533 results on '"Westermarck, Jukka"'
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2. Germline-specific RNA helicase DDX4 forms cytoplasmic granules in cancer cells and promotes tumor growth
3. Structural mechanism for inhibition of PP2A-B56α and oncogenicity by CIP2A
4. CIP2A coordinates phosphosignaling, mitosis, and the DNA damage response
5. Endogenous PP2A inhibitor CIP2A degradation by chaperone-mediated autophagy contributes to the antitumor effect of mitochondrial complex I inhibition
6. Cisplatin overcomes radiotherapy resistance in OCT4-expressing head and neck squamous cell carcinoma
7. Chk1 Inhibition Ameliorates Alzheimer’s Disease Pathogenesis and Cognitive Dysfunction Through CIP2A/PP2A Signaling
8. Development of actionable targets of multi-kinase inhibitors (AToMI) screening platform to dissect kinase targets of staurosporines in glioblastoma cells
9. CIP2A Constrains Th17 Differentiation by Modulating STAT3 Signaling
10. CIP2A coordinates phosphosignaling, mitosis, and the DNA damage response
11. Cancer cell line microarray as a novel screening method for identification of radioresistance biomarkers in head and neck squamous cell carcinoma
12. (2S, 4R)-4-[18F]Fluoroglutamine for In vivo PET Imaging of Glioma Xenografts in Mice: an Evaluation of Multiple Pharmacokinetic Models
13. Direct Activation of Protein Phosphatase 2A (PP2A) by Tricyclic Sulfonamides Ameliorates Alzheimer’s Disease Pathogenesis in Cell and Animal Models
14. Inactivation of PP2A by a recurrent mutation drives resistance to MEK inhibitors
15. CIP2A-promoted astrogliosis induces AD-like synaptic degeneration and cognitive deficits
16. PP2A‐based triple‐strike therapy overcomes mitochondrial apoptosis resistance in brain cancer cells
17. Enhanced expression of MycN/CIP2A drives neural crest toward a neural stem cell-like fate : Implications for priming of neuroblastoma
18. Abstract 4727: RAS and PP2A activities converge on phosphoregulation of epigenetic complexes in cancer
19. Supplementary Table S1 from Senescence Sensitivity of Breast Cancer Cells Is Defined by Positive Feedback Loop between CIP2A and E2F1
20. Supplementary Methods from Senescence Sensitivity of Breast Cancer Cells Is Defined by Positive Feedback Loop between CIP2A and E2F1
21. Supplementary Figure from Ovarian Cancers with Low CIP2A Tumor Expression Constitute an APR-246–Sensitive Disease Subtype
22. Data from Senescence Sensitivity of Breast Cancer Cells Is Defined by Positive Feedback Loop between CIP2A and E2F1
23. Supplementary Figure 3 from Senescence Sensitivity of Breast Cancer Cells Is Defined by Positive Feedback Loop between CIP2A and E2F1
24. Supplementary Figure 4 from Senescence Sensitivity of Breast Cancer Cells Is Defined by Positive Feedback Loop between CIP2A and E2F1
25. Supplementary Figure 2 from Senescence Sensitivity of Breast Cancer Cells Is Defined by Positive Feedback Loop between CIP2A and E2F1
26. Supplementary Figure 1 from Senescence Sensitivity of Breast Cancer Cells Is Defined by Positive Feedback Loop between CIP2A and E2F1
27. Supplementary Figure 5 from Senescence Sensitivity of Breast Cancer Cells Is Defined by Positive Feedback Loop between CIP2A and E2F1
28. Supplementary Data from Ovarian Cancers with Low CIP2A Tumor Expression Constitute an APR-246–Sensitive Disease Subtype
29. Supplementary Table from Ovarian Cancers with Low CIP2A Tumor Expression Constitute an APR-246–Sensitive Disease Subtype
30. Integrin α2β1 Mediates Isoform-Specific Activation of p38 and Upregulation of Collagen Gene Transcription by a Mechanism Involving the α2 Cytoplasmic Tail
31. Table S1 from PP2A Inactivation Mediated by PPP2R4 Haploinsufficiency Promotes Cancer Development
32. Supplementary Figure legends from PP2A Inhibitor PME-1 Drives Kinase Inhibitor Resistance in Glioma Cells
33. Figure S4 from PP2A Inactivation Mediated by PPP2R4 Haploinsufficiency Promotes Cancer Development
34. Supplementary Table S4 from PP2A Inhibitor PME-1 Drives Kinase Inhibitor Resistance in Glioma Cells
35. Data from PP2A Inhibitor PME-1 Drives Kinase Inhibitor Resistance in Glioma Cells
36. Supplementary Data from CIP2A Interacts with TopBP1 and Drives Basal-Like Breast Cancer Tumorigenesis
37. Supplementary table S3 from UBR5 Is Coamplified with MYC in Breast Tumors and Encodes an Ubiquitin Ligase That Limits MYC-Dependent Apoptosis
38. Supplementary materials and methods from UBR5 Is Coamplified with MYC in Breast Tumors and Encodes an Ubiquitin Ligase That Limits MYC-Dependent Apoptosis
39. Supplementary Figure S1 from PP2A Inhibitor PME-1 Drives Kinase Inhibitor Resistance in Glioma Cells
40. Supplementary information from Discovery of a Novel CIP2A Variant (NOCIVA) with Clinical Relevance in Predicting TKI Resistance in Myeloid Leukemias
41. Supplementary Material and Methods from PP2A Inhibitor PME-1 Drives Kinase Inhibitor Resistance in Glioma Cells
42. Supplementary Figure Legends from PP2A Inactivation Mediated by PPP2R4 Haploinsufficiency Promotes Cancer Development
43. Supplementary Figures 1-6 from UBR5 Is Coamplified with MYC in Breast Tumors and Encodes an Ubiquitin Ligase That Limits MYC-Dependent Apoptosis
44. Data from CIP2A Is Associated with Human Breast Cancer Aggressivity
45. Supplementary Data from CIP2A Is Associated with Human Breast Cancer Aggressivity
46. Data from Chk1 Targeting Reactivates PP2A Tumor Suppressor Activity in Cancer Cells
47. Data from PME-1 Protects Extracellular Signal-Regulated Kinase Pathway Activity from Protein Phosphatase 2A–Mediated Inactivation in Human Malignant Glioma
48. Supplementary Figure 5 from Chk1 Targeting Reactivates PP2A Tumor Suppressor Activity in Cancer Cells
49. Supplementary Methods, Figure Legend from Chk1 Targeting Reactivates PP2A Tumor Suppressor Activity in Cancer Cells
50. Supplementary Figure 1 from Chk1 Targeting Reactivates PP2A Tumor Suppressor Activity in Cancer Cells
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