755 results on '"Vasmatzis, George"'
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2. Unraveling the genomic underpinnings of unbalanced MYC break-apart FISH results using whole genome sequencing analysis
3. Individualized Cell-Free DNA Monitoring With Chromosomal Junctions for Mesothelioma
4. Subtype of SCLC Is an Intrinsic and Persistent Feature Through Systemic Treatment
5. FGFR2-IIIb Expression by Immunohistochemistry Has High Specificity in Cholangiocarcinoma with FGFR2 Genomic Alterations
6. Identification of EWSR1 rearrangements in patients with immature hematopoietic neoplasms: A case series and review of literature
7. The Role of Hydrophobicity in Peptide-MHC Binding
8. Tumor Junction Burden and Antigen Presentation as Predictors of Survival in Mesothelioma Treated With Immune Checkpoint Inhibitors
9. Evaluation of tumor response to immune checkpoint inhibitors by a 3D immunotumoroid model
10. Supplementary Figure from NKG7 Is a T-cell–Intrinsic Therapeutic Target for Improving Antitumor Cytotoxicity and Cancer Immunotherapy
11. Supplementary Data from NKG7 Is a T-cell–Intrinsic Therapeutic Target for Improving Antitumor Cytotoxicity and Cancer Immunotherapy
12. Complex balanced intrachromosomal rearrangement involving PITX2 identified as a cause of Axenfeld‐Rieger Syndrome
13. Evaluating NetMHCpan performance on non-European HLA alleles not present in training data
14. Evasive Spike Variants Elucidate the Preservation of T Cell Immune Response to the SARS-CoV-2 Omicron Variant
15. Clinical utility of next generation sequencing to detect IGH/IL3 rearrangements [t(5;14)(q31.1;q32.1)] in B-lymphoblastic leukemia/lymphoma
16. In search for biomarkers and potential drug targets for uterine serous endometrial cancer
17. Detection of cryptic CCND1 rearrangements in mantle cell lymphoma by next generation sequencing
18. EXTH-37. FUNCTIONAL GENOMICS IDENTIFIES THERAPEUTIC VULNERABILITIES OF GLIOMA SUBTYPES AND YAP/TAZ-ENRICHED TUMORS
19. INNV-20. PREDICTING RESPONSE TO CHEMOTHERAPY IN HIGH GRADE GLIOMAS USING EX VIVO 3D CELL CULTURE FUNCTIONAL PRECISION ONCOLOGY PLATFORM – A MULTI-INSTITUTIONAL EXPERIENCE
20. The Role of Hydrophobicity in Peptide-MHC Binding
21. Using Genomics to Differentiate Multiple Primaries From Metastatic Lung Cancer
22. Immune Cell Infiltration May Be a Key Determinant of Long-Term Survival in Small Cell Lung Cancer
23. Neoantigenic Potential of Complex Chromosomal Rearrangements in Mesothelioma
24. Constitutional chromosome rearrangements that mimic the 2017 world health organization “acute myeloid leukemia with recurrent genetic abnormalities”: A study of three cases and review of the literature
25. UCH-L1 bypasses mTOR to promote protein biosynthesis and is required for MYC-driven lymphomagenesis in mice
26. Geographically Biased Composition of NetMHCpan Training Datasets and Evaluation of MHC-Peptide Binding Prediction Accuracy on Novel Alleles
27. Subtype of Small Cell Lung Carcinoma is an Intrinsic and Persist Feature through Systemic Treatment
28. Myeloid malignancies with 5q and 7q deletions are associated with extreme genomic complexity, biallelic TP53 variants, and very poor prognosis
29. SVAtools for junction detection of genome-wide chromosomal rearrangements by mate-pair sequencing (MPseq)
30. Identification of a pyruvate-to-lactate signature in pancreatic intraductal papillary mucinous neoplasms
31. Characterization of a cryptic KMT2A/AFF1 gene fusion by mate-pair sequencing (MPseq) in a young adult with newly diagnosed B-lymphoblastic leukemia
32. Characterization of a t(1;2)(p36;p21) involving the PRDM16 gene region by mate-pair sequencing (MPseq) in a patient with newly diagnosed acute myeloid leukemia with myelodysplasia-related changes
33. Epigenomics may begin to explain in vitro differential response to hypomethylating agents in MMR-D hypermethylated endometrial cancer
34. Supplementary Table S2 from Repurposing Ceritinib Induces DNA Damage and Enhances PARP Inhibitor Responses in High-Grade Serous Ovarian Carcinoma
35. Data from Repurposing Ceritinib Induces DNA Damage and Enhances PARP Inhibitor Responses in High-Grade Serous Ovarian Carcinoma
36. Supplementary Figures 1-8 from Repurposing Ceritinib Induces DNA Damage and Enhances PARP Inhibitor Responses in High-Grade Serous Ovarian Carcinoma
37. Supplementary Materials and Methods from Repurposing Ceritinib Induces DNA Damage and Enhances PARP Inhibitor Responses in High-Grade Serous Ovarian Carcinoma
38. Data from NKG7 Is a T-cell–Intrinsic Therapeutic Target for Improving Antitumor Cytotoxicity and Cancer Immunotherapy
39. Supplementary Figure from NKG7 Is a T-cell–Intrinsic Therapeutic Target for Improving Antitumor Cytotoxicity and Cancer Immunotherapy
40. Supplementary Data from NKG7 Is a T-cell–Intrinsic Therapeutic Target for Improving Antitumor Cytotoxicity and Cancer Immunotherapy
41. Table S2 from YAP Tyrosine Phosphorylation and Nuclear Localization in Cholangiocarcinoma Cells Are Regulated by LCK and Independent of LATS Activity
42. Data from YAP Tyrosine Phosphorylation and Nuclear Localization in Cholangiocarcinoma Cells Are Regulated by LCK and Independent of LATS Activity
43. Supplemental Methods from YAP Tyrosine Phosphorylation and Nuclear Localization in Cholangiocarcinoma Cells Are Regulated by LCK and Independent of LATS Activity
44. Figure S1 from YAP Tyrosine Phosphorylation and Nuclear Localization in Cholangiocarcinoma Cells Are Regulated by LCK and Independent of LATS Activity
45. Supplemental Figure Legends from YAP Tyrosine Phosphorylation and Nuclear Localization in Cholangiocarcinoma Cells Are Regulated by LCK and Independent of LATS Activity
46. Genomic rearrangements in sporadic lymphangioleiomyomatosis: an evolving genetic story
47. Management of Multifocal Lung Cancer: Results of a Survey
48. A novel cytogenetic and molecular characterization of renal metanephric adenoma: Identification of partner genes involved in translocation t(9;15)(p24;q24)
49. TFEB-VEGFA (6p21.1) co-amplified renal cell carcinoma: a distinct entity with potential implications for clinical management
50. Investigation of somatic CNVs in brains of synucleinopathy cases using targeted SNCA analysis and single cell sequencing
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