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3. Disease mutations and phosphorylation alter the allosteric pathways involved in autoinhibition of protein phosphatase 2A.

4. Inherited duplications of PPP2R3B predispose to nevi and melanoma via a C21orf91-driven proliferative phenotype

5. B56δ long-disordered arms form a dynamic PP2A regulation interface coupled with global allostery and Jordan’s syndrome mutations

8. Extended regulation interface coupled to the allosteric network and disease mutations in the PP2A-B56δ holoenzyme

15. Coupling to short linear motifs creates versatile PME-1 activities in PP2A holoenzyme demethylation and inhibition

16. Small-molecule inhibitors that disrupt the MTDH–SND1 complex suppress breast cancer progression and metastasis

19. Structural Mechanism of Demethylation and Inactivation of Protein Phosphatase 2A

20. Generation of an Allelic Series at the Ahr Locus Using an Edited Recombinant Approach

23. Inherited duplications ofPPP2R3Bpromote naevi and melanoma via a novelC21orf91-driven proliferative phenotype

26. Trace derivatives of kynurenine potently activate the aryl hydrocarbon receptor (AHR)

27. Methylation-regulated decommissioning of multimeric PP2A complexes

28. PP2A-B' holoenzyme substrate recognition, regulation and role in cytokinesis

29. PP2A-B′ holoenzyme substrate recognition, regulation and role in cytokinesis

33. MTDH-SND1 Interaction Is Crucial for Expansion and Activity of Tumor-Initiating Cells in Diverse Oncogene- and Carcinogen-Induced Mammary Tumors

48. Structure of the Protein Phosphatase 2A Holoenzyme

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