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1. The evolving genetic landscape of telomere biology disorder dyskeratosis congenita

2. New WHO classification of genetic variants causing G6PD deficiency

3. The clinical picture of ERCC6L2 disease: from bone marrow failure to acute leukemia

7. Germline thymidylate synthase deficiency impacts nucleotide metabolism and causes dyskeratosis congenita

11. The Human Phenotype Ontology in 2017

12. ClinGen Myeloid Malignancy Variant Curation Expert Panel recommendations for germline RUNX1 variants

13. Genome instability is a consequence of transcription deficiency in patients with bone marrow failure harboring biallelic ERCC6L2 variants

14. Germline NPM1 mutations lead to altered rRNA 2′-O-methylation and cause dyskeratosis congenita

15. The complex genetic landscape of familial MDS and AML reveals pathogenic germline variants

16. GATA2 monoallelic expression underlies reduced penetrance in inherited GATA2-mutated MDS/AML

17. P008: ClinGen Glucose-6-phosphate dehydrogenase (G6PD) Variant Curation Expert Panel: Addressing the need for genetic variant classification in G6PD deficiency*

22. Germline ERCC excision repair 6 like 2 (ERCC6L2) mutations lead to impaired erythropoiesis and reshaping of the bone marrow microenvironment

23. Revertant Somatic Mosaicism by Mitotic Recombination in Dyskeratosis Congenita

25. Multinational Study on the Clinical and Genetic Features of the ERCC6L2-Disease

42. Autores

43. Inflammatory Skin and Bowel Disease Linked to ADAM17 Deletion

49. Contributors

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