155 results on '"Wu, Yee Ling"'
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2. Lung-resident memory B cells maintain allergic IgE responses in the respiratory tract
3. B cell intrinsic Toll-like receptor 9 signaling regulates class switching to IgE
4. An in vivo overexpression system to study the functions of complement activated products in a mouse model of lupus
5. Allergen inhalation transforms the lungs into a permissive environment for local IgE responses
6. Complement C4 gene copy number variations and polymorphisms, autoantibodies, and clinical manifestations of juvenile dermatomyositis– a multi-center study
7. Complement in Rheumatic Diseases
8. Low copy numbers of complementC4andC4Adeficiency are risk factors for myositis, its subgroups and autoantibodies
9. Three Distinct Profiles of Serum Complement C4 Proteins in Pediatric Systemic Lupus Erythematosus (SLE) Patients: Tight Associations of Complement C4 and C3 Protein Levels in SLE but not in Healthy Subjects
10. Complement Components C4A and C4B in Human Lupus
11. Lung memory B cells promote local IgE production in the respiratory tract
12. Strong Association of Combined Genetic Deficiencies in the Classical Complement Pathway With Risk of Systemic Lupus Erythematosus and Primary Sjögren's Syndrome
13. Sex-specific association of X-linked Toll-like receptor 7 (TLR7) with male systemic lupus erythematosus
14. Loop extrusion promotes an alternate pathway for isotype switching
15. Molecular fine-tuning of affinity maturation in germinal centers
16. Human Complement C4B Allotypes and Deficiencies in Selected Cases With Autoimmune Diseases
17. Great genotypic and phenotypic diversities associated with copy-number variations of complement C4 and RP-C4-CYP21-TNX (RCCX) modules: A comparison of Asian-Indian and European American populations
18. The HLA 8.1 ancestral haplotype is strongly linked to the C allele of −429T > C promoter polymorphism of receptor of the advanced glycation endproduct (RAGE) gene. Haplotype-independent association of the −429C allele with high hemoglobin A1C levels in diabetic patients
19. Gene CNVs and protein levels of complement C4A and C4B as novel biomarkers for partial disease remissions in new-onset type 1 diabetes patients
20. Increased frequency of complement C4B deficiency in rheumatoid arthritis
21. Low Complement C4 Levels in Juvenile Dermatomyositis (JDM) are Strongly Associated with C4A NULL Alleles
22. Toll-like receptor signaling directly modulates B cell antibody responses
23. Complement Deficiencies in Human Systemic Lupus Erythematosus (SLE) and SLE Nephritis: Epidemiology and Pathogenesis
24. Assessment of Complement C4 Gene Copy Number Using the Paralog Ratio Test
25. The HLA 8.1 ancestral haplotype is strongly linked to the C allele of −429T > C promoter polymorphism of receptor of the advanced glycation endproduct (RAGE) gene. Haplotype-independent association of the −429C allele with high hemoglobinA1C levels in diabetic patients
26. COMPLEX DIVERSITIES OF COMPLEMENT C4 AND RP-C4-CYP21-TNX (RCCX) MODULES IN CAUCASIANS, AFRICANS, CHINESE AND INDIANS
27. Elevated serum complement levels and higher gene copy number of complement C4B are associated with hypertension and effective response to statin therapy in childhood-onset systemic lupus erythematosus (SLE)
28. Opposite Profiles of Complement in Antiphospholipid Syndrome (APS) and Systemic Lupus Erythematosus (SLE) Among Patients With Antiphospholipid Antibodies (aPL)
29. Direct activation of Toll-like receptors induce differential B cell responses
30. 190 Opposite profiles of complement in antiphospholipid syndrome (APS) and systemic lupus erythematosus (SLE) among patients with antiphospholipid antibodies
31. SLE Comprises Four Immune-Phenotypes, Which Differ Regarding HLA-DRB1 and Clinical Associations
32. Effects of Complement C4 Gene Copy Number Variations, Size Dichotomy, and C4A Deficiency on Genetic Risk and Clinical Presentation of Systemic Lupus Erythematosus in East Asian Populations
33. Intrinsic transcriptional heterogeneity in B cells controls early class switching to IgE
34. Asynchronous digital activation of the germline Iγ1 and Iɛ promoters in single cells reveals that temporal control determines the fate of switching to IgE in B cells
35. Effects of Complement C4 Gene Copy-Number Variations, Gene Size Dichotomy and C4A-Deficiency on Genetic Risk and Clinical Presentation of East-Asian and European Systemic Lupus Erythematosus (SLE)
36. Early Components of the Complement Classical Activation Pathway in Human Systemic Autoimmune Diseases
37. Molecular fine-tuning of affinity maturation in germinal centers
38. Gene Copy-Number Variations (CNVs) and Protein Levels of Complement C4A and C4B as Novel Biomarkers for Partial Disease Remissions in New-Onset Type 1 Diabetes Patients
39. Gene copy-number variations (CNVs) of complement C4 and C4A deficiency in genetic risk and pathogenesis of juvenile dermatomyositis
40. Three Distinct Profiles of Serum Complement C4 Proteins in Pediatric Systemic Lupus Erythematosus (SLE) Patients: Tight Associations of Complement C4 and C3 Protein Levels in SLE but not in Healthy Subjects
41. Active RNAP pre-initiation sites are highly mutated by cytidine deaminases in yeast, with AID targeting small RNA genes
42. Author response: Active RNAP pre-initiation sites are highly mutated by cytidine deaminases in yeast, with AID targeting small RNA genes
43. DNA deaminases induce break-associated mutation showers with implication of APOBEC3B and 3A in breast cancer kataegis
44. Abstract 611: AID/APOBEC cytidine deaminases can mimic the phenomenon of localized hypermutation in cancer orkataegis.
45. Author response: DNA deaminases induce break-associated mutation showers with implication of APOBEC3B and 3A in breast cancer kataegis
46. Gene copy-number variations (CNVs) of complement C4 and C4A deficiency in genetic risk and pathogenesis of juvenile dermatomyositis.
47. Intrinsic transcriptional heterogeneity in B cells controls early class switching to IgE
48. Gene CNVs and protein levels of complement C4A and C4B as novel biomarkers for partial disease remissions in new-onset type 1 diabetes patients
49. Molecular basis of complement C1r deficiency in a male African American patient with systemic lupus erythematosus
50. Low gene copy-number of complement C4A, the presence of HLA-DR3, and the presence of HLA-DR2 are independent and additive risk factors for human systemic lupus erythematosus
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