34 results on '"Unni, Arun"'
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2. Genome-wide CRISPR screens in spheroid culture reveal that the tumor suppressor LKB1 inhibits growth via the PIKFYVE lipid kinase
3. MEK inhibitor resistance in lung adenocarcinoma is associated with addiction to sustained ERK suppression
4. Co-occurrence and mutual exclusivity: what cross-cancer mutation patterns can tell us
5. LKB1 suppresses growth and promotes the internalization of EGFR through the PIKFYVE lipid kinase
6. Integrative Genomic Analyses Identifies GGA2 as a Cooperative Driver of EGFR-Mediated Lung Tumorigenesis
7. Type 2 Diabetes: Evidence for Linkage on Chromosome 20 in 716 Finnish Affected Sib Pairs
8. Supplementary Data from Drug Sensitivity and Allele Specificity of First-Line Osimertinib Resistance EGFR Mutations
9. Supplementary cfDNA Mutation Dataset from Drug Sensitivity and Allele Specificity of First-Line Osimertinib Resistance EGFR Mutations
10. Data from Drug Sensitivity and Allele Specificity of First-Line Osimertinib Resistance EGFR Mutations
11. MEK inhibitor resistance in lung cancer cells associated with addiction to sustained ERK suppression
12. Intrinsic Sensor of Oncogenic Transformation Induces a Signal for Innate Immunosurveillance
13. CCL5 production in lung cancer cells leads to an altered immune microenvironment and promotes tumor development
14. Production Cost Optimization to Assess the Operational Export Potential of LAO PDR by 2025
15. Enhancement of anti-tumor CD8 immunity by IgG1-mediated targeting of Fc receptors
16. CCL5 production in lung cancer cells leads to an altered immune microenvironment and promotes tumor development.
17. Drug Sensitivity and Allele Specificity of First-Line Osimertinib Resistance EGFR Mutations
18. Abstract A38: Oncogenes drive production of immunosuppressive cytokines to facilitate lung cancer progression
19. A Large Set of Finnish Affected Sibling Pair Families With Type 2 Diabetes Suggests Susceptibility Loci on Chromosomes 6, 11, and 14
20. Generation of pulmonary neuroendocrine cells and SCLC-like tumors from human embryonic stem cells
21. Hyperactivation of ERK by multiple mechanisms is toxic to RTK-RAS mutation-driven lung adenocarcinoma cells
22. Author response: Hyperactivation of ERK by multiple mechanisms is toxic to RTK-RAS mutation-driven lung adenocarcinoma cells
23. Generation of pulmonary neuro-endocrine cells and tumors resembling small cell lung cancers from human embryonic stem cells
24. Hyperactivation of extracellular signal-regulated kinase (ERK) by RAS-mediated signaling or inhibition of dual specificity phosphatase 6 (DUSP6) is associated with toxicity in lung adenocarcinoma cells with mutations in KRAS or EGFR
25. P1.02-055 Synthetic Lethality Dictates the Mutual Exclusivity of Oncogenic Mutations in Lung Adenocarcinoma
26. How Cancer Genomics Drives Cancer Biology: Does Synthetic Lethality Explain Mutually Exclusive Oncogenic Mutations?
27. Integrative Genomic Analyses Identifies GGA2 as a Cooperative Driver of EGFR-Mediated Lung Tumorigenesis.
28. The Finland--United States Investigation of Non--Insulin-Dependent Diabetes Mellitus Genetics (FUSION) Study. II. An Autosomal Genome Scan for Diabetes-Related Quantitative-Trait Loci
29. The Finland--United States Investigation of Non--Insulin-Dependent Diabetes Mellitus Genetics (FUSION) Study. I. An Autosomal Genome Scan for Genes That Predispose to Type 2 Diabetes
30. Evidence that synthetic lethality underlies the mutual exclusivity of oncogenic KRAS and EGFR mutations in lung adenocarcinoma
31. Author response: Evidence that synthetic lethality underlies the mutual exclusivity of oncogenic KRAS and EGFR mutations in lung adenocarcinoma
32. Enhancement of anti-tumor CD8 immunity by IgG1-mediated targeting of Fc receptors
33. How Cancer Genomics Drives Cancer Biology: Does Synthetic Lethality Explain Mutually Exclusive Oncogenic Mutations?
34. LKB1 suppresses growth and promotes the internalization of EGFR through the PIKFYVE lipid kinase.
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