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4. The Hubble Deep Field South Flanking Fields

Author

Lucas, Ray A., Baum, Stefi A., Brown, Thomas M., Casertano, Stefano, Conselice, Chris, de Mello, Duilia, Dickinson, Mark E., Ferguson, Henry C., Fruchter, Andrew S., Gardner, Jonathan P., Gilmore, Diane, Gonzalez-Lopezlira, Rosa A., Heyer, Inge, Hook, Richard N., Kaiser, Mary Elizabeth, Mack, Jennifer, Makidon, Russell, Martin, Crystal L., Mutchler, Max, Smith, T. Ed, Stiavelli, Massimo, Teplitz, Harry I., Wiggs, Michael S., Williams, Robert E., and Zurek, David R.

Subjects
Astrophysics
Abstract

As part of the Hubble Deep Field South program, a set of shorter 2-orbit observations were obtained of the area adjacent to the deep fields. The WFPC2 flanking fields cover a contiguous solid angle of 48 square arcminutes. Parallel observations with the STIS and NICMOS instruments produce a patchwork of additional fields with optical and near-infrared (1.6 micron) response. Deeper parallel exposures with WFPC2 and NICMOS were obtained when STIS observed the NICMOS deep field. These deeper fields are offset from the rest, and an extended low surface brightness object is visible in the deeper WFPC2 flanking field. In this data paper, which serves as an archival record of the project, we discuss the observations and data reduction, and present SExtractor source catalogs and number counts derived from the data. Number counts are broadly consistent with previous surveys from both ground and space. Among other things, these flanking field observations are useful for defining slit masks for spectroscopic follow-up over a wider area around the deep fields, for studying large-scale structure that extends beyond the deep fields, for future supernova searches, and for number counts and morphological studies, but their ultimate utility will be defined by the astronomical community., Comment: 46 pages, 15 figures. Images and full catalogs available via the HDF-S at http://www.stsci.edu/ftp/science/hdfsouth/hdfs.html at present. The paper is accepted for the February 2003 Astronomical Journal. Full versions of the catalogs will also be available on-line from AJ after publication

Published
2002
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5. WFPC2 Observations of the Hubble Deep Field-South

Author

Casertano, Stefano, de Mello, Duilia, Dickinson, Mark, Ferguson, Henry C., Fruchter, Andrew S., Gonzalez-Lopezlira, Rosa A., Heyer, Inge, Hook, Richard N., Levay, Zolt, Lucas, Ray A., Mack, Jennifer, Makidon, Russell B., Mutchler, Max, Smith, T. Ed, Stiavelli, Massimo, Wiggs, Michael S., and Williams, Robert E.

Subjects
Astrophysics
Abstract

The Hubble Deep Field-South observations targeted a high-galactic-latitude field near QSO J2233-606. We present WFPC2 observations of the field in four wide bandpasses centered at roughly 300, 450, 606, and 814 nm. Observations, data reduction procedures, and noise properties of the final images are discussed in detail. A catalog of sources is presented, and the number counts and color distributions of the galaxies are compared to a new catalog of the HDF-N that has been constructed in an identical manner. The two fields are qualitatively similar, with the galaxy number counts for the two fields agreeing to within 20%. The HDF-S has more candidate Lyman-break galaxies at z > 2 than the HDF-N. The star-formation rate per unit volume computed from the HDF-S, based on the UV luminosity of high-redshift candidates, is a factor of 1.9 higher than from the HDF-N at z ~ 2.7, and a factor of 1.3 higher at z ~ 4., Comment: 93 pages, 25 figures; contains very long tables

Published
2000
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6. Females display relatively preserved muscle quality compared with males during the onset and early stages of C26-induced cancer cachexia

Author

Cabrera, Ana Regina, primary, Deaver, J. William, additional, Lim, Seongkyun, additional, Morena da Silva, Francielly, additional, Schrems, Eleanor R., additional, Saling, Landen W., additional, Tsitkanou, Stavroula, additional, Rosa-Caldwell, Megan E., additional, Wiggs, Michael P., additional, Washington, Tyrone A., additional, and Greene, Nicholas P., additional

Published
2023
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9. Muscle weakness precedes atrophy during cancer cachexia and is linked to muscle-specific mitochondrial stress

Author

Delfinis, Luca J., primary, Bellissimo, Catherine A., additional, Gandhi, Shivam, additional, DiBenedetto, Sara N., additional, Garibotti, Madison C., additional, Thuhan, Arshdeep K., additional, Tsitkanou, Stavroula, additional, Rosa-Caldwell, Megan E., additional, Rahman, Fasih A., additional, Cheng, Arthur J., additional, Wiggs, Michael P., additional, Schlattner, Uwe, additional, Quadrilatero, Joe, additional, Greene, Nicholas P., additional, and Perry, Christopher G.R., additional

Published
2022
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19. Development of metabolic and contractile alterations in development of cancer cachexia in female tumor-bearing mice

Author

Lim, Seongkyun, primary, Deaver, J. William, additional, Rosa-Caldwell, Megan E., additional, Haynie, Wesley S., additional, Morena da Silva, Francielly, additional, Cabrera, Ana Regina, additional, Schrems, Eleanor R., additional, Saling, Landen W., additional, Jansen, Lisa T., additional, Dunlap, Kirsten R., additional, Wiggs, Michael P., additional, Washington, Tyrone A., additional, and Greene, Nicholas P., additional

Published
2022
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22. Muscle weakness precedes atrophy during cancer cachexia and is associated with muscle-specific mitochondrial stress

Author

Delfinis, Luca J, primary, Bellissimo, Catherine A, additional, Gandhi, Shivam, additional, DiBenedetto, Sara N, additional, Rosa-Caldwell, Megan E, additional, Rahman, Fasih A, additional, Wiggs, Michael P, additional, Schlattner, Uwe, additional, Quadrilatero, Joe, additional, Greene, Nicholas P, additional, and Perry, Christopher G R, additional

Published
2021
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23. Insulin resistance syndrome blunts the mitochondrial anabolic response following resistance exercise

Author

Nilsson, Mats I., Greene, Nicholas P., Dobson, Justin P., Wiggs, Michael P., Gasier, Heath G., Macias, Brandon R., Shimkus, Kevin L., and Fluckey, James D.

Subjects
Insulin resistance -- Physiological aspects, Insulin resistance -- Care and treatment, Mitochondria -- Properties, Exercise -- Physiological aspects, Exercise -- Research, Biological sciences
Abstract

Metabolic risk factors associated with insulin resistance syndrome may attenuate augmentations in skeletal muscle protein anabolism following contractile activity. The purpose of this study was to investigate whether or not the anabolic response, as defined by an increase in cumulative fractional protein synthesis rates (24-h FSR) following resistance exercise (RE), is blunted in skeletal muscle of a well-established rodent model of insulin resistance syndrome. Four-month-old lean (Fa/?) and obese (fa/fa) Zucker rats engaged in four lower body RE sessions over 8 days, with the last bout occurring 16 h prior to muscle harvest. A priming dose of deuterium oxide ([sup.2][H.sub.2]O) and [sup.2][H.sub.2]O -enriched drinking water were administered 24 h prior to euthanization for assessment of cumulative FSR. Fractional synthesis rates of mixed (-5%), mitochondrial (-1%), and cytosolic (+15%), but not myofibrillar, proteins (-16%, P = 0.012) were normal or elevated in gastrocnemius muscle of unexercised obese rats. No statistical differences were found in the anabolic response of cytosolic and myofibrillar subfractions between phenotypes, but obese rats were not able to augment 24-h FSR of mitochondria to the same extent as lean rats following RE (+14% vs. +28%, respectively). We conclude that the mature obese Zucker rat exhibits a mild, myofibrillar-specific suppression in basal FSR and a blunted mitochondrial response to contractile activity in mixed gastrocnemius muscle. These findings underscore the importance of assessing synthesis rates of specific myocellular subfractions to fully elucidate perturbations in basal protein turnover rates and differential adaptations to exercise stimuli in metabolic disease. skeletal muscle; protein synthesis; diabetes; [sup.2][H.sub.2]O doi: 10.1152/ajpendo.00118.2010.

Published
2010

25. A comparison of [sup.2][H.sub.2]O and phenylalanine flooding dose to investigate muscle protein synthesis with acute exercise in rats

Author

Gasier, Heath G., Riechman, Steven E., Wiggs, Michael P., Previs, Stephen F., and Fluckey, James D.

Subjects
Protein biosynthesis -- Measurement, Phenylalanine -- Physiological aspects, Muscles -- Properties, Exercise -- Physiological aspects, Exercise -- Research, Biological sciences
Abstract

The primary objective of this investigation was to determine whether [sup.2][H.sub.2]O and phenylalanine (Phe) flooding dose methods yield comparable fractional rates of protein synthesis (FSR) in skeletal muscle following a single bout of high-intensity resistance exercise (RE). Sprague-Dawley rats were assigned by body mass to either 4-h control (CON 4 h; n = 6), 4-h resistance exercise (RE 4 h; n = 6), 24-h control (CON 24 h; n = 6), or 24-h resistance exercise (RE 24 h; n = 6). The RE groups were operantly conditioned to engage in a single bout of high-intensity, 'squat-like' RE. All rats were given an intraperitoneal injection of 99.9% [sup.2][H.sub.2]O and provided 4.0% [sup.2][H.sub.2]O drinking water for either 24 (n = 12) or 4 h (n = 12) prior to receiving a flooding dose of L-[2,3,4,5,6-[sup.3]H]Phe 16 h post-RE. Neither method detected an effect of RE on FSR in the mixed gastrocnemius, plantaris, or soleus muscle. Aside from the qualitative similarities between methods, the 4-h [sup.2][H.sub.2]O FSR measurements, when expressed in percent per hour, were quantitatively greater than the 24-h [sup.2][H.sub.2]O and Phe flooding in all muscles (P < 0.001), and the 24-h [sup.2][H.sub.2]O was greater than the Phe flooding dose in the mixed gastrocnemius and plantaris (P < 0.05). In contrast, the actual percentage of newly synthesized protein was significantly higher in the 24- vs. 4-h [sup.2][H.sub.2]O and Phe flooding dose groups (P < 0.001). These results suggest that the methodologies provide 'qualitatively' similar results when a perturbation such as RE is studied. However, due to potential quantitative differences between methods, the experimental question should determine what approach should be used. fractional rates of protein synthesis; skeletal muscle

Published
2009

26. Female mice may have exacerbated catabolic signalling response compared to male mice during development and progression of disuse atrophy

Author

Rosa‐Caldwell, Megan E., primary, Lim, Seongkyun, additional, Haynie, Wesley A., additional, Brown, Jacob L., additional, Deaver, John William, additional, Morena Da Silva, Francielly, additional, Jansen, Lisa T., additional, Lee, David E., additional, Wiggs, Michael P., additional, Washington, Tyrone A., additional, and Greene, Nicholas P., additional

Published
2021
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34. Sex Differences in Anabolic Regulators during Development of Atrophic Pathology in Hindlimb-Unloading-Induced Disuse

Author

Jansen, Lisa T., primary, Rosa-Caldwell, Megan E., additional, Haynie, Wesley S., additional, Lim, Seongkyun, additional, Dunlap, Kirsten, additional, Brown, Jacob L., additional, Lee, David E., additional, Perry, Richard A., additional, Wiggs, Michael P., additional, Washington, Tyrone A., additional, and Greene, Nicholas P., additional

Published
2019
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36. 1-03 Role of Activin A in human cancer cachexia (ACTICA study)

Author

Baracos, Vickie, Muscaritoli, Maurizio, Aversa, Zaira, Rossi Fanelli, Filippo, Loumaye, A, de Barsy, M, Nachit, M, Frateur, L, Lause, P, Van Maanen, A, Thissen, JP, Awasthi, R, Gillis, C, Liberman, S, Stein, B, Charlebois, P, Carli, F, Ahmad, Sultan, Chiche, Dan, Reza Kazemi-Bajestani, Seyyed Mohammad, Becher, Harald, Venner, Peter, North, Scott, Vickie, Baracos, Li, Yi-Ping, Zhang, Guohua, Ma, Jennifer, Hall, Derek, Patel, Devang, Robinson, Samantha, Di Marco, Sergio, Gallouzi, Imed-Eddine, Sartori, Roberta, Penna, Fabio, Costelli, Paola, Sandri, Marco, Gregorevic, Paul, Toth, Michael J, Miller, Mark S, Callahan, Damien M, Couch, Marion E, Dittus, Kim, Sharma, Mridula, Argilés, Josep M, Bedard, N, Wiles, B, Jammoul, S, Miao, M, Wykes, L, Coyne, E, Hallauer, PL, Hastings, KEM, Stretch, C, Baracos, VE, Chevalier, S, Wing, SS, Marchildon, François, Lala, Neena, St-Louis, Catherine, Wiper-Bergeron, Nadine, Santos Silva, Kleiton Augusto, Dong, Jiangling, Tweardy, David J, Mitch, William E, Zhang, Liping, Hall, Derek T, Ma, Jennifer F, Griss, Takla, Jones, Russell G, Marco, Sergio Di, Marks, Daniel L, Rudnicki, Michael A, Laine, Aaron, Narayanan, Sriram, Choy, Hak, Girard, Luc, Gazdar, Adi, Minna, John, Infante, Rodney, Iyengar, Puneeth, Petruzzelli, Michele, Schweiger, Martina, Rincon, Mercedes, Robertson, Graham, Zechner, Rudolf, Wagner, Erwin F, Pin, Fabrizio, Costamagna, Domiziana, Camperi, Andrea, Sampaolesi, Maurilio, He, Wei, Talbert, Erin, Londhe, Priya, Bloomston, Mark, Croce, Carlo, Guttridge, Denis, Breit, Samuel N, Tsai, Vicky WW, Manandhar, Rakesh, Lin, Shu, Sainsbury, Amanda, Brown, David A, Ferrara, Michele, Reano, Simone, Angelino, Elia, Sabry, Omar, Filigheddu, Nicoletta, Graziani, Andrea, Tsoli, Maria, Allen, John, Taylor, Ryland, Scwheiger, Martina, Swarbrick, Michael M, Ehteda, Anahid, Miekle, Peter, Molloy, Mark, Waning, David L, Mohammad, Khalid S, Reiken, Steven, Xie, Wenjun, Marks, Andrew R, Guise, Theresa A, Moya, Rosita, Zhao, Chunfang, Davies, Joanna D, Carson, James, Everett Couch, Marion, Stecher, Michael, El Mouelhi, Mohamed, Tseng, Yu-Chou, Kulp, Samuel K, Lai, I-Lu, Hsu, En-Chi, He, Wei A, Frankhouser, David E, Yan, Pearlly S, Mo, Xiaokui, Lesinski, Gregory B, Marcucci, Guido, Guttridge, Denis C, Bekaii-Saab, Tanios, Chen, Ching-Shih, Haddad, AHI Al, Al-Azwani, EK, Mahamoud, Y, Safi, F, Salhat, H El, Malek, JA, Adrian, TE, Fearon, K, Temel, J, Currow, D, Gleich, L, Friend, J, Abernethy, A, Garcia, Jose M, Dubé, A, Patoine, D, Lemire, BB, Thériault, M-E, Ribeiro, F, Debigaré, R, Maltais, F, Smuder, Ashley J, Min, Kisuk, Kwon, Oh-Sung, Wiggs, Michael P, Sollanenk, Kurt J, Christou, Demetra D, Yoo, Jeung-Ki, Hwang, Moon-Hyon, Szeto, Hazel H, Kavazis, Andreas N, Powers, Scott K, Kroenke, Candyce H, Meyerhardt, Jeffrey A, Kwan, Marilyn L, Prado, Carla, Xiao, Jingjie, Weltzien, Erin, Castillo, Adrienne, Caan, Bette J, Guan, Chen, Giles, Kaitlin, Wing, Simon, Mazurak, Vera, Jagoe, R Thomas, Wiles, Benjamin, Miao, Miao, Coyne, Erin, Larose, Louise, Cybulsky, Andrey V, Wing, Simon S, Marino, Francesco Elia, Risbridger, Gail, Gold, Elspeth, Segatto, Marco, Fittipaldi, Raffaella, Caretti, Giuseppina, Lee, Hwabin, Fu, Dechen, Dwarkasing, JT, Boekschoten, MV, Argilès, JM, van Dijk, M, Busquets, S, Penna, F, Toledo, M, Laviano, A, Witkamp, RF, van Norren, K, Bédard, Nathalie, Plourde, Marie, Chevalier, Stéphanie, Lala-Tabbert, Neena, Marchildon, Francois, Torabi, S, Glare, P, Plodkowski, A, Margaron, Yoran, Fernandes, Mathieu, Morales, Delphine, Poydenot, Pauline, Menager, Pauline, Fuchs, Alexandra, Degot, Sébastien, Calore, Federica, Canella, Alessandro, Croce, Carlo M, Srinivasan, Kalayarasan, Pulliparracharuvil, Suprabha, Meyer, Jeffrey, Scherer, Philipp E, Kambadur, Ravi, Martinelli, Giulia B, Talamini, Laura, Previdi, Sara, Piccirillo, Rosanna, Jafri, Syed H, Previgliano, Carlos, Khandelwal, Keerti, Shi, Runhua, Mills, Glenn, Amato, Robert, Coats, Valérie, Ribeiro, Fernanda, Tremblay, Lise, Lacasse, Yves, Maltais, François, Saey, Didier, AL Vigano, Antonio, Ciutto, Lorella, Tomasso, Jonathan Di, Kilgour, Robert D, Morais, José A, Borod, Manuel, Almasud, A, Giles, K, Baracos, V, Guan, L, Mazurak, V, Khanuja, Jasleen, Gresham, Gillian, Osipov, Arsen, Tan, Carlyn-Rose, Tuli, Richard, Hendifar, Andrew, Narasimhan, Ashok, Greiner, Russell, Yasui, Yutaka, Bathe, Oliver, Fearon, Kenneth, Damaraju, Sambasivarao, Banh, Taylor, Kliewer, Kara, Hsiao, Yung-Hsuan, Belury, Martha A, Caan, Bette, Quesenberry, Charles, Kwan, Marilyn, Prado, Carla MM, Baracos, Vickie E, Birdsell, Laura, Stuyckens, Kim, Park, Youn Choi, Parekh, Trilok, Sawyer, Michael B, Wu, C, Fernandez, SA, Criswell, T, Chidiac, T, Guttridge, D, Villalona-Calero, M, Bekaii-Saab, T, Khan, Sarah, ALVigano, Antonio, Matos-Neto, EM, Figuerêdo, RG, Camargo, RG, Lima, JDCC, Alves, MJ, Riccardi, D, Alcantara, PS, Pinhata, J, Maximiano, L, Seelaender, M, Lamarche, Émilie, Au, Ernie D, Desai, Aditya P, Koniaris, Leonidas G, Zimmers, Teresa A, Manring, Heather, Weisleder, Noah, Okamura, Heidi, Frankhouser, David, Yan, Pearlly, Tomasso, Jonathan di, Fabbro, Egidio G Del, Davis, Mellar P, Fearon, Kenneth CH, Jatoi, Aminah A, Vigano, Antonio, Putman, Ted, Kezhuo, Zhang, Sladek, Robert, Enjiu, LM, Gomes, SP, Matos-Neto, E, Rossi-Fanneli, F, Seelaender, MC, Ebadi, Maryam, Mazurak, Vera C, Bhatia, Nikita, Padliya, Neerav D, Stadler, Volker, Dariani, Maghsoud, Hariri, Robert J, Parker, Valorie A, Matthews, Ryan R, Bonetto, Andrea, Puppa, Melissa, Kang, Kyung Shin, Mohammed, Khalid S, Robling, Alexander G, Toledo, Míriam, Oliva, Francesc, Luque, Melania, Betancourt, Angelica, Marmonti, Enrica, López-Soriano, Francisco J, Busquets, Sílvia, Theal, Rebecca, Jiang, Heng, Sanchez, Anthony, Hussain, Sabah N, Serpe, R, Madeddu, C, Gudiño, V, Gabba, S, Antoni, G, Macciò, A, and Banni, S

Subjects
Abstracts
Published
2015

37. Comparative Efficacy of Angiotensin II Type 1 Receptor Blockers Against Ventilator‐Induced Diaphragm Dysfunction in Rats.

Author

Hall, Stephanie E., Ahn, Bumsoo, Smuder, Ashley J., Morton, Aaron B., Hinkley, J. Matthew, Wiggs, Michael P., Sollanek, Kurt J., Hyatt, Hayden, and Powers, Scott K.

Subjects
ANGIOTENSIN II, ANGIOTENSIN-receptor blockers, DIAPHRAGM (Anatomy), RESPIRATORY muscles, PHRENIC nerve
Abstract

Mechanical ventilation (MV) is a life‐saving intervention for many critically ill patients. Unfortunately, prolonged MV results in the rapid development of inspiratory muscle weakness due to diaphragmatic atrophy and contractile dysfunction (termed ventilator‐induced diaphragm dysfunction (VIDD)). Although VIDD is a major risk factor for problems in weaning patients from MV, a standard therapy to prevent VIDD does not exist. However, emerging evidence suggests that pharmacological blockade of angiotensin II type 1 receptors (AT1Rs) protects against VIDD. Nonetheless, the essential characteristics of AT1R blockers (ARBs) required to protect against VIDD remain unclear. To determine the traits of ARBs that are vital for protection against VIDD, we compared the efficacy of two clinically relevant ARBs, irbesartan and olmesartan; these ARBs differ in molecular structure and effects on AT1Rs. Specifically, olmesartan blocks both angiotensin II (AngII) binding and mechanical activation of AT1Rs, whereas irbesartan prevents only AngII binding to AT1Rs. Using a well‐established preclinical model of prolonged MV, we tested the hypothesis that compared with irbesartan, olmesartan provides greater protection against VIDD. Our results reveal that irbesartan does not protect against VIDD whereas olmesartan defends against both MV‐induced diaphragmatic atrophy and contractile dysfunction. These findings support the hypothesis that olmesartan is superior to irbesartan in protecting against VIDD and are consistent with the concept that blockade of mechanical activation of AT1Rs is a required property of ARBs to shield against VIDD. These important findings provide a foundation for future clinical trials to evaluate ARBs as a therapy to protect against VIDD. [ABSTRACT FROM AUTHOR]

Published
2021
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39. Transcriptomic analysis of the development of skeletal muscle atrophy in cancer-cachexia in tumor-bearing mice

Author

Blackwell, Thomas A., primary, Cervenka, Igor, additional, Khatri, Bhuwan, additional, Brown, Jacob L., additional, Rosa-Caldwell, Megan E., additional, Lee, David E., additional, Perry, Richard A., additional, Brown, Lemuel A., additional, Haynie, Wesley S., additional, Wiggs, Michael P., additional, Bottje, Walter G., additional, Washington, Tyrone A., additional, Kong, Byungwhi C., additional, Ruas, Jorge L., additional, and Greene, Nicholas P., additional

Published
2018
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41. Protein imbalance in the development of skeletal muscle wasting in tumour‐bearing mice

Author

Brown, Jacob L., primary, Lee, David E., additional, Rosa‐Caldwell, Megan E., additional, Brown, Lemuel A., additional, Perry, Richard A., additional, Haynie, Wesley S., additional, Huseman, Kendra, additional, Sataranatarajan, Kavithalakshmi, additional, Van Remmen, Holly, additional, Washington, Tyrone A., additional, Wiggs, Michael P., additional, and Greene, Nicholas P., additional

Published
2018
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42. Cancer Cachexia

Author

Ramey, Sarah, primary, Rosa-Caldwell, Megan E., additional, Brown, Jacob L., additional, Perry, Richard A., additional, Haynie, Wesley A., additional, Caldwell, Aaron R., additional, Washington, Tyrone A., additional, Wiggs, Michael P., additional, and Greene, Nicholas P., additional

Published
2018
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46. Cancer cachexia-induced muscle atrophy: evidence for alterations in microRNAs important for muscle size

Author

Lee, David E., primary, Brown, Jacob L., additional, Rosa-Caldwell, Megan E., additional, Blackwell, Thomas A., additional, Perry, Richard A., additional, Brown, Lemuel A., additional, Khatri, Bhuwan, additional, Seo, Dongwon, additional, Bottje, Walter G., additional, Washington, Tyrone A., additional, Wiggs, Michael P., additional, Kong, Byung-Whi, additional, and Greene, Nicholas P., additional

Published
2017
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50. Cover Image, Volume 117, Number 8, August 2016

Author

Lee, David E., primary, Brown, Jacob L., additional, Rosa, Megan E., additional, Brown, Lemuel A., additional, Perry, Richard A., additional, Wiggs, Michael P., additional, Nilsson, Mats I., additional, Crouse, Stephen F., additional, Fluckey, James D., additional, Washington, Tyrone A., additional, and Greene, Nicholas P., additional

Published
2016
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