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1. SEPN1-related myopathy depends on the oxidoreductase ERO1A and is druggable with the chemical chaperone TUDCA

6. Defective endoplasmic reticulum-mitochondria contacts and bioenergetics in SEPN1-related myopathy

12. The ER stress response mediator ERO1 triggers cancer metastasis by favoring the angiogenic switch in hypoxic conditions

14. Defective endoplasmic reticulum-mitochondria contacts and bioenergetics in SEPN1-related myopathy

15. SELENON (SEPN1) protects skeletal muscle from saturated fatty acid-induced ER stress and insulin resistance

16. A maladaptive ER stress response triggers dysfunction in highly active muscles of mice with SELENON loss

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