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Your search keyword '"Vogt, Thomas F"' showing total 247 results

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1. Huntington’s disease cellular phenotypes are rescued non-cell autonomously by healthy cells in mosaic telencephalic organoids

3. Contributors

4. DNA methylation study of Huntington's disease and motor progression in patients and in animal models.

5. MicroRNA signatures of endogenous Huntingtin CAG repeat expansion in mice.

6. Huntington's disease accelerates epigenetic aging of human brain and disrupts DNA methylation levels

7. Huntingtin loss in hepatocytes is associated with altered metabolism, adhesion, and liver zonation

8. Protein-coding variants implicate novel genes related to lipid homeostasis contributing to body-fat distribution

9. Huntingtin loss in hepatocytes is associated with altered metabolism, adhesion, and liver zonation

12. Protein-altering variants associated with body mass index implicate pathways that control energy intake and expenditure in obesity

13. Conformational dynamics and DNA recognition by human MutSbeta

14. Supplementary Figures 1-4 from PDK1 Attenuation Fails to Prevent Tumor Formation in PTEN-Deficient Transgenic Mouse Models

15. Mapping brain gene coexpression in daytime transcriptomes unveils diurnal molecular networks and deciphers perturbation gene signatures

16. Developing HDAC4-Selective Protein Degraders To Investigate the Role of HDAC4 in Huntington’s Disease Pathology

17. Publisher Correction: Protein-altering variants associated with body mass index implicate pathways that control energy intake and expenditure in obesity

18. Supplement to: Myocardial Infarction Genetics and CARDIoGRAM Exome Consortia Investigators. Coding variation in ANGPTL4, LPL, and SVEP1 and the risk of coronary disease.

19. Rare and low-frequency coding variants alter human adult height

20. Publisher Correction: Protein-altering variants associated with body mass index implicate pathways that control energy intake and expenditure in obesity

21. HDinHD: A Rich Data Portal for Huntington’s Disease Research

23. Shape deformation analysis reveals the temporal dynamics of cell-type-specific homeostatic and pathogenic responses to mutant huntingtin

26. Shape deformation analysis reveals the temporal dynamics of cell-type-specific homeostatic and pathogenic responses to mutant huntingtin

27. Author response: Shape deformation analysis reveals the temporal dynamics of cell-type-specific homeostatic and pathogenic responses to mutant huntingtin

30. Large scale transgenic and cluster deletion analysis of the HoxD complex separate an ancestral regulatory module from evolutionary innovation

31. Ultrasensitive quantitative measurement of huntingtin phosphorylation at residue S13

32. Fringe is a glycosyltransferase that modifies Notch

33. Antagonists go out on a limb

35. Genetic and physical mapping of the mouse Ulnaless locus

41. 'Formins': proteins deduced from the alternative transcripts of the limb deformity gene

42. Protein-altering variants associated with body mass index implicate pathways that control energy intake and expenditure in obesity

43. Coding Variation in ANGPTL4, LPL, and SVEP1 and the Risk of Coronary Disease (vol 374, pg 1134, 2016)

44. Genetic invalidation of Lp-PLA2 as a therapeutic target: Large-scale study of five functional Lp-PLA2-lowering alleles

45. Systematic evaluation of pleiotropy identifies 6 further loci associated with coronary artery disease

46. Genetic invalidation of Lp-PLA2 as a therapeutic target:Large-scale study of five functional Lp-PLA2-lowering alleles

47. Systematic Evaluation of Pleiotropy Identifies 6 Further Loci Associated With Coronary Artery Disease

48. Coding Variation in ANGPTL4, LPL, and SVEP1 and the Risk of Coronary Disease

49. Disruption of formin-encoding transcripts in two mutant limb deformity alleles

50. Trans-ancestry meta-analyses identify rare and common variants associated with blood pressure and hypertension

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