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6. Glioblastoma cell motility depends on enhanced oxidative stress coupled with mobilization of a sulfurtransferase

7. PATZ1 fusions define a novel molecularly distinct neuroepithelial tumor entity with a broad histological spectrum

10. Analysis of a pituitary tumour with histological features of central neurocytoma points towards the emergence of a new entity recognizable by a specific epigenetic signature

13. Desmoplastic myxoid tumor, SMARCB1-mutant: clinical, histopathological and molecular characterization of a pineal region tumor encountered in adolescents and adults

15. An Atypical Heel Pain

16. CELF2 Sustains a Proliferating/OLIG2+ Glioblastoma Cell Phenotype via the Epigenetic Repression of SOX3

21. The molecular landscape of glioma in patients with Neurofibromatosis 1

22. Inactivation of Pif1 helicase causes a mitochondrial myopathy in mice

23. A Positive Feed-forward Loop Associating EGR1 and PDGFA Promotes Proliferation and Self-renewal in Glioblastoma Stem Cells

26. Analysis of a pituitary tumour with histological features of central neurocytoma points towards the emergence of a new entity recognisable by a specific epigenetic signature.

27. Identification of Metabolomic Markers in Frozen or Formalin-Fixed and Paraffin-Embedded Samples of Diffuse Glioma from Adults.

28. Data from ERK-Mediated Loss of miR-199a-3p and Induction of EGR1 Act as a “Toggle Switch” of GBM Cell Dedifferentiation into NANOG- and OCT4-Positive Cells

29. Supplementary Data from ERK-Mediated Loss of miR-199a-3p and Induction of EGR1 Act as a “Toggle Switch” of GBM Cell Dedifferentiation into NANOG- and OCT4-Positive Cells

30. Supplementary tables S1-S3 from ERK-Mediated Loss of miR-199a-3p and Induction of EGR1 Act as a “Toggle Switch” of GBM Cell Dedifferentiation into NANOG- and OCT4-Positive Cells

31. figure legends and tables from ERK-Mediated Loss of miR-199a-3p and Induction of EGR1 Act as a “Toggle Switch” of GBM Cell Dedifferentiation into NANOG- and OCT4-Positive Cells

34. Changes in chromatin state reveal ARNT2 at a node of a tumorigenic transcription factor signature driving glioblastoma cell aggressiveness

35. A driver role for GABA metabolism in controlling stem and proliferative cell state through GHB production in glioma

39. Autophagy inactivation in osteosarcoma leads to the appearance of poor prognosis-associated factors

40. Tumor cells with neuronal intermediate progenitor features define a subgroup of 1p/19q co‐deleted anaplastic gliomas

45. Rosette‐forming glioneuronal tumours are midline, FGFR1‐mutated tumours

47. Human glioblastoma cell motility depends on the activity of the cysteine metabolism enzyme 3‐Mercaptopyruvate sulfurtransferase

48. Characteristics of diffuse hemispheric gliomas, H3 G34-mutant in adults

50. Prognostic Relevance of Histomolecular Classification of Diffuse Adult High-Grade Gliomas with Necrosis

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