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1. Monitoring clonal burden as an alternative to blast count for myelodysplastic neoplasm treatment response: EARLY PHASE STUDIES

3. Bam-readcount -- rapid generation of basepair-resolution sequence metrics

4. A synthetic small molecule stalls pre-mRNA splicing by promoting an early-stage U2AF2-RNA complex

6. Genomic landscape of TP53-mutated myeloid malignancies

7. Utility of targeted gene sequencing to differentiate myeloid malignancies from other cytopenic conditions

8. A functional link between lariat debranching enzyme and the intron-binding complex is defective in non-photosensitive trichothiodystrophy

9. TP53 mutation status divides myelodysplastic syndromes with complex karyotypes into distinct prognostic subgroups

10. Immunosuppression and outcomes in adult patients with de novo acute myeloid leukemia with normal karyotypes

12. Proteomic and phosphoproteomic landscapes of acute myeloid leukemia

15. Systematic Analysis of Splice-Site-Creating Mutations in Cancer

18. SF3B1-mutant MDS as a distinct disease subtype: a proposal from the International Working Group for the Prognosis of MDS

20. The DNA methyltransferase inhibitor 5-aza-4’-thio-2’-deoxycytidine induces C>G transversions and acute lymphoid leukemia development

22. Plasma Proteomic Signature Predicts Myeloid Neoplasm Risk

24. Table S2 from R-Loop Accumulation in Spliceosome Mutant Leukemias Confers Sensitivity to PARP1 Inhibition by Triggering Transcription–Replication Conflicts

25. Figure S1-S8 from R-Loop Accumulation in Spliceosome Mutant Leukemias Confers Sensitivity to PARP1 Inhibition by Triggering Transcription–Replication Conflicts

26. Systematic Analysis of Splice-Site-Creating Mutations in Cancer

27. Correlation between peripheral blood and bone marrow mutations among patients with MDS from the National MDS Study

30. Plasma Proteomic Signature Predicts Risk of Myeloid Neoplasm

31. R-loop accumulation in spliceosome mutant leukemias confers sensitivity to PARP1 inhibition by triggering transcription-replication conflicts

32. A Pilot Study of High-Throughput, Sequence-Based Mutational Profiling of Primary Human Acute Myeloid Leukemia Cell Genomes

35. CpG Island Hypermethylation Mediated by DNMT3A Is a Consequence of AML Progression

36. U2af1 is a haplo-essential gene required for hematopoietic cancer cell survival in mice

39. Comprehensive genomic analysis reveals FLT3 activation and a therapeutic strategy for a patient with relapsed adult B-lymphoblastic leukemia

41. Supplementary Table from Convergent Clonal Evolution of Signaling Gene Mutations Is a Hallmark of Myelodysplastic Syndrome Progression

42. Supplementary Table from Genetic and Transcriptional Contributions to Relapse in Normal Karyotype Acute Myeloid Leukemia

43. Supplementary Figure from Convergent Clonal Evolution of Signaling Gene Mutations Is a Hallmark of Myelodysplastic Syndrome Progression

44. Data from Convergent Clonal Evolution of Signaling Gene Mutations Is a Hallmark of Myelodysplastic Syndrome Progression

45. Supplementary Data from Convergent Clonal Evolution of Signaling Gene Mutations Is a Hallmark of Myelodysplastic Syndrome Progression

46. Supplementary Data from Genetic and Transcriptional Contributions to Relapse in Normal Karyotype Acute Myeloid Leukemia

47. Data from Genetic and Transcriptional Contributions to Relapse in Normal Karyotype Acute Myeloid Leukemia

49. Genomic analysis of germ line and somatic variants in familial myelodysplasia/acute myeloid leukemia

50. Mutant U2AF1 Expression Alters Hematopoiesis and Pre-mRNA Splicing In Vivo

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