361 results on '"Westhoff, Mike-Andrew"'
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2. Tumor Treating Fields (TTFields) combined with the drug repurposing approach CUSP9v3 induce metabolic reprogramming and synergistic anti-glioblastoma activity in vitro
3. Trauma-associated extracellular histones mediate inflammation via a MYD88-IRAK1-ERK signaling axis and induce lytic cell death in human adipocytes
4. Correction to: Precision medicine in pediatric oncology
5. The Alcatraz‐Strategy: a roadmap to break the connectivity barrier in malignant brain tumours
6. OGDH and Bcl-xL loss causes synthetic lethality in glioblastoma
7. Methodological Approaches for Assessing Metabolomic Changes in Glioblastomas
8. Suppressing PD-L1 Expression via AURKA Kinase Inhibition Enhances Natural Killer Cell-Mediated Cytotoxicity against Glioblastoma.
9. Inhibition of Survivin Homodimerization Decreases Neuroblastoma Cell Growth
10. Aurora kinase A inhibition reverses the Warburg effect and elicits unique metabolic vulnerabilities in glioblastoma
11. HDAC inhibitors elicit metabolic reprogramming by targeting super-enhancers in glioblastoma models
12. Dual metabolic reprogramming by ONC201/TIC10 and 2-Deoxyglucose induces energy depletion and synergistic anti-cancer activity in glioblastoma
13. Compare and contrast: pediatric cancer versus adult malignancies
14. FBXW7 mutations reduce binding of NOTCH1, leading to cleaved NOTCH1 accumulation and target gene activation in CLL
15. The Potential Role of the Extracellular Matrix Glycoprotein Reelin in Glioblastoma Biology.
16. Long‐term haematological response and maintained immunological function after laparoscopic subtotal splenectomy in patients with hereditary spherocytosis
17. Combined inhibition of RAC1 and Bcl-2/Bcl-xL synergistically induces glioblastoma cell death through down-regulation of the Usp9X/Mcl-1 axis
18. Novel Approaches to Apoptosis-Inducing Therapies
19. The limitations of targeting MEK signalling in Glioblastoma therapy
20. Control of focal adhesion kinase by Src-mediated phosphorylation
21. Targeting cellular respiration as a therapeutic strategy in glioblastoma
22. Abstract LB304: Mechanisms of cell death escape in glioblastoma
23. Supplementary Materials, Figures 1 - 6 from Inhibition of NF-κB Signaling Ablates the Invasive Phenotype of Glioblastoma
24. Data from Inhibition of NF-κB Signaling Ablates the Invasive Phenotype of Glioblastoma
25. Data from MET Inhibition Elicits PGC1α-Dependent Metabolic Reprogramming in Glioblastoma
26. Data from Metabolic Reprogramming by Dual AKT/ERK Inhibition through Imipridones Elicits Unique Vulnerabilities in Glioblastoma
27. Supplementary Data from Combined HDAC and Bromodomain Protein Inhibition Reprograms Tumor Cell Metabolism and Elicits Synthetic Lethality in Glioblastoma
28. Supplementary Methods from Metabolic Reprogramming by Dual AKT/ERK Inhibition through Imipridones Elicits Unique Vulnerabilities in Glioblastoma
29. Data from Induction of Synthetic Lethality by Activation of Mitochondrial ClpP and Inhibition of HDAC1/2 in Glioblastoma
30. Supplementary Data from Induction of Synthetic Lethality by Activation of Mitochondrial ClpP and Inhibition of HDAC1/2 in Glioblastoma
31. Supplementary Data from MET Inhibition Elicits PGC1α-Dependent Metabolic Reprogramming in Glioblastoma
32. Supplementary Figures 1-9 from Metabolic Reprogramming by Dual AKT/ERK Inhibition through Imipridones Elicits Unique Vulnerabilities in Glioblastoma
33. Supplementary Figures S1-S5 from Sensitization for γ-Irradiation–Induced Apoptosis by Second Mitochondria-Derived Activator of Caspase
34. Supplementary Tables 1-2 from Phosphatidylinositol 3-Kinase Inhibition Broadly Sensitizes Glioblastoma Cells to Death Receptor– and Drug-Induced Apoptosis
35. Supplementary Figure Legends 1-5, Methods from Phosphatidylinositol 3-Kinase Inhibition Broadly Sensitizes Glioblastoma Cells to Death Receptor– and Drug-Induced Apoptosis
36. Supplementary Figures 1-5 from Phosphatidylinositol 3-Kinase Inhibition Broadly Sensitizes Glioblastoma Cells to Death Receptor– and Drug-Induced Apoptosis
37. How to Respond to Misinformation From the Anti-Vaccine Movement
38. How to Respond to Misinformation From the Anti-Vaccine Movement
39. Precision medicine in pediatric oncology
40. Anti-glioma Activity of Dapsone and Its Enhancement by Synthetic Chemical Modification
41. Abstracts of the 52nd Workshop for Pediatric Research: Frankfurt, Germany. 27-28 October 2016
42. EXTH-72. TTFIELDS ENHANCE THE ANTINEOPLASTIC ACTIVITY OF THE DRUG-REPURPOSING APPROACH CUSP9V3 IN GLIOBLASTOMA IN VITRO
43. TMET-39. INHIBITION OF THE TCA-CYCLE IS SYNTHETICALLY LETHAL WITH LOSS OF FUNCTION OF BCL-XL IN GLIOBLASTOMA
44. EXTH-48. INHIBITION OF SREBP-1 IS SYNTHETICALLY LETHAL WITH BCL-XL/BCL-2 INHIBITION IN GLIOBLASTOMA IN VITRO
45. EXTH-86. BH3 PROFILING IDENTIFIES ONC201/TIC10 AS A PROMISING PARTNER OF ABT-263 IN MEDULLOBLASTOMA IN VITRO
46. TMET-38. LOSS OF FUNCTION OF CDK7 IS SYNTHETICALLY LETHAL WITH FATTY ACID OXIDATION INHIBITION IN GLIOBLASTOMA
47. Therapeutic Drug-Induced Metabolic Reprogramming in Glioblastoma
48. Dual Inhibition of Bcl-2/Bcl-xL and XPO1 is synthetically lethal in glioblastoma model systems
49. Cell death-based treatment of childhood cancer
50. Cathepsin G-mediated proteolytic degradation of MHC class I molecules to facilitate immune detection of human glioblastoma cells
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