17 results on '"Xu, Bao-yan"'
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2. Rifampicin induces clathrin-dependent endocytosis and ubiquitin–proteasome degradation of MRP2 via oxidative stress-activated PKC-ERK/JNK/p38 and PI3K signaling pathways in HepG2 cells
3. The neutrophil‐to‐lymphocyte ratio represents a systemic inflammation marker and reflects the relationship with 90‐day mortality in non‐cirrhotic chronic severe hepatitis
4. Genome-wide microarray-based analysis of miRNAs expression in patients with acute-on-chronic liver failure
5. Efficacy of hyperbaric oxygen combined with escitalopram in depression and its effect on cognitive function
6. Efficacy of short-term dexamethasone therapy in acute-on-chronic pre-liver failure
7. Prevalence and clinical significance of portal vein thrombosis in cirrhotic patients with acute decompensation
8. Rifampicin induces clathrin-dependent endocytosis and ubiquitin–proteasome degradation of MRP2 via oxidative stress-activated PKC-ERK/JNK/p38 and PI3K signaling pathways in HepG2 cells
9. PS-104-Prognosis assessment of acute-on-chronic liver failure in patients with hepatitis B virus-related cirrhosis requires specific criteria
10. Acute-on-Chronic Liver Failure in China: Rationale for Developing a Patient Registry and Baseline Characteristics
11. Serum Testosterone Levels and Androgen Receptor CAG Polymorphism Correlate with Hepatitis B Virus (HBV)-Related Acute Liver Failure in Male HBV Carriers
12. Efficacy of short-term dexamethasone therapy in acute-on-chronic pre-liver failure
13. SAT145 - Prevalence and clinical significance of portal vein thrombosis in cirrhotic patients with acute decompensation.
14. Plasma Exchange-Based Non-bioartificial Liver Support System Improves the Short-Term Outcomes of Patients With Hepatitis B Virus-Associated Acute-on-Chronic Liver Failure: A Multicenter Prospective Cohort Study.
15. [The amplification and high efficient cloning of the whole genome sequence of hepatitis B virus].
16. [Difference analysis of genome-wide DNA methylation status of CpG islands between the monozygotic twins with disconcordant phenotypes of chronic hepatitis B virus infection].
17. [The primary comparative analysis between the host genetic factors and their relationships with clinical phenotype of HBV infected twins].
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