial Board Member, Lucas, Karen, Editorial Board Member, Maretto, Marco, Editorial Board Member, Neuhaus, Fabian, Editorial Board Member, Nijhuis, Steffen, Editorial Board Member, Aráujo de Oliveira, Vitor Manuel, Editorial Board Member, Silver, Christopher, Editorial Board Member, Strappa, Giuseppe, Editorial Board Member, Vojnovic, Igor, Editorial Board Member, Whitehand, Jeremy W. R., Editorial Board Member, Yamu, Claudia, Editorial Board Member, and Sharma, Poonam, editor

Published
2021
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17. List of contributors

Author

Arora, Vinay, primary, Balaraman, Ekambaram, additional, Daw, Prosenjit, additional, Kumar, Akshai, additional, Kundu, Sabuj, additional, Mohanty, Aisa, additional, Nandi, Pran Gobinda, additional, Paul, Bhaskar, additional, Sharma, Raju, additional, Shee, Sujan, additional, Sivakumar, Ganesan, additional, Yadav, Vinita, additional, and Yasmin, Eileen, additional

Published
2022
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20. Study of tear function before and after laser-assisted in-situ keratomileusis

Author

Singh, Smita, Shukla, Ajay, Garkal, Priyanka, and Yadav, Vinita

Subjects
Dry eye syndromes -- Risk factors, Laser in situ keratomileusis -- Complications and side effects, Membranes (Biology) -- Health aspects, Health
Abstract

Byline: Smita. Singh, Ajay. Shukla, Priyanka. Garkal, Vinita. Yadav Purpose: This work assesses the change in tear function after LASIK surgery. Methods: This prospective, observational study was conducted in the [...]

Published
2023

22. ENTPD-1 disrupts inflammasome IL-1[beta]-driven venous thrombosis

Author

Yadav, Vinita, Chi, Liguo, Zhao, Raymond, Tourdot, Benjamin E., Yalavarthi, Srilakshmi, Jacobs, Benjamin N., Banka, Alison, Liao, Hui, Koonse, Sharon, Anyanwu, Anuli C., Visovatti, Scott H., Holinstat, Michael A., Kahlenberg, J. Michelle, Knight, Jason S., Pinsky, David J., and Kanthi, Yogendra

Subjects
Fibrin -- Analysis -- Health aspects, Inflammation -- Analysis -- Health aspects, Enzymes -- Analysis -- Health aspects, Thrombophlebitis -- Analysis -- Health aspects, Anakinra -- Analysis -- Health aspects, Stenosis, Hydrolysis, Thrombosis, Nucleotides, Homeostasis, Bonds (Securities), Endothelium, Antibodies, Health care industry
Abstract

Deep vein thrombosis (DVT), caused by alterations in venous homeostasis, is the third most common cause of cardiovascular mortality, however, key molecular determinants in venous thrombosis have not been fully elucidated. Several lines of evidence indicate that DVT occurs at the intersection of dysregulated inflammation and coagulation. The enzyme ectonucleoside tri(di)phosphohydrolase (ENTPD1, also known as CD39) is a vascular ecto-apyrase on the surface of leukocytes and the endothelium that inhibits intravascular inflammation and thrombosis by hydrolysis of phosphodiester bonds from nucleotides released by activated cells. Here, we evaluated the contribution of CD39 to venous thrombosis in a restricted-flow model of murine inferior vena cava stenosis. CD39 deficiency conferred a greater than 2-fold increase in venous thrombogenesis, characterized by increased leukocyte engagement, neutrophil extracellular trap formation, fibrin, and local activation of tissue factor in the thrombotic milieu. This venous thrombogenesis was orchestrated by increased phosphorylation of the p65 subunit of NF-[kappa]B, activation of the NLR family pyrin domain-containing 3 (NLRP3) inflammasome, and IL-1[beta] release in CD39-deficient mice. Substantiating these findings, an IL-1[beta]-neutralizing antibody or the IL-1 receptor inhibitor anakinra attenuated the thrombosis risk in CD39- deficient mice. These data demonstrate that IL-1[beta] is a key accelerant of venous thrombo-inflammation, which can be suppressed by CD39. CD39 inhibits in vivo crosstalk between inflammation and coagulation pathways and is a critical vascular checkpoint in venous thrombosis., Introduction The cell-surface enzyme ectonucleoside tri(di)phosphohydrolase-1 (ENTPD1), also known as CD39), is a key molecular factor protecting blood fluidity at the vessel-wall interface by catalyzing the hydrolytic cleavage of phosphodiester [...]

Published
2019
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31. Mucoepidermoid Carcinoma of Trachea: A Rare Entity

Author

Chawla, Rakesh K., primary, Chawla, Aditya K., additional, Arora, Harsh Nandini, additional, Chauhan, Avinash, additional, Chawla, Primal, additional, Chawla, Madhav K., additional, Yadav, Vinita, additional, and Banerjee, Sumi, additional

Published
2022
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46. Prothrombotic autoantibodies in serum from hospitalized patients with COVID-19

Author

Zuo, Yu, Estes, Shanea K., Ali, Ramadan A., Gandhi, Alex A., Yalavarthi, Srilakshmi, Shi, Hui, Sule, Gautam, Gockman, Kelsey, Madison, Jacqueline A., Zuo, Melanie, Yadav, Vinita, Wang, Jintao, Woodward, Wrenn, Lezak, Sean P., Lugogo, Njira L., Smith, Stephanie A., Morrissey, James H., Kanthi, Yogendra, and Knight, Jason S.

Subjects
immune system diseases, Article
Abstract

Patients with coronavirus disease 19 (COVID-19) are at high risk for thrombotic arterial and venous occlusions. Lung histopathology often reveals fibrin-based occlusions in the small blood vessels of patients who succumb to the disease. Antiphospholipid syndrome is an acquired and potentially life-threatening thrombophilia in which patients develop pathogenic autoantibodies targeting phospholipids and phospholipid-binding proteins (aPL antibodies). Case series have recently detected aPL antibodies in patients with COVID-19. Here, we measured eight types of aPL antibodies in serum samples from 172 patients hospitalized with COVID-19. These included anticardiolipin IgG, IgM and IgA; anti-β2 glycoprotein I IgG, IgM, and IgA; and anti- phosphatidylserine/prothrombin (aPS/PT) IgG and IgM. We detected aPS/PT IgG in 24% of serum samples, anticardiolipin IgM in 23% of serum samples, and aPS/PT IgM in 18% of serum samples. Antiphospholipid autoantibodies were present in 52% of serum samples using the manufacturer’s threshold and in 30% using a more stringent cutoff (≥40 ELISA-specific units). Higher titers of aPL antibodies were associated with neutrophil hyperactivity including the release of neutrophil extracellular traps (NETs), higher platelet counts, more severe respiratory disease, and lower glomerular filtration rate. Similar to IgG from patients with antiphospholipid syndrome, IgG fractions isolated from patients with COVID-19 promoted NET release from neutrophils isolated from healthy individuals. Furthermore, injection of IgG purified from the serum of COVID-19 patients into mice accelerated venous thrombosis. Taken together, these findings suggest that half of patients hospitalized with COVID-19 become at least transiently positive for aPL antibodies and that these autoantibodies are potentially pathogenic.

Published
2020

48. Prothrombotic autoantibodies in serum from patients hospitalized with COVID-19

Author

Zuo, Yu, primary, Estes, Shanea K., additional, Ali, Ramadan A., additional, Gandhi, Alex A., additional, Yalavarthi, Srilakshmi, additional, Shi, Hui, additional, Sule, Gautam, additional, Gockman, Kelsey, additional, Madison, Jacqueline A., additional, Zuo, Melanie, additional, Yadav, Vinita, additional, Wang, Jintao, additional, Woodard, Wrenn, additional, Lezak, Sean P., additional, Lugogo, Njira L., additional, Smith, Stephanie A., additional, Morrissey, James H., additional, Kanthi, Yogendra, additional, and Knight, Jason S., additional

Published
2020
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