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2. Immunologic resilience and COVID-19 survival advantage

Author

Abdalla, Mohamed I., Adams, Sandra G., Agnew, Joseph, Ali, Saleem, Barker, Jennifer, Birdwell, Angela, Bradford, Stephen, Briggs, Heather, Marin Corral, Judith, Dacus, Jennifer J., Danaher, Patrick J., DePaul, Scott A., Dickerson, Jill, Doanne, Jollynn, Elbel, Samantha, Escamilla, Corina, Farrar, Robert, Feldman, David, Flynn, Julianne, Ford, Delvina, Foy, Joanna D., Freeman, Megan, Galley, Samantha, Garza, Maritza, Gilman, Sherraine, Gomez, Jennifer, Goyal, Varun K., Grassmuck, Sally, Hanson, Joshua, Harris, Brande, Hastings, Gabrielyd, Haywood, Audrey, Hinojosa, Cecilia, Ho, Tony T., Hopkins, Teri, Jewell, Pamela, Johnson, Thomas B., Kotogiannes, Vasiliki, Lawler, Austin C., Lester, Chadwick S., Levine, Stephanie M., Lewis, Haidee V., Louder, Angel, Mainor, Charmaine, Maldonado, Rachel, Martinez, Yvette, McElligott, Neil, Medlin, Laura, Mireles, Myra, Morneau, Kathleen, Munro, Samuel B., Nambiar, Anoop, Nassery, Daniel, Nathanson, Robert, O’Rorke, Jane, Padgett, Cheryl, Pascual-Guardia, Sergi, Patterson, Marisa, Perez, Rogelio, Phillips, Robert E., Polk, Patrick B., Pomager, Michael A., Preston, Kristy J., Proud, Kevin C., Rangel, Michelle, Ratcliffe, Temple A., Reichelderfer, Renee L., Renz, Evan M., Ross, Jeanette, Rudd, Teresa, Sanchez, Maria E., Sanders, Tammy, Schindler, Kevin C., Schmit, David, Solorzano, Claudio, Soni, Nilam, Tam, Win S., Tovar, Edward J., Tyler, Anna R., Vasquez, Anjuli, Veloso, Maria C., Venticinque, Steven G., Villalpando, Jorge A., Villanueva, Melissa, Villegas, Lauren, Wallace, Andrew, Wang, Emily, Williamson, Andreia, Trammell Velasquez, Sadie A., Yunes, Andrea, Zentner, Katharine H., Lee, Grace C., Restrepo, Marcos I., Harper, Nathan, Manoharan, Muthu Saravanan, Smith, Alisha M., Meunier, Justin A., Sanchez-Reilly, Sandra, Ehsan, Aamir, Branum, Anne P., Winter, Caitlyn, Winter, Lauryn, Jimenez, Fabio, Pandranki, Lavanya, Carrillo, Andrew, Perez, Graciela L., Anzueto, Antonio, Trinh, Hanh, Lee, Monica, Hecht, Joan M., Martinez-Vargas, Celida, Sehgal, Raj T., Cadena, Jose, Walter, Elizabeth A., Oakman, Kimberly, Benavides, Raymond, Pugh, Jacqueline A., Letendre, Scott, Steri, Maristella, Orrù, Valeria, Fiorillo, Edoardo, Cucca, Francesco, Moreira, Alvaro G., Zhang, Nu, Leadbetter, Elizabeth, Agan, Brian K., Richman, Douglas D., He, Weijing, Clark, Robert A., Okulicz, Jason F., and Ahuja, Sunil K.

Published
2021
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4. Immunologic resilience and COVID-19 survival advantage

Author

Lee, Grace C., primary, Restrepo, Marcos I., additional, Harper, Nathan, additional, Manoharan, Muthu Saravanan, additional, Smith, Alisha M., additional, Meunier, Justin A., additional, Sanchez-Reilly, Sandra, additional, Ehsan, Aamir, additional, Branum, Anne P., additional, Winter, Caitlyn, additional, Winter, Lauryn, additional, Jimenez, Fabio, additional, Pandranki, Lavanya, additional, Carrillo, Andrew, additional, Perez, Graciela L., additional, Anzueto, Antonio, additional, Trinh, Hanh, additional, Lee, Monica, additional, Hecht, Joan M., additional, Martinez-Vargas, Celida, additional, Sehgal, Raj T., additional, Cadena, Jose, additional, Walter, Elizabeth A., additional, Oakman, Kimberly, additional, Benavides, Raymond, additional, Pugh, Jacqueline A., additional, Letendre, Scott, additional, Steri, Maristella, additional, Orrù, Valeria, additional, Fiorillo, Edoardo, additional, Cucca, Francesco, additional, Moreira, Alvaro G., additional, Zhang, Nu, additional, Leadbetter, Elizabeth, additional, Agan, Brian K., additional, Richman, Douglas D., additional, He, Weijing, additional, Clark, Robert A., additional, Okulicz, Jason F., additional, Ahuja, Sunil K., additional, Abdalla, Mohamed I., additional, Adams, Sandra G., additional, Agnew, Joseph, additional, Ali, Saleem, additional, Barker, Jennifer, additional, Birdwell, Angela, additional, Bradford, Stephen, additional, Briggs, Heather, additional, Marin Corral, Judith, additional, Dacus, Jennifer J., additional, Danaher, Patrick J., additional, DePaul, Scott A., additional, Dickerson, Jill, additional, Doanne, Jollynn, additional, Elbel, Samantha, additional, Escamilla, Corina, additional, Farrar, Robert, additional, Feldman, David, additional, Flynn, Julianne, additional, Ford, Delvina, additional, Foy, Joanna D., additional, Freeman, Megan, additional, Galley, Samantha, additional, Garza, Maritza, additional, Gilman, Sherraine, additional, Gomez, Jennifer, additional, Goyal, Varun K., additional, Grassmuck, Sally, additional, Hanson, Joshua, additional, Harris, Brande, additional, Hastings, Gabrielyd, additional, Haywood, Audrey, additional, Hinojosa, Cecilia, additional, Ho, Tony T., additional, Hopkins, Teri, additional, Jewell, Pamela, additional, Johnson, Thomas B., additional, Kotogiannes, Vasiliki, additional, Lawler, Austin C., additional, Lester, Chadwick S., additional, Levine, Stephanie M., additional, Lewis, Haidee V., additional, Louder, Angel, additional, Mainor, Charmaine, additional, Maldonado, Rachel, additional, Martinez, Yvette, additional, McElligott, Neil, additional, Medlin, Laura, additional, Mireles, Myra, additional, Morneau, Kathleen, additional, Munro, Samuel B., additional, Nambiar, Anoop, additional, Nassery, Daniel, additional, Nathanson, Robert, additional, O’Rorke, Jane, additional, Padgett, Cheryl, additional, Pascual-Guardia, Sergi, additional, Patterson, Marisa, additional, Perez, Rogelio, additional, Phillips, Robert E., additional, Polk, Patrick B., additional, Pomager, Michael A., additional, Preston, Kristy J., additional, Proud, Kevin C., additional, Rangel, Michelle, additional, Ratcliffe, Temple A., additional, Reichelderfer, Renee L., additional, Renz, Evan M., additional, Ross, Jeanette, additional, Rudd, Teresa, additional, Sanchez, Maria E., additional, Sanders, Tammy, additional, Schindler, Kevin C., additional, Schmit, David, additional, Solorzano, Claudio, additional, Soni, Nilam, additional, Tam, Win S., additional, Tovar, Edward J., additional, Tyler, Anna R., additional, Vasquez, Anjuli, additional, Veloso, Maria C., additional, Venticinque, Steven G., additional, Villalpando, Jorge A., additional, Villanueva, Melissa, additional, Villegas, Lauren, additional, Wallace, Andrew, additional, Wang, Emily, additional, Williamson, Andreia, additional, Trammell Velasquez, Sadie A., additional, Yunes, Andrea, additional, and Zentner, Katharine H., additional

Published
2021
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6. Facilitation of quantal release induced by a D1-like receptor on bovine chromaffin cells

Author

Villanueva, Melissa and Wightman, R. Mark

Subjects
Bovine somatotropin -- Structure, Bovine somatotropin -- Research, Chromaffin cells -- Structure, Chromaffin cells -- Chemical properties, Exocytosis -- Research, Biological sciences, Chemistry
Abstract

The role of dopaminergic receptors on exocytosis from bovine adrenal chromaffin cells is reexamined by using a brief (subsecond) chemical depolarizing stimuli. It is indicated that released catecholamines enhance their own release by interaction with a D1-like receptor on bovine adrenal chromaffin cells.

Published
2007

12. Administrative Record for the 2012 Add-Alter Projects at Cannon Air Force Base

Author

Estes, Bob, Rierson, Michael, Hamilton, Candis, Villanueva, Melissa, and Figiera, Anthony S.

Subjects
Fitness Center, New Mexico (State / Territory), Weapons Cleaning Room, Cannon Air Force Base, Parking Lot, Building 2315, Curry (County)
Abstract

Correspondence and records regarding the finding of no effect for a series of projects as part of the AFSOC beddown requirements on Cannon AFB for 2012.

Published
2012
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13. Electrochemical detection of modulation of exocytosis from chromaffin cells monitored with amperometry

Author

Villanueva, Melissa

Abstract

Biological intercellular communication necessitates rapid and efficient processes. Cells must be able to relay and receive messages rapidly and this is accomplished readily through exocytosis. Chemical messengers are packaged into membrane-bound organelles known as vesicles or granules, which fuse with the outer plasma membrane upon an increase in intracellular [Ca2+] caused by influx from the extracellular space or from intracellular stores. Ca2+ increases can be effected through membrane depolarization or through receptor activation. The chemical messengers are then extruded into the extracellular space where they can bind to target cells and initiate a response. Because of the timescale of vesicular release and Ca2+ influx, it is necessary to utilize detection techniques that have sufficient temporal resolution. Additionally, the minute amounts that are secreted from these vesicles, whose dimensions are characteristically at the nanometer level, require a technique that possesses sensitivity sufficient to detect them. The electrochemical technique, constant potential amperometry, used in conjunction with carbon-fiber microelectrodes, offers excellent temporal and spatial resolution as well as sensitivity for electroactive chemical messengers. Moreover, the use of a fluorescent dye, fura-2 allows study of the Ca2+ dynamics occurring during exocytosis. Both amperometry and fluorescence measurements via fura-2 allow for the characterization of exocytosis in bovine and murine chromaffin cells. Amperometry has been advantageous in studies involving chromaffin cells. Using this technique, an autoreceptor effect that causes facilitation in bovine chromaffin cells was revealed. Since this autoreceptor effect appears to occur on very rapid timescales, the exquisite temporal resolution of amperometry has permitted the observation and manipulation of this effect with minimal perturbation of the cells under study. In experiments involving genetically altered mice, amperometry revealed aspects of the exocytotic process that may be affected by specific proteins. In studies conducted on R6/2 mice that serve as models for Huntington's Disease, it was noted that vesicular quanta was significantly diminished. Also, in other experiments conducted in mice that lack the intracellular phosphoprotein, synapsin, vesicular release frequency was significantly enhanced. The studies mentioned demonstrate that amperometry with carbon-fiber microelectrodes is a powerful tool in studying exocytotic processes.

Published
2006
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18. KiSS1 suppresses TNFalpha-induced breast cancer cell invasion via an inhibition of RhoA-mediated NF-kappaB activation.

Author

Cho SG, Li D, Stafford LJ, Luo J, Rodriguez-Villanueva M, Wang Y, and Liu M

Subjects
Cell Adhesion, Cell Movement, Cell Proliferation, Down-Regulation, Female, Humans, Kisspeptins, NF-kappa B metabolism, Oligopeptides metabolism, Breast Neoplasms pathology, NF-kappa B antagonists & inhibitors, Neoplasm Invasiveness, Tumor Necrosis Factor-alpha pharmacology, Tumor Suppressor Proteins physiology, rhoA GTP-Binding Protein antagonists & inhibitors
Abstract

Tumor necrosis factor-alpha (TNFalpha) induces cancer development and metastasis, which is prominently achieved by nuclear factor-kappa B (NF-kappaB) activation. TNFalpha-induced NF-kappaB activation enhances cellular mechanisms including proliferation, migration, and invasion. KiSS1, a key regulator of puberty, was initially discovered as a tumor metastasis suppressor. The expression of KiSS1 was lost or down-regulated in different metastatic tumors. However, it is unclear whether KiSS1 regulates TNFalpha-induced NF-kappaB activation and further tumor cell migration. In this study, we demonstrate that KiSS1 suppresses the migration of breast cancer cells by inhibiting TNFalpha-induced NF-kappaB pathway and RhoA activation. Both KiSS1 overexpression and KP10 (kisspeptin-10) stimulation inhibited TNFalpha-induced NF-kappaB activity, suppressed TNFalpha-induced cell migration and cell attachment to fibronectin in breast cancer cells while KP10 has little effect on cancer cell proliferation. Furthermore, KP10 inhibited TNFalpha-induced cell migration and RhoA GTPase activation. Therefore, our data demonstrate that KiSS1 inhibits TNFalpha-induced NF-kappaB activation via downregulation of RhoA activation and suppression of breast cancer cell migration and invasion., ((c) 2009 Wiley-Liss, Inc.)

Published
2009
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