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15 results on '"Williams, Laurina O."'

9. Genetically Characterized Positive Control Cell Lines Derived from Residual Clinical Blood Samples

Author

Bernacki, Susan H, primary, Beck, Jeanne C, primary, Stankovic, Ana K, primary, Williams, Laurina O, primary, Amos, Jean, primary, Snow-Bailey, Karen, primary, Farkas, Daniel H, primary, Friez, Michael J, primary, Hantash, Feras M, primary, Matteson, Karla J, primary, Monaghan, Kristin G, primary, Muralidharan, Kasinathan, primary, Pratt, Victoria M, primary, Prior, Thomas W, primary, Richie, Kristy L, primary, Levin, Barbara C, primary, Rohlfs, Elizabeth M, primary, Schaefer, Frederick V, primary, Shrimpton, Antony E, primary, Spector, Elaine B, primary, Stolle, Catherine A, primary, Strom, Charles M, primary, Thibodeau, Stephen N, primary, Cole, Eugene C, primary, Goodman, Barbara K, primary, and Stenzel, Timothy T, primary

Published
2005
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10. Developing a Sustainable Process to Provide Quality Control Materials for Genetic Testing

Author

Chen, Bin, primary, O'Connell, Catherine D., additional, Boone, D Joe, additional, Amos, Jean A., additional, Beck, Jeanne C., additional, Chan, Maria M., additional, Farkas, Daniel H., additional, Lebo, Roger V., additional, Richards, Carolyn Sue, additional, Roa, Benjamin B., additional, Silverman, Lawrence M., additional, Barton, David E., additional, Bejjani, Bassem A., additional, Belloni, Dorothy R., additional, Bernacki, Susan H., additional, Caggana, Michele, additional, Charache, Patricia, additional, Dequeker, Elisabeth, additional, Ferreira-Gonzalez, Andrea, additional, Friedman, Kenneth J., additional, Greene, Carol L., additional, Grody, Wayne W., additional, Highsmith, William Edward, additional, Hinkel, Cecelia S., additional, Kalman, Lisa V., additional, Lubin, Ira M., additional, Lyon, Elaine, additional, Payne, Deborah A., additional, Pratt, Victoria M., additional, Rohlfs, Elizabeth, additional, Rundell, Clark A., additional, Schneider, Erasmus, additional, Willey, Ann M., additional, Williams, Laurina O., additional, Willey, James C., additional, Winn-Deen, Emily S., additional, and Wolff, Daynna J., additional

Published
2005
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12. Laboratory performance in HTLV-I/II analysis

Author

Williams, Laurina O., primary, Blumer, Sharon O., additional, Schalla, William O., additional, Robinson, Pamela H., additional, Handsfield, James H., additional, Fehd, Ronald J., additional, Hancock, John S., additional, Lipman, Harvey B., additional, and Hearn, Thomas L., additional

Published
2000
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13. Is Maternal Age a Risk Factor for Mental Retardation among Children?

Author

Williams, Laurina O. and Decouflé, Pierre

Subjects
*MATERNAL age, *INTELLECTUAL disabilities, *PEDIATRIC epidemiology, *COGNITION disorders in children, *DEVELOPMENTAL disabilities, *CENTRAL nervous system abnormalities, *DOWN syndrome, *SOCIODEMOGRAPHIC factors, *DISEASE risk factors, *DISABILITIES
Abstract

The purpose of this study was to determine whether older or very young maternal age at delivery is associated with mental retardation in children. Ten-year-old children with mental retardation (an intelligence quotient of 70 or less) were identified in 1985–1987 from multiple sources in the metropolitan Atlanta, Georgia, area. These children were subdivided into two case groups according to whether they had concomitant developmental disabilities or birth defects affecting the central nervous system (codevelopmental retardation) or did not have such disabilities (isolated retardation). Control children were randomly chosen from the regular education files of the public school systems in the study area. Data on sociodemographic variables were gathered from birth certificates. Children of teenaged mothers were not at increased risk for either form of retardation and children of mothers aged ⋛30 years were not at increased risk for isolated retardation, in comparison with children of mothers aged 20–29 years. A markedly elevated risk of codevelopmental retardation was seen among black children of mothers aged ⋛30 years that was not attributable to Down syndrome. A modest increase in risk for codevelopmental retardation was observed among white children born to older mothers, but it was entirely due to Down syndrome. Am J Epidemiol 1999;149:814–23. [ABSTRACT FROM PUBLISHER]

Published
2009

14. KPC-4 Is Encoded within a Truncated Tn4401in an IncL/M Plasmid, pNE1280, Isolated from Enterobacter cloacaeand Serratia marcescens

Author

Bryant, Kendall A., Van Schooneveld, Trevor C., Thapa, Ishwor, Bastola, Dhundy, Williams, Laurina O., Safranek, Thomas J., Hinrichs, Steven H., Rupp, Mark E., and Fey, Paul D.

Abstract

ABSTRACTWe describe the transfer of blaKPC-4from Enterobacter cloacaeto Serratia marcescensin a single patient. DNA sequencing revealed that KPC-4 was encoded on an IncL/M plasmid, pNE1280, closely related to pCTX-M360. Further analysis found that KPC-4 was encoded within a novel Tn4401element (Tn4401f) containing a truncated tnpAand lacking tnpR, ISKpn7left, and Tn4401IRL-1, which are conserved in other Tn4401transposons. This study highlights the continued evolution of Tn4401transposons and movement to multiple plasmid backbones that results in acquisition by multiple species of Gram-negative bacilli.

Published
2012
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15. KPC-4 Is encoded within a truncated Tn4401 in an IncL/M plasmid, pNE1280, isolated from Enterobacter cloacae and Serratia marcescens.

Author

Bryant KA, Van Schooneveld TC, Thapa I, Bastola D, Williams LO, Safranek TJ, Hinrichs SH, Rupp ME, and Fey PD

Subjects
Anti-Bacterial Agents pharmacology, Anti-Bacterial Agents therapeutic use, Coinfection, Enterobacter cloacae drug effects, Enterobacter cloacae isolation & purification, Enterobacteriaceae Infections drug therapy, Enterobacteriaceae Infections microbiology, Fatal Outcome, Female, Humans, Middle Aged, Sequence Analysis, DNA, Serratia Infections drug therapy, Serratia Infections microbiology, Serratia marcescens drug effects, Serratia marcescens isolation & purification, beta-Lactamases genetics, DNA Transposable Elements, Enterobacter cloacae genetics, Gene Transfer, Horizontal, Plasmids, Serratia marcescens genetics, beta-Lactam Resistance genetics
Abstract

We describe the transfer of bla(KPC-4) from Enterobacter cloacae to Serratia marcescens in a single patient. DNA sequencing revealed that KPC-4 was encoded on an IncL/M plasmid, pNE1280, closely related to pCTX-M360. Further analysis found that KPC-4 was encoded within a novel Tn4401 element (Tn4401f) containing a truncated tnpA and lacking tnpR, ISKpn7 left, and Tn4401 IRL-1, which are conserved in other Tn4401 transposons. This study highlights the continued evolution of Tn4401 transposons and movement to multiple plasmid backbones that results in acquisition by multiple species of Gram-negative bacilli.

Published
2013
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