18 results on '"Vouret-Craviari, Valerie"'
Search Results
2. Author Response: Activation of the P2RX7/IL-18 pathway in immune cells attenuates lung fibrosis
- Author
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Janho dit Hreich, Serena, primary, Juhel, Thierry, additional, Leroy, Sylvie, additional, Ghinet, Alina, additional, Brau, Frederic, additional, Hofman, Veronique, additional, Hofman, Paul, additional, and Vouret-Craviari, Valerie, additional
- Published
- 2024
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3. Activation of the P2RX7/IL-18 pathway in immune cells attenuates lung fibrosis.
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dit Hreich, Serena Janho, Juhel, Thierry, Leroy, Sylvie, Ghinet, Alina, Brau, Frederic, Hofman, Veronique, Hofman, Paul, and Vouret-Craviari, Valerie
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- 2024
- Full Text
- View/download PDF
4. Amplification loop of the inflammatory process is induced by P2[X.sub.7]R activation in intestinal epithelial cells in response to neutrophil transepithelial migration
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Cesaro, Annabelle, Brest, Patrick, Hofman, Veronique, Hebuterne, Xavier, Wildman, Scott, Ferrua, Bernard, Marchetti, Sandrine, Doglio, Alain, Vouret-Craviari, Valerie, Galland, Franck, Naquet, Philippe, Mograbi, Baharia, Unwin, Robert, and Hofman, Paul
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Epithelial cells -- Properties ,Cell migration -- Research ,Neutrophils -- Properties ,Natural immunity -- Research ,Crohn's disease -- Development and progression ,Biological sciences - Abstract
Inflammatory bowel diseases (IBD) are characterized during their active phase by polymorphonuclear leukocyte (PMNL) transepithelial migration. The efflux of PMNL into the mucosa is associated with the production of proinflammatory cytokines and the release of ATP from damaged and necrotic cells. The expression and function of purinergic [P2X.sub.7] receptor ([P2X.sub.7]R) in intestinal epithelial cells (IEC) and its potential role in the 'cross talk' between IEC and PMNL have not been explored. The aims of the present study were 1) to examine [P2X.sub.7]R expression in IEC (T84 cells) and in human intestinal biopsies; 2) to detect any changes in [P2X.sub.7]R expression in T84 cells during PMNL transepithelial migration, and during the active and quiescent phases of IBD; and 3) to test whether [P2X.sub.7]TR stimulation in T84 monolayers can induce caspase-1 activation and IL-1[beta] release by IEC. We found that a functional ATP-sensitive [P2X.sub.7]R is constitutively expressed at the apical surface of IEC T84 cells. PMNL transmigration regulates dynamically [P2X.sub.7]R expression and alters its distribution from the apical to basolateral surface of IEC during the early phase of PMNL transepithelial migration in vitro. [P2X.sub.7]R expression was weak in intestinal biopsies obtained during the active phase of IBD. We show that activation of epithelial [P2X.sub.7]R is mandatory for PMNL-induced caspase-1 activation and IL-1[beta] release by IEC. Overall, these changes in [P2X.sub.7]R function may serve to tailor the intensity of the inflammatory response and to prevent IL-1[beta] overproduction and inflammatory disease. innate immunity; Crohn's disease doi: 10.1152/ajpgi.00282.2009.
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- 2010
5. Differential expression and regulation of ADAM17 and TIMP3 in acute inflamed intestinal epithelia
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Cesaro, Annabelle, Abakar-Mahamat, Abakar, Brest, Patrick, Lassalle, Sandra, Selva, Eric, Filippi, Jerome, Hebuterne, Xavier, Hugot, Jean-Pierre, Doglio, Alain, Galland, Franck, Naquet, Philippe, Vouret-Craviari, Valerie, Mograbi, Baharia, and Hofman, Paul M.
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Crohn's disease -- Genetic aspects ,Crohn's disease -- Care and treatment ,Crohn's disease -- Research ,Epithelial cells -- Physiological aspects ,Epithelial cells -- Genetic aspects ,Epithelial cells -- Research ,Gene expression -- Research ,Immunohistochemistry -- Usage ,Immunohistochemistry -- Methods ,Biological sciences - Abstract
The acute phase of Crohn's disease (CD) is characterized by a large afflux of polymorphonuclear leukocytes (PMNL) into the mucosa and by the release of TNF-[alpha]. Conversion of inactive TNF-[alpha] into an active form requires the cleavage of a transmembrane TNF-[alpha] precursor by the TNF-[alpha]-converting enzyme (ADAM17), a protease mainly regulated by the tissue inhibitor of metalloproteinase 3 (TIMP3). The aim of the present study was to investigate in an in vitro model of PMNL transepithelial migration and in the intestinal mucosa of patients with CD the expression and regulation of ADAM17 and TIMP3 in intestinal epithelial cells (IEC). ADAM17 and TIMP3 expression was analyzed by Western blotting, RT-PCR, confocal microscopy, and immunohistochemistry by using the T84 model and digestive biopsies. ADAM17 expression in IEC was increased at a posttranscriptional level during the early phase (from 2 to 4 h) of PMNL transepithelial migration whereas TIMP3 was only increased 24 h later. TNF-[alpha] induced an early upregulation of ADAM17 in T84 cells, whereas PMNL adhesion, [H.sub.2][O.sub.2], or epithelial tight junction opening alone did not affect the amount of ADAM 17. Immunohistochemistry of intestinal biopsies revealed that strong expression of ADAM 17 was associated with a high activity of CD. In contrast, TIMP3 was very poorly expressed in these biopsies. ADAM 17 and TIMP3 profiling did not correlated with the NOD2/CARD 15 status. The ADAM 17 activity was higher both in the early phase of PMNL transepithelial migration and in active CD. These results showed early posttranscriptional upregulation of ADAM17 in IEC linked to PMNL transepithelial migration and a high activity of CD. Crohn's disease; intestinal epithelial cell; neutrophil
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- 2009
6. Differential expression and regulation of ADAM 17 and TIMP3 in acute inflamed intestinal epithelia.
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Cesaro, Annabelle, Abakar-Mahamat, Abakar, Brest, Patrick, Lassalle, Sandra, Selva, Eric, Filippi, Jérôme, Hébuterne, Xavier, Hugot, Jean-Pierre, Doglio, Main, Galland, Franck, Naquet, Philippe, Vouret-Craviari, Valerie, Mograbi, Baharia, and Hofman, Paul M.
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CROHN'S disease ,LEUKOCYTES ,METALLOPROTEINASES ,ENZYME regulation ,WESTERN immunoblotting ,PHYSIOLOGY - Abstract
The acute phase of Crohn's disease (CD) is characterized by a large afflux of polymorphonuclear leukocytes (PMNL) into the mucosa and by the release of TNF-α. Conversion of inactive TNF-α into an active form requires the cleavage of a transmembrane TNF-α precursor by the TNF-α-converting enzyme (ADAM17), a protease mainly regulated by the tissue inhibitor of metalloproteinase 3 (TIMP3). The aim of the present study was to investigate in an in vitro model of PMNL transepithelial migration and in the intestinal mucosa of patients with CD the expression and regulation of ADAM17 and TIMP3 in intestinal epithelial cells (IEC). ADAM17 and TIMP3 expression was analyzed by Western blotting, RT-PCR, confocal microscopy, and immunohistochemistry by using the T84 model and digestive biopsies. ADAM17 expression in IEC was increased at a posttranscriptional level during the early phase (from 2 to 4 h) of PMNL transepithelial migration whereas TIMP3 was only increased 24 h later. TNF-α induced an early upregulation of ADAM17 in T84 cells, whereas PMNL adhesion, H
2 O2 , or epithelial tight junction opening alone did not affect the amount of ADAM17. Immunohistochemistry of intestinal biopsies revealed that strong expression of ADAM17 was associated with a high activity of CD. In contrast, TIMP3 was very poorly expressed in these biopsies. ADAM17 and TIMP3 profiling did not correlated with the NOD2/CARD15 status. The ADAM17 activity was higher both in the early phase of PMNL transepithelial migration and in active CD. These results showed early posttranscriptional upregulation of ADAM17 in IEC linked to PMNL transepithelial migration and a high activity of CD. [ABSTRACT FROM AUTHOR]- Published
- 2009
- Full Text
- View/download PDF
7. Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
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Klionsky, Daniel J, Abdelmohsen, Kotb, Abe, Akihisa, Abedin, Md Joynal, Abeliovich, Hagai, Arozena, Abraham Acevedo, Adachi, Hiroaki, Adams, Christopher M, Adams, Peter D, Adeli, Khosrow, Adhihetty, Peter J, Adler, Sharon G, Agam, Galila, Agarwal, Rajesh, Aghi, Manish K, Agnello, Maria, Agostinis, Patrizia, Aguilar, Patricia V, Aguirre-Ghiso, Julio, Airoldi, Edoardo M, Ait-Si-Ali, Slimane, Akematsu, Takahiko, Akporiaye, Emmanuel T, Al-Rubeai, Mohamed, Albaiceta, Guillermo M, Albanese, Chris, Albani, Diego, Albert, Matthew L, Aldudo, Jesus, Alguel, Hana, Alirezaei, Mehrdad, Alloza, Iraide, Almasan, Alexandru, Almonte-Beceril, Maylin, Alnemri, Emad S, Alonso, Covadonga, Altan-Bonnet, Nihal, Altieri, Dario C, Alvarez, Silvia, Alvarez-Erviti, Lydia, Alves, Sandro, Amadoro, Giuseppina, Amano, Atsuo, Amantini, Consuelo, Ambrosio, Santiago, Amelio, Ivano, Amer, Amal O, Amessou, Mohamed, Amon, Angelika, An, Zhenyi, Anania, Frank A, Andersen, Stig U, Andley, Usha P, Andreadi, Catherine K, Andrieu-Abadie, Nathalie, Anel, Alberto, Ann, David K, Anoopkumar-Dukie, Shailendra, Antonioli, Manuela, Aoki, Hiroshi, Apostolova, Nadezda, Aquila, Saveria, Aquilano, Katia, Araki, Koichi, Arama, Eli, Aranda, Agustin, Araya, Jun, Arcaro, Alexandre, Arias, Esperanza, Arimoto, Hirokazu, Ariosa, Aileen R, Armstrong, Jane L, Arnould, Thierry, Arsov, Ivica, Asanuma, Katsuhiko, Askanas, Valerie, Asselin, Eric, Atarashi, Ryuichiro, Atherton, Sally S, Atkin, Julie D, Attardi, Laura D, Auberger, Patrick, Auburger, Georg, Aurelian, Laure, Autelli, Riccardo, Avagliano, Laura, Avantaggiati, Maria Laura, Avrahami, Limor, Awale, Suresh, Azad, Neelam, Bachetti, Tiziana, Backer, Jonathan M, Bae, Dong-Hun, Bae, Jae-Sung, Bae, Ok-Nam, Bae, Soo Han, Baehrecke, Eric H, Baek, Seung-Hoon, Baghdiguian, Stephen, Bagniewska-Zadworna, Agnieszka, Bai, Hua, Bai, Jie, Bai, Xue-Yuan, Bailly, Yannick, Balaji, Kithiganahalli Narayanaswamy, Balduini, Walter, Ballabio, Andrea, Balzan, Rena, Banerjee, Rajkumar, Banhegyi, Gabor, Bao, Haijun, Barbeau, Benoit, Barrachina, Maria D, Barreiro, Esther, Bartel, Bonnie, Bartolome, Alberto, Bassham, Diane C, Bassi, Maria Teresa, Bast, Robert C, Basu, Alakananda, Batista, Maria Teresa, Batoko, Henri, Battino, Maurizio, Bauckman, Kyle, Baumgarner, Bradley L, Bayer, Ulrich K, Beale, Rupert, Beaulieu, Jean-Francois, Beck, George R, Becker, Christoph, Beckham, David J, Bedard, Pierre-Andre, Bednarski, Patrick J, Begley, Thomas J, Behl, Christian, Behrends, Christian, Behrens, Georg MN, Behrns, Kevin E, Bejarano, Eloy, Belaid, Amine, Belleudi, Francesca, Benard, Giovanni, Berchem, Guy, Bergamaschi, Daniele, Bergami, Matteo, Berkhout, Ben, Berliocchi, Laura, Bernard, Amelie, Bernard, Monique, Bernassola, Francesca, Bertolotti, Anne, Bess, Amanda S, Besteiro, Sebastien, Bettuzzi, Saverio, Bhalla, Savita, Bhattacharyya, Shalmoli, Bhutia, Sujit K, Biagosch, Caroline, Bianchi, Michele Wolfe, Biard-Piechaczyk, Martine, Billes, Viktor, Bincoletto, Claudia, Bingol, Baris, Bird, Sara W, Bitoun, Marc, Bjedov, Ivana, Blackstone, Craig, Blanc, Lionel, Blanco, Guillermo A, Blomhoff, Heidi Kiil, Boada-Romero, Emilio, Boeckler, Stefan, Boes, Marianne, Boesze-Battaglia, Kathleen, Boise, Lawrence H, Bolino, Alessandra, Boman, Andrea, Bonaldo, Paolo, Bordi, Matteo, Bosch, Jurgen, Botana, Luis M, Botti, Joelle, Bou, German, Bouche, Marina, Bouchecareilh, Marion, Boucher, Marie-Josee, Boulton, Michael E, Bouret, Sebastien G, Boya, Patricia, Boyer-Guittaut, Michaeel, Bozhkov, Peter V, Brady, Nathan, Braga, Vania MM, Brancolini, Claudio, Braus, Gerhard H, Bravo-San Pedro, Jose M, Brennan, Lisa A, Bresnick, Emery H, Brest, Patrick, Bridges, Dave, Bringer, Marie-Agnes, Brini, Marisa, Brito, Glauber C, Brodin, Bertha, Brookes, Paul S, Brown, Eric J, Brown, Karen, Broxmeyer, Hal E, Bruhat, Alain, Brum, Patricia Chakur, Brumell, John H, Brunetti-Pierri, Nicola, Bryson-Richardson, Robert J, Buch, Shilpa, Buchan, Alastair M, Budak, Hikmet, Bulavin, Dmitry V, Bultman, Scott J, Bultynck, Geert, Bumbasirevic, Vladimir, Burelle, Yan, Burke, Robert E, Burmeister, Margit, Buetikofer, Peter, Caberlotto, Laura, Cadwell, Ken, Cahova, Monika, Cai, Dongsheng, Cai, Jingjing, Cai, Qian, Calatayud, Sara, Camougrand, Nadine, Campanella, Michelangelo, Campbell, Grant R, Campbell, Matthew, Campello, Silvia, Candau, Robin, Caniggia, Isabella, Cantoni, Lavinia, Cao, Lizhi, Caplan, Allan B, Caraglia, Michele, Cardinali, Claudio, Cardoso, Sandra Morais, Carew, Jennifer S, Carleton, Laura A, Carlin, Cathleen R, Carloni, Silvia, Carlsson, Sven R, Carmona-Gutierrez, Didac, Carneiro, Leticia AM, Carnevali, Oliana, Carra, Serena, Carrier, Alice, Carroll, Bernadette, Casas, Caty, Casas, Josefina, Cassinelli, Giuliana, Castets, Perrine, Castro-Obregon, Susana, Cavallini, Gabriella, Ceccherini, Isabella, Cecconi, Francesco, Cederbaum, Arthur I, Cena, Valentin, Cenci, Simone, Cerella, Claudia, Cervia, Davide, Cetrullo, Silvia, Chaachouay, Hassan, Chae, Han-Jung, Chagin, Andrei S, Chai, Chee-Yin, Chakrabarti, Gopal, Chamilos, Georgios, Chan, Edmond YW, Chan, Matthew TV, Chandra, Dhyan, Chandra, Pallavi, Chang, Chih-Peng, Chang, Raymond Chuen-Chung, Chang, Ta Yuan, Chatham, John C, Chatterjee, Saurabh, Chauhan, Santosh, Che, Yongsheng, Cheetham, Michael E, Cheluvappa, Rajkumar, Chen, Chun-Jung, Chen, Gang, Chen, Guang-Chao, Chen, Guoqiang, Chen, Hongzhuan, Chen, Jeff W, Chen, Jian-Kang, Chen, Min, Chen, Mingzhou, Chen, Peiwen, Chen, Qi, Chen, Quan, Chen, Shang-Der, Chen, Si, Chen, Steve SL, Chen, Wei, Chen, Wei-Jung, Chen, Wen Qiang, Chen, Wenli, Chen, Xiangmei, Chen, Yau-Hung, Chen, Ye-Guang, Chen, Yin, Chen, Yingyu, Chen, Yongshun, Chen, Yu-Jen, Chen, Yue-Qin, Chen, Yujie, Chen, Zhen, Chen, Zhong, Cheng, Alan, Cheng, Christopher HK, Cheng, Hua, Cheong, Heesun, Cherry, Sara, Chesney, Jason, Cheung, Chun Hei Antonio, Chevet, Eric, Chi, Hsiang Cheng, Chi, Sung-Gil, Chiacchiera, Fulvio, Chiang, Hui-Ling, Chiarelli, Roberto, Chiariello, Mario, Chieppa, Marcello, Chin, Lih-Shen, Chiong, Mario, Chiu, Gigi NC, Cho, Dong-Hyung, Cho, Ssang-Goo, Cho, William C, Cho, Yong-Yeon, Cho, Young-Seok, Choi, Augustine MK, Choi, Eui-Ju, Choi, Eun-Kyoung, Choi, Jayoung, Choi, Mary E, Choi, Seung-Il, Chou, Tsui-Fen, Chouaib, Salem, Choubey, Divaker, Choubey, Vinay, Chow, Kuan-Chih, Chowdhury, Kamal, Chu, Charleen T, Chuang, Tsung-Hsien, Chun, Taehoon, Chung, Hyewon, Chung, Taijoon, Chung, Yuen-Li, Chwae, Yong-Joon, Cianfanelli, Valentina, Ciarcia, Roberto, Ciechomska, Iwona A, Ciriolo, Maria Rosa, Cirone, Mara, Claerhout, Sofie, Clague, Michael J, Claria, Joan, Clarke, Peter GH, Clarke, Robert, Clementi, Emilio, Cleyrat, Cedric, Cnop, Miriam, Coccia, Eliana M, Cocco, Tiziana, Codogno, Patrice, Coers, Joern, Cohen, Ezra EW, Colecchia, David, Coletto, Luisa, Coll, Nuria, Colucci-Guyon, Emma, Comincini, Sergio, Condello, Maria, Cook, Katherine L, Coombs, Graham H, Cooper, Cynthia D, Cooper, Mark J, Coppens, Isabelle, Corasaniti, Maria Tiziana, Corazzari, Marco, Corbalan, Ramon, Corcelle-Termeau, Elisabeth, Cordero, Mario D, Corral-Ramos, Cristina, Corti, Olga, Cossarizza, Andrea, Costelli, Paola, Costes, Safia, Coto-Montes, Ana, Cottet, Sandra, Couve, Eduardo, Covey, Lori R, Cowart, Ashley L, Cox, Jeffery S, Coxon, Fraser P, Coyne, Carolyn B, Cragg, Mark S, Craven, Rolf J, Crepaldi, Tiziana, Crespo, Jose L, Criollo, Alfredo, Crippa, Valeria, Cruz, Maria Teresa, Cuervo, Ana Maria, Cuezva, Jose M, Cui, Taixing, Cutillas, Pedro R, Czaja, Mark J, Czyzyk-Krzeska, Maria F, Dagda, Ruben K, Dahmen, Uta, Dai, Chunsun, Dai, Wenjie, Dai, Yun, Dalby, Kevin N, Valle, Luisa Dalla, Dalmasso, Guillaume, D'Amelio, Marcello, Damme, Markus, Darfeuille-Michaud, Arlette, Dargemont, Catherine, Darley-Usmar, Victor M, Dasarathy, Srinivasan, Dasgupta, Biplab, Dash, Srikanta, Dass, Crispin R, Davey, Hazel Marie, Davids, Lester M, Davila, David, Davis, Roger J, Dawson, Ted M, Dawson, Valina L, Daza, Paula, de Belleroche, Jackie, de Figueiredo, Paul, Bressan Queiroz de Figueiredo, Regina Celia, de la Fuente, Jose, De Martino, Luisa, De Matteis, Antonella, De Meyer, Guido RY, De Milito, Angelo, De Santi, Mauro, de Souza, Wanderley, De Tata, Vincenzo, De Zio, Daniela, Debnath, Jayanta, Dechant, Reinhard, Decuypere, Jean-Paul, Deegan, Shane, Dehay, Benjamin, Del Bello, Barbara, Del Re, Dominic P, Delage-Mourroux, Regis, Delbridge, Lea MD, Deldicque, Louise, Delorme-Axford, Elizabeth, Deng, Yizhen, Dengjel, Joern, Denizot, Melanie, Dent, Paul, Der, Channing J, Deretic, Vojo, Derrien, Benoit, Deutsch, Eric, Devarenne, Timothy P, Devenish, Rodney, Di Bartolomeo, Sabrina, Di Daniele, Nicola, Di Domenico, Fabio, Di Nardo, Alessia, Di Paola, Simone, Di Pietro, Antonio, Di Renzo, Livia, DiAntonio, Aaron, Diaz-Araya, Guillermo, Diaz-Laviada, Ines, Diaz-Meco, Maria T, Diaz-Nido, Javier, Dickey, Chad A, Dickson, Robert C, Diederich, Marc, Digard, Paul, Dikic, Ivan, Dinesh-Kumar, Savithrama P, Ding, Chan, Ding, Wen-Xing, Ding, Zufeng, Dini, Luciana, Distler, Joerg HW, Diwan, Abhinav, Djavaheri-Mergny, Mojgan, Dmytruk, Kostyantyn, Dobson, Renwick CJ, Doetsch, Volker, Dokladny, Karol, Dokudovskaya, Svetlana, Donadelli, Massimo, Dong, Charlie X, Dong, Xiaonan, Dong, Zheng, Donohue, Terrence M, Doran, Kelly S, D'Orazi, Gabriella, Dorn, Gerald W, Dosenko, Victor, Dridi, Sami, Drucker, Liat, Du, Jie, Du, Li-Lin, Du, Lihuan, du Toit, Andre, Dua, Priyamvada, Duan, Lei, Duann, Pu, Dubey, Vikash Kumar, Duchen, Michael R, Duchosal, Michel A, Duez, Helene, Dugail, Isabelle, Dumit, Veronica I, Duncan, Mara C, Dunlop, Elaine A, Dunn, William A, Dupont, Nicolas, Dupuis, Luc, Duran, Raul V, Durcan, Thomas M, Duvezin-Caubet, Stephane, Duvvuri, Umamaheswar, Eapen, Vinay, Ebrahimi-Fakhari, Darius, Echard, Arnaud, Eckhart, Leopold, Edelstein, Charles L, Edinger, Aimee L, Eichinger, Ludwig, Eisenberg, Tobias, Eisenberg-Lerner, Avital, Eissa, Tony N, El-Deiry, Wafik S, El-Khoury, Victoria, Elazar, Zvulun, Eldar-Finkelman, Hagit, Elliott, Chris JH, Emanuele, Enzo, Emmenegger, Urban, Engedal, Nikolai, Engelbrecht, Anna-Mart, Engelender, Simone, Enserink, Jorrit M, Erdmann, Ralf, Erenpreisa, Jekaterina, Eri, Rajaraman, Eriksen, Jason L, Erman, Andreja, Escalante, Ricardo, Eskelinen, Eeva-Liisa, Espert, Lucile, Esteban-Martinez, Lorena, Evans, Thomas J, Fabri, Mario, Fabrias, Gemma, Fabrizi, Cinzia, Facchiano, Antonio, Faergeman, Nils J, Faggioni, Alberto, Fairlie, Douglas W, Fan, Chunhai, Fan, Daping, Fan, Jie, Fang, Shengyun, Fanto, Manolis, Fanzani, Alessandro, Farkas, Thomas, Faure, Mathias, Favier, Francois B, Fearnhead, Howard, Federici, Massimo, Fei, Erkang, Felizardo, Tania C, Feng, Hua, Feng, Yibin, Feng, Yuchen, Ferguson, Thomas A, Fernandez, Alvaro F, Fernandez-Barrena, Maite G, Fernandez-Checa, Jose C, Fernandez-Lopez, Arsenio, Fernandez-Zapico, Martin E, Feron, Olivier, Ferraro, Elisabetta, Ferreira-Halder, Carmen Verissima, Fesus, Laszlo, Feuer, Ralph, Fiesel, Fabienne C, Filippi-Chiela, Eduardo C, Filomeni, Giuseppe, Fimia, Gian Maria, Fingert, John H, Finkbeiner, Steven, Finkel, Toren, Fiorito, Filomena, Fisher, Paul B, Flajolet, Marc, Flamigni, Flavio, Florey, Oliver, Florio, Salvatore, Floto, Andres R, Folini, Marco, Follo, Carlo, Fon, Edward A, Fornai, Francesco, Fortunato, Franco, Fraldi, Alessandro, Franco, Rodrigo, Francois, Arnaud, Francois, Aurelie, Frankel, Lisa B, Fraser, Iain DC, Frey, Norbert, Freyssenet, Damien G, Frezza, Christian, Friedman, Scott L, Frigo, Daniel E, Fu, Dongxu, Fuentes, Jose M, Fueyo, Juan, Fujitani, Yoshio, Fujiwara, Yuuki, Fujiya, Mikihiro, Fukuda, Mitsunori, Fulda, Simone, Fusco, Carmela, Gabryel, Bozena, Gaestel, Matthias, Gailly, Philippe, Gajewska, Malgorzata, Galadari, Sehamuddin, Galili, Gad, Galindo, Inmaculada, Galindo, Maria F, Galliciotti, Giovanna, Galluzzi, Lorenzo, Galluzzi, Luca, Galy, Vincent, Gammoh, Noor, Gandy, Sam, Ganesan, Anand K, Ganesan, Swamynathan, Ganley, Ian G, Gannage, Monique, Gao, Fen-Biao, Gao, Feng, Gao, Jian-Xin, Garcia Nannig, Lorena, Vescovi, Eleonora Garcia, Garcia-Macia, Marina, Garcia-Ruiz, Carmen, Garg, Abhishek D, Garg, Pramod Kumar, Gargini, Ricardo, Gassen, Nils Christian, Gatica, Damian, Gatti, Evelina, Gavard, Julie, Gavathiotis, Evripidis, Ge, Liang, Ge, Pengfei, Ge, Shengfang, Gean, Po-Wu, Gelmetti, Vania, Genazzani, Armando A, Geng, Jiefei, Genschik, Pascal, Gerner, Lisa, Gestwicki, Jason E, Gewirtz, David A, Ghavami, Saeid, Ghigo, Eric, Ghosh, Debabrata, Giammarioli, Anna Maria, Giampieri, Francesca, Giampietri, Claudia, Giatromanolaki, Alexandra, Gibbings, Derrick J, Gibellini, Lara, Gibson, Spencer B, Ginet, Vanessa, Giordano, Antonio, Giorgini, Flaviano, Giovannetti, Elisa, Girardin, Stephen E, Gispert, Suzana, Giuliano, Sandy, Gladson, Candece L, Glavic, Alvaro, Gleave, Martin, Godefroy, Nelly, Gogal, Robert M, Gokulan, Kuppan, Goldman, Gustavo H, Goletti, Delia, Goligorsky, Michael S, Gomes, Aldrin V, Gomes, Ligia C, Gomez, Hernando, Gomez-Manzano, Candelaria, Gomez-Sanchez, Ruben, Goncalves, Dawit AP, Goncu, Ebru, Gong, Qingqiu, Gongora, Celine, Gonzalez, Carlos B, Gonzalez-Alegre, Pedro, Gonzalez-Cabo, Pilar, Ana Gonzalez-Polo, Rosa, Goping, Ing Swie, Gorbea, Carlos, Gorbunov, Nikolai V, Goring, Daphne R, Gorman, Adrienne M, Gorski, Sharon M, Goruppi, Sandro, Goto-Yamada, Shino, Gotor, Cecilia, Gottlieb, Roberta A, Gozes, Illana, Gozuacik, Devrim, Graba, Yacine, Graef, Martin, Granato, Giovanna E, Grant, Gary Dean, Grant, Steven, Gravina, Giovanni Luca, Green, Douglas R, Greenhough, Alexander, Greenwood, Michael T, Grimaldi, Benedetto, Gros, Frederic, Grose, Charles, Groulx, Jean-Francois, Gruber, Florian, Grumati, Paolo, Grune, Tilman, Guan, Jun-Lin, Guan, Kun-Liang, Guerra, Barbara, Guillen, Carlos, Gulshan, Kailash, Gunst, Jan, Guo, Chuanyong, Guo, Lei, Guo, Ming, Guo, Wenjie, Guo, Xu-Guang, Gust, Andrea A, Gustafsson, Asa B, Gutierrez, Elaine, Gutierrez, Maximiliano G, Gwak, Ho-Shin, Haas, Albert, Haber, James E, Hadano, Shinji, Hagedorn, Monica, Hahn, David R, Halayko, Andrew J, Hamacher-Brady, Anne, Hamada, Kozo, Hamai, Ahmed, Hamann, Andrea, Hamasaki, Maho, Hamer, Isabelle, Hamid, Qutayba, Hammond, Ester M, Han, Feng, Han, Weidong, Handa, James T, Hanover, John A, Hansen, Malene, Harada, Masaru, Harhaji-Trajkovic, Ljubica, Harper, Wade J, Harrath, Abdel Halim, Harris, Adrian L, Harris, James, Hasler, Udo, Hasselblatt, Peter, Hasui, Kazuhisa, Hawley, Robert G, Hawley, Teresa S, He, Congcong, He, Cynthia Y, He, Fengtian, He, Gu, He, Rong-Rong, He, Xian-Hui, He, You-Wen, He, Yu-Ying, Heath, Joan K, Hebert, Marie-Josee, Heinzen, Robert A, Helgason, Gudmundur Vignir, Hensel, Michael, Henske, Elizabeth P, Her, Chengtao, Herman, Paul K, Hernandez, Agustin, Hernandez, Carlos, Hernandez-Tiedra, Sonia, Hetz, Claudio, Hiesinger, Robin P, Higaki, Katsumi, Hilfiker, Sabine, Hill, Bradford G, Hill, Joseph A, Hill, William D, Hino, Keisuke, Hofius, Daniel, Hofman, Paul, Hoeglinger, Guenter U, Hoehfeld, Joerg, Holz, Marina K, Hong, Yonggeun, Hood, David 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8. EXPRESSION OF MONOCYTE CHEMOTACTIC PROTEIN-3 IN HUMAN MONOCYTES EXPOSED TO THE MYCOBACTERIAL CELL WALL COMPONENT LIPOARABINOMANNAN
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Vouret-Craviari, Valérie, Cenzuales, Salvatore, Poli, Guido, and Mantovani, Alberto
- Published
- 1997
- Full Text
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9. THE THROMBIN RECEPTOR : ACTIVATION AND COUPLING TO MITOGENIC SIGNALING SYSTEMS
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Van Obberghen-Schilling, Ellen, Chambard, Jean-Claude, Vouret-Craviari, Valérie, Chen, Yao-hui, Grall, Dominique, and Pouysségur, Jacques
- Published
- 1995
- Full Text
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10. Post-translational and Activation-dependent Modifications of the G Protein-coupled Thrombin Receptor
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Vouret-Craviari, Valérie, Grall, Dominique, Chambard, Jean-Claude, Rasmussen, Ulla B., Pouysségur, Jacques, and Van Obberghen-Schilling, Ellen
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- 1995
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11. Distinct Mechanisms Regulate 5-HT2 and Thrombin Receptor Desensitization
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Vouret-Craviari, Valérie, Auberger, Patrick, Pouysségur, Jacques, and Van Obberghen-Schilling, Ellen
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- 1995
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12. Multimer Formation and Ligand Recognition by the Long Pentraxin PTX3: SIMILARITIES AND DIFFERENCES WITH THE SHORT PENTRAXINS C-REACTIVE PROTEIN AND SERUM AMYLOID P COMPONENT
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Bottazzi, Barbara, Vouret-Craviari, Valérie, Bastone, Antonio, De Gioia, Luca, Matteucci, Cristian, Peri, Giuseppe, Spreafico, Fabio, Pausa, Mario, D'Ettorre, Cinzia, Gianazza, Elisabetta, Tagliabue, Aldo, Salmona, Mario, Tedesco, Francesco, Introna, Martino, and Mantovani, Alberto
- Published
- 1997
- Full Text
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13. cDNA cloning and expression of a hamster α-thrombin receptor coupled to Ca2+ mobilization
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Rasmussen, Ulla B., Vouret-Craviari, Valérie, Jallat, Sophie, Schlesinger, Yasmin, Pagès, Gilles, Pavirani, Andrea, Lecocq, Jean-Pierre, Pouysségur, Jacques, and Van Obberghen-Schilling, Ellen
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- 1991
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14. Genetic and Pharmacological Inactivation of the Purinergic P2RX7 Receptor Dampens Inflammation but Increases Tumor Incidence in a Mouse Model of Colitis-Associated Cancer.
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Hofman, Paul, Cherfils-Vicini, Julien, Bazin, Marie, Ilie, Marius, Juhel, Thierry, Hébuterne, Xavier, Gilson, Eric, Schmid-Alliana, Annie, Boyer, Olivier, Adriouch, Sahil, and Vouret-Craviari, Valerie
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CANCER research , *COLITIS , *INFLAMMATORY bowel diseases , *T cells , *MAST cells , *CYTOKINES , *LABORATORY mice - Abstract
Colitis-associated cancer (CAC) is a complication of inflammatory bowel disease (IBD). Binding of extracellular ATP to the purinergic receptor P2RX7 has emerged as a critical event in controlling intestinal inflammation, acting to limit elevation of proinflammatory mast cells and cytokines and promote survival of regulatory T cells (Treg) and enteric neurons. In this study, we investigated the effect of P2RX7 blockade in an established mouse model of CAC. Using genetic and pharmacologic tools, we found unexpectedly that while P2RX7 mediated inflammatory responses, it also acted at an early time to suppress CAC development. P2RX7 blockade enhanced proliferation of intestinal epithelial cells and protected them from apoptosis. The proliferative effects of P2RX7 blockade were associated with an increased production of TGFb1 that was sufficient to stimulate the proliferation of intestinal epithelial cells. Finally, P2RX7 blockade also altered immune cell infiltration and promoted Treg accumulation within lesions of the digestive system. Taken together, our findings reveal an unexpected role for P2RX7 in preventing CAC, suggesting cautions in the use of P2RX7 inhibitors to treat IBD given the possibility of increasing risks CAC as a result. [ABSTRACT FROM AUTHOR]
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- 2015
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15. To inhibit or to boost the ATP/P2RX7 pathway to fight cancer, that is the question
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Jonathan Benzaquen, Paul Hofman, Serena Janho Dit Hreich, Valérie Vouret-Craviari, Vouret-Craviari, Valerie, Institut de Recherche sur le Cancer et le Vieillissement (IRCAN), Université Nice Sophia Antipolis (1965 - 2019) (UNS), COMUE Université Côte d'Azur (2015-2019) (COMUE UCA)-COMUE Université Côte d'Azur (2015-2019) (COMUE UCA)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS)-Université Côte d'Azur (UCA), Centre de Lutte contre le Cancer Antoine Lacassagne [Nice] (UNICANCER/CAL), UNICANCER-Université Côte d'Azur (UCA), Hôpital Pasteur [Nice] (CHU), and Université Nice Sophia Antipolis (... - 2019) (UNS)
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tumors ,antitumor immunity ,[SDV.CAN]Life Sciences [q-bio]/Cancer ,ectonucleases ,[SDV.IMM.II]Life Sciences [q-bio]/Immunology/Innate immunity ,03 medical and health sciences ,Cellular and Molecular Neuroscience ,0302 clinical medicine ,Immune system ,Adenosine Triphosphate ,purinergic signalling ,[SDV.CAN] Life Sciences [q-bio]/Cancer ,Neoplasms ,medicine ,Animals ,Humans ,Receptor ,Molecular Biology ,[SDV.IMM.II] Life Sciences [q-bio]/Immunology/Innate immunity ,030304 developmental biology ,Cell Proliferation ,0303 health sciences ,Invited Review ,Antitumor immunity ,business.industry ,Cancer ,Cell Biology ,P2RX7 ,Human physiology ,[SDV.IMM.IMM]Life Sciences [q-bio]/Immunology/Immunotherapy ,Purinergic signalling ,immunotherapies ,medicine.disease ,3. Good health ,Large pore ,030220 oncology & carcinogenesis ,Cancer research ,Immunotherapy ,Receptors, Purinergic P2X7 ,[SDV.IMM.IMM] Life Sciences [q-bio]/Immunology/Immunotherapy ,business ,P2X7 ,Signal Transduction - Abstract
International audience; Despite new biological insights and recent therapeutic advances, many tumors remain at baseline during treatments. Therefore, there is an urgent need to find new therapeutic strategies to improve the care of patients with solid tumors. P2RX7 receptor (P2XR7), an ATPgated ion channel characterized by its ability to form large pore within the cell membrane, is described by most of the investigators as a "chef d'orchestre" of the antitumor immune response. The purpose of this review is to detail the recent information concerning different cellular mechanisms linking P2RX7 to hallmarks of cancer and to discuss different progresses in elucidating how activation of the ATP/P2RX7/NLRP3/IL-18 pathway is a very promising approach to fight cancer progression by increasing antitumor immune responses.
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- 2021
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16. P2RX7B is a new theranostic marker for lung adenocarcinoma patients
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Vouret-Craviari, Valérie, Infection bactérienne, inflammation, et carcinogenèse digestive, Université Nice Sophia Antipolis (... - 2019) (UNS), COMUE Université Côte d'Azur (2015-2019) (COMUE UCA)-COMUE Université Côte d'Azur (2015-2019) (COMUE UCA)-IFR50-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Côte d'Azur (UCA), Université Nice Sophia Antipolis (1965 - 2019) (UNS), and Vouret-Craviari, Valerie
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[SDV.CAN] Life Sciences [q-bio]/Cancer ,[SDV.CAN]Life Sciences [q-bio]/Cancer ,ComputingMilieux_MISCELLANEOUS - Abstract
International audience
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- 2020
17. Pyroglutamide-Based P2X7 Receptor Antagonists Targeting Inflammatory Bowel Disease
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Nicolas Renault, Rogatien Charlet, Sahil Adriouch, Germain Homerin, Davy Baudelet, Guillaume Ragé, Jean-François Wiart, Emmanuelle Lipka, Samir Jawhara, Boualem Sendid, Camille Richeval, Alina Ghinet, Christophe Furman, Amaury Farce, Régis Millet, Jordan Leroy, Benoît Rigo, Pierrick Dufrénoy, Valérie Vouret-Craviari, Mélodie Phanithavong, Xavier Dezitter, Delphine Allorge, Institute for Translational Research in Inflammation - U 1286 (INFINITE (Ex-Liric)), Institut National de la Santé et de la Recherche Médicale (INSERM)-Université de Lille-Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille), Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille), Physiopathologie et biothérapies des maladies inflammatoires et autoimmunes, Université de Rouen Normandie (UNIROUEN), Normandie Université (NU)-Normandie Université (NU)-Institut National de la Santé et de la Recherche Médicale (INSERM), Institut de Recherche sur le Cancer et le Vieillissement (IRCAN), Université Nice Sophia Antipolis (1965 - 2019) (UNS), COMUE Université Côte d'Azur (2015-2019) (COMUE UCA)-COMUE Université Côte d'Azur (2015-2019) (COMUE UCA)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS)-Université Côte d'Azur (UCA), Alexandru Ioan Cuza University of Iași [Romania], Vouret-Craviari, Valerie, Université de Lille, CNRS, Lille Inflammation Research International Center - U 995 [LIRIC], Lille Inflammation Research International Center (LIRIC) - U995, Institut de Recherche Translationnelle sur l'Inflammation (INFINITE) - U1286, Toxicologie et Génopathies [CHRU Lille], Laboratoire d'hématologie [Rouen], Institut de Recherche sur le Cancer et le Vieillissement [IRCAN], Lille Inflammation Research International Center - U 995 (LIRIC), Institut Pasteur de Lille, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université de Lille-Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille), Laboratoire de pharmacochimie [Lille], Université catholique de Lille (UCL)-Hautes Etudes d’Ingénieur [Lille] (HEI), JUNIA (JUNIA), Université catholique de Lille (UCL)-Université catholique de Lille (UCL)-JUNIA (JUNIA), Université catholique de Lille (UCL), Institut de Chimie Pharmaceutique Albert Lespagnol (ICPAL), Université de Lille, Droit et Santé, Laboratoire de Chimie Analytique (EA 4034), Pôle de Biologie Pathologie Génétique [CHU Lille], Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille)-Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille), Institute for Research and Innovation in Biomedicine (IRIB), Normandie Université (NU)-Normandie Université (NU)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), We gratefully acknowledge the 'Métropole Européenne de Lille' (MEL) for G.H.’s Ph.D. scholarship, the DigestScience Foundation (Foundation on Digestive Tract Diseases and Nutrition), and the Institut National du Cancer, France (INCa, project P2X7 2015–137) for financial support., We also acknowledge the National Cancer Institute (NCI) for biological evaluation of compounds on their 60-cell panel: the testing was performed by the Developmental Therapeutics Program, Division of Cancer Treatment and Diagnosis (the URL to the Program’s website: http://dtp.cancer.gov). We thank Thierry Juhel (IRCAN) for assistance with animals. We thank Nathalie Jouy and Emilie Floquet of the BICeL Facility for access to systems and technical advices in flow cytometry., and Université Nice Sophia Antipolis (... - 2019) (UNS)
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[CHIM.THER]Chemical Sciences/Medicinal Chemistry ,Pharmacology ,P2RX7 ,01 natural sciences ,Inflammatory bowel disease ,Mice ,Drug Delivery Systems ,Drug Discovery ,Pyroglutamic acid ,Pyroglutamides ,Pyrroloimidazolediones ,IBD ,Inflammation ,Purinergic receptors ,Receptor ,P2x7 receptor ,media_common ,0303 health sciences ,Chemistry ,Dextran Sulfate ,3. Good health ,Molecular Medicine ,[SDV.IMM]Life Sciences [q-bio]/Immunology ,Female ,Drug ,Purinergic P2X Receptor Antagonists ,Cell Survival ,media_common.quotation_subject ,[CHIM.THER] Chemical Sciences/Medicinal Chemistry ,03 medical and health sciences ,medicine ,[CHIM]Chemical Sciences ,Animals ,Humans ,[SDV.BBM]Life Sciences [q-bio]/Biochemistry, Molecular Biology ,Colitis ,030304 developmental biology ,HEK 293 cells ,Antagonist ,[SDV.MHEP.HEG]Life Sciences [q-bio]/Human health and pathology/Hépatology and Gastroenterology ,medicine.disease ,Inflammatory Bowel Diseases ,[SDV.MHEP.HEG] Life Sciences [q-bio]/Human health and pathology/Hépatology and Gastroenterology ,0104 chemical sciences ,Mice, Inbred C57BL ,010404 medicinal & biomolecular chemistry ,HEK293 Cells ,Docking (molecular) ,Receptors, Purinergic P2X7 - Abstract
International audience; This report deals with the design, the synthesis, and the pharmacological evaluation of pyroglutamide-based P2X7 antagonists. A dozen were shown to possess improved properties, among which inhibition of YO-PRO-1/TO-PRO-3 uptake and IL1β release upon BzATP activation of the receptor and dampening signs of DSS-induced colitis on mice, in comparison with reference antagonist GSK1370319A. Docking study and biological evaluation of synthesized compounds has highlighted new SAR, and low toxicity profiles of pyroglutamides herein described are clues for the finding of a usable h-P2X7 antagonist drug. Such a drug would raise the hope for a cure to many P2X7-dependent pathologies, including inflammatory, neurological, and immune diseases.
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- 2020
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18. Alternative splicing of P2RX7 pre-messenger RNA in health and diseases: Myth or reality?
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Jonathan, Benzaquen, Simon, Heeke, Séréna, Janho Dit Hreich, Laetitia, Douguet, Charles Hugo, Marquette, Paul, Hofman, Valérie, Vouret-Craviari, Centre Hospitalier Universitaire de Nice (CHU Nice), COMUE Université Côte d'Azur (2015-2019) (COMUE UCA), Université Côte d'Azur (UCA), Institut Necker Enfants-Malades (INEM - UM 111 (UMR 8253 / U1151)), Université Paris Descartes - Paris 5 (UPD5)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), Hôpital Pasteur [Nice] (CHU), Immunité muqueuse et vaccination, Institut National de la Santé et de la Recherche Médicale (INSERM), Infection bactérienne, inflammation, et carcinogenèse digestive, Université Nice Sophia Antipolis (... - 2019) (UNS), COMUE Université Côte d'Azur (2015-2019) (COMUE UCA)-COMUE Université Côte d'Azur (2015-2019) (COMUE UCA)-IFR50-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Côte d'Azur (UCA), Université Nice Sophia Antipolis (1965 - 2019) (UNS), and Vouret-Craviari, Valerie
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lcsh:R5-920 ,Onco-immunology ,Purinergic signaling ,[SDV]Life Sciences [q-bio] ,[SDV.CAN]Life Sciences [q-bio]/Cancer ,Review Article ,ATP ,[SDV] Life Sciences [q-bio] ,Alternative Splicing ,Adenosine Triphosphate ,lcsh:Biology (General) ,[SDV.CAN] Life Sciences [q-bio]/Cancer ,Animals ,Humans ,RNA, Messenger ,Receptors, Purinergic P2X7 ,lcsh:Medicine (General) ,P2X7 ,lcsh:QH301-705.5 ,ComputingMilieux_MISCELLANEOUS ,Cell Proliferation - Abstract
Alternative splicing (AS) tremendously increases the use of genetic information by generating protein isoforms that differ in protein–protein interactions, catalytic activity and/or subcellular localization. This review is not dedicated to AS in general, but rather we focus our attention on AS of P2RX7 pre-mRNA. Whereas P2RX7 mRNA is expressed by virtually all eukaryotic mammalian cells, the expression of this channel receptor is restrained to certain cells. When expressed at the cell membrane, P2RX7 controls downstream events including release of inflammatory molecules, phagocytosis, cell proliferation and death and metabolic events. Therefore, P2RX7 is an important actor of health and diseases. In this review, we summarize the general mechanisms leading to AS. Further, we recapitulate our current knowledge concerning the functional regions in P2RX7, identified at the genetic or exonic levels, and how AS may affect the expression of these regions. Finally, the potential of P2RX7 splice variants to control the fate of cancer cells is discussed. Keywords: Purinergic signaling, P2X7, ATP, Onco-immunology
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- 2019
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