189 results on '"Wozniak, Eva"'
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2. [11C]metomidate PET-CT versus adrenal vein sampling for diagnosing surgically curable primary aldosteronism: a prospective, within-patient trial
3. Single cell sequencing data identify distinct B cell and fibroblast populations in stricturing Crohn's disease
4. Somatic mutations of GNA11 and GNAQ in CTNNB1-mutant aldosterone-producing adenomas presenting in puberty, pregnancy or menopause
5. Transcriptomic profiling reveals a pronociceptive role for angiotensin II in inflammatory bowel disease.
6. Common variants at 19p13 are associated with susceptibility to ovarian cancer (vol 42, pg 880, 2010)
7. Corrigendum: Common variants at 19p13 are associated with susceptibility to ovarian cancer.
8. Neuronal let-7b-5p acts through the Hippo-YAP pathway in neonatal encephalopathy
9. The genomic loci of specific human tRNA genes exhibit ageing-related DNA hypermethylation
10. FRI144 Using Single Cell Technology To Predict The Cell Of Origin Of Aldosterone-producing Adrenal Adenomas
11. OR02-02 Pre-operative Blood Pressure Response To Aldosterone Antagonists And Urinary Hybrid Steroid Ratios Predict Clinical Outcomes In Unilateral Primary Aldosteronism For At Least 2 Years Post-adrenalectomy
12. Single cell sequencing data identify distinct B cell and fibroblast populations in stricturing Crohns disease
13. Loss of Trem2 in microglia leads to widespread disruption of cell coexpression networks in mouse brain
14. Comparison of transcriptome responses to glyphosate, isoxaflutole, quizalofop-p-ethyl and mesotrione in the HepaRG cell line
15. Epigenetic analysis leads to identification of HNF1B as a subtype-specific susceptibility gene for ovarian cancer
16. Common variants at 19p13 are associated with susceptibility to ovarian cancer.
17. Transcriptomic profiling reveals a pro-nociceptive role for Angiotensin II in inflammatory bowel disease
18. Genotype-independent association between vitamin D deficiency and polycystic ovarian syndrome in Lahore, Pakistan
19. Integrated transcriptomics and metabolomics reveal signatures of lipid metabolism dysregulation in HepaRG liver cells exposed to PCB 126
20. ADAMS project: a genetic Association study in individuals from Diverse Ancestral backgrounds with Multiple Sclerosis based in the UK
21. Supplementary Tables 1-9 from Biomarker-Based Ovarian Carcinoma Typing: A Histologic Investigation in the Ovarian Tumor Tissue Analysis Consortium
22. Supplementary Figure Legend from Biomarker-Based Ovarian Carcinoma Typing: A Histologic Investigation in the Ovarian Tumor Tissue Analysis Consortium
23. Supplementary Figure 1 from Biomarker-Based Ovarian Carcinoma Typing: A Histologic Investigation in the Ovarian Tumor Tissue Analysis Consortium
24. Data from Biomarker-Based Ovarian Carcinoma Typing: A Histologic Investigation in the Ovarian Tumor Tissue Analysis Consortium
25. Supplementary Table 1 from Association Study of Prostate Cancer Susceptibility Variants with Risks of Invasive Ovarian, Breast, and Colorectal Cancer
26. Data from Association Study of Prostate Cancer Susceptibility Variants with Risks of Invasive Ovarian, Breast, and Colorectal Cancer
27. Deep palmar phenotyping in atopic eczema: patterns associated with Filaggrin variants, disease severity and barrier function in a South Asian population
28. Disease evolution and outcomes in familial AML with germline CEBPA mutations
29. Complete clinical cure of primary aldosteronism (PA) is predictable and sustained for at least two years
30. DNA methylation profiles between airway epithelium and proxy tissues in children
31. Hormone-receptor expression and ovarian cancer survival: an Ovarian Tumor Tissue Analysis consortium study
32. Recruitment of newly diagnosed ovarian cancer patients proved challenging in a multicentre biobanking study
33. 11C-metomidate PET CT versus Adrenal Vein Sampling for diagnosing surgically curable primary aldosteronism: prospective test validation, and impact of somatic genotype and ethnicity on outcomes
34. Somatic mutations of GNA11 and GNAQ in CTNNB1-mutant aldosterone-producing adenomas presenting in puberty, pregnancy or menopause.
35. Negligible impact of rare autoimmune-locus coding-region variants on missing heritability
36. TREM2 impacts brain microglia, oligodendrocytes and endothelial co-expression modules revealing genes and pathways important in Alzheimer’s disease
37. BS18 Profiling endothelial gene expression in coronary atherosclerotic plaques in a human-like D374Y-PCSK9 hyperlipidaemic porcine model
38. Transcriptomic Profiling to Understand Inhibitor Development in Previously Untreated Patients with Severe Hemophilia a
39. Somatic mutations of CADM1in aldosterone-producing adenomas and gap junction-dependent regulation of aldosterone production
40. Somatic Mutations of GNA11 and GNAQ in CTNNB1-Mutant Aldosterone-Producing Adenomas Increases Aldosterone and Aldosterone Synthase (CYP11B2)
41. CTNNB1-Mutant Aldosterone-Producing Adenomas With Somatic Mutations of GNA11/GNAQ Have Distinct Phenotype and Genotype
42. Novel mechanism of action for neuronal let-7b-5p in neonatal encephalopathy through the Hippo pathway
43. Common alleles in candidate susceptibility genes associated with risk and development of epithelial ovarian cancer
44. OR34-02 Somatic Transmembrane Domain Mutations of a Cell Adhesion Molecule, CADM1, Cause Primary Aldosteronism by Preventing Gap Junction Communication Between Adrenocortical Cells
45. The Genomic Loci of Specific Human tRNA Genes Exhibit Ageing-Related DNA Hypermethylation
46. Somatic transmembrane domain mutations of a cell adhesion molecule, CADM1, cause primary aldosteronism by preventing gap junction communication between adrenocortical cells
47. Quizalofop-p-Ethyl Induces Adipogenesis in 3T3-L1 Adipocytes
48. Somatic mutations of GNA11and GNAQin CTNNB1-mutant aldosterone-producing adenomas presenting in puberty, pregnancy or menopause
49. Integrated transcriptomics and metabolomics reveal signatures of lipid metabolism dysregulation in HepaRG liver cells exposed to PCB 126
50. Loss of Trem2 in microglia leads to widespread disruption of cell co-expression networks in mouse brain
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