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1. A mouse model of inherited choline kinase β-deficiency presents with specific cardiac abnormalities and a predisposition to arrhythmia

2. Mechanism of action and therapeutic route for a muscular dystrophy caused by a genetic defect in lipid metabolism

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5. Cytotoxicity and exhaustion markers of chimeric antigen receptor T cells targeting BCMA in multiple myeloma cell lines between patients and healthy donors.

6. Cytotoxicity and exhaustion markers of chimeric antigen receptor T cells targeting BCMA in multiple myeloma cell lines between patients and healthy donors

7. Comparison of the efficacy of second and third generation lentiviral vector transduced CAR CD19 T cells for use in the treatment of acute lymphoblastic leukemia both in vitro and in vivo models

18. Orthogonal analysis of dystrophin protein and mRNA as a surrogate outcome for drug development

19. Vamorolone targets dual nuclear receptors to treat inflammation and dystrophic cardiomyopathy

20. Ryanodine channel complex stabilizer compound S48168/ARM210 as a disease modifier in dystrophin‐deficientmdxmice: proof‐of‐concept study and independent validation of efficacy

22. OPN‐a induces muscle inflammation by increasing recruitment and activation of pro‐inflammatory macrophages

23. Ryanodine channel complex stabilizer compound S48168/ARM210 as a disease modifier in dystrophin-deficient mdx mice: proof-of-concept study and independent validation of efficacy.

24. Elusive sources of variability of dystrophin rescue by exon skipping

25. Omigapil Treatment Decreases Fibrosis and Improves Respiratory Rate in dy2J Mouse Model of Congenital Muscular Dystrophy

26. VBP15, a Glucocorticoid Analogue, Is Effective at Reducing Allergic Lung Inflammation in Mice

29. Omigapil Treatment Decreases Fibrosis and Improves Respiratory Rate in dy2J Mouse Model of Congenital Muscular Dystrophy.

30. Myoblasts Isolated from Hypertrophy-Responsive Callipyge Muscles Show Altered Growth Rates and Increased Resistance to Serum Deprivation-Induced Apoptosis.

31. Osteopontin deficiency delays inflammatory infiltration and the onset of muscle regeneration in a mouse model of muscle injury