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1. Chromatin loop anchors are associated with genome instability in cancer and recombination hotspots in the germline

2. When TADs go bad: chromatin structure and nuclear organisation in human disease [version 1; referees: 2 approved]

3. Mutational bias in spermatogonia impacts the anatomy of regulatory sites in the human genome

4. Retrospective evaluation of whole exome and genome mutation calls in 746 cancer samples

5. Mutational Biases Drive Elevated Rates of Substitution at Regulatory Sites across Cancer Types

6. Nonrandom Gene Loss from the Drosophila miranda Neo-Y Chromosome

7. Evolution of Sex Chromosomes in Insects

8. Nucleotide diversity inSilene latifoliaautosomal and sex-linked genes

9. Out of the Andes: patterns of diversification in clearwing butterflies

10. Genetic Recombination and Molecular Evolution

11. Mutational Biases Drive Elevated Rates of Substitution at Regulatory Sites across Cancer Types

12. Insertion Events of CR1 Retrotransposable Elements Elucidate the Phylogenetic Branching Order in Galliform Birds

13. NONRANDOM DISTRIBUTION OF GENES WITH SEX-BIASED EXPRESSION IN THE CHICKEN GENOME

14. Gene loss on Y chromosomes: a consequence of purifying selection?

15. Conservation and de novo acquisition of dosage compensation on newly evolved sex chromosomes in Drosophila

16. The epigenome of evolving Drosophila neo-sex chromosomes: dosage compensation and heterochromatin formation

17. A new plant sex-linked gene with high sequence diversity and possible introgression of the X copy

18. Muller's Ratchet and the Degeneration of the Drosophila miranda Neo-Y Chromosome

19. Slcyt, a newly identified sex-linked gene, has recently moved onto the X chromosome in Silene latifolia (Caryophyllaceae)

20. The effects of deleterious mutations on evolution in non-recombining genomes

21. De novo transcriptome assembly reveals sex-specific selection acting on evolving neo-sex chromosomes in Drosophila miranda

22. The epigenome of evolving Drosophila neo-sex chromosomes: dosage compensation and heterochromatin formation.

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