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9 results on '"Tomato, E."'

1. Impact of Mast Cells in Rejection of Allografts

Author

Kritas, S.K., primary, Saggini, A., additional, Varvara, G., additional, Murmura, G., additional, Caraffa, A., additional, Antinolfi, P., additional, Tomato, E., additional, Pantalone, A., additional, Neri, G., additional, Frydas, S., additional, Rosati, M., additional, Tei, M., additional, Speziali, A., additional, Saggini, R., additional, Pandolfi, F., additional, Cerulli, G., additional, Theoharides, T.C., additional, and Conti, P., additional

Published
2013
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2. Impact of IL-33 on PGD2 Generation by Activated Human Cord Blood-Derived Mast Cell: Lack of Effect on Tryptase Release

Author

Nicoletti, M., primary, Maccauro, G., additional, Tripodi, D., additional, Saggini, A., additional, Potalivo, G., additional, Castellani, M.L., additional, Conti, F., additional, Rosati, M., additional, Tomato, E., additional, Caraffa, A., additional, Antinolfi, P., additional, Conti, P., additional, and Theoharides, T.C., additional

Published
2012
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3. Tumor Necrosis Factor-Alpha and Mast Cells: Revisited Study

Author

Saggini, A., primary, Tripodi, D., additional, Maccauro, G., additional, Castellani, M.L., additional, Anogeianaki, A., additional, Teté, S., additional, Felaco, P., additional, De Luths, M.A., additional, Galzio, R., additional, Fulcheri, M., additional, Theoharides, T.C., additional, Caraffa, A., additional, Antinolfi, P., additional, Felaco, M., additional, Conti, F., additional, Neri, G., additional, Pandolfi, F., additional, Tomato, E., additional, and Shaik-Dasthagirisaheb, Y.B., additional

Published
2011
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7. Interleukin 27 (IL-27): A Novel Pleiotropic Cytokine Involved in T Cell Differentiation and T Cell Response Modulation

Author

Petrarca, C., primary, Frydas, S., additional, Donelan, J., additional, Boucher, W., additional, Papadopoulou, N., additional, Cao, J., additional, Castellani, M.L., additional, Conti, P., additional, Tomato, E., additional, Robuffo, I., additional, Vecchiet, J., additional, Iezzi, T., additional, Madhappan, B., additional, and Kempuraj, D., additional

Published
2005
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8. Endothelial Cell Anergy is Mediated by bFGF through the Sustained Activation of p38-MAPK and NF-?B Inhibition

Author

Flati, V., Pastore, L.I., Griffioen, A.W., Satijn, S., Tomato, E., D'Alimonte, I., Laglia, E., Marchetti, P., Gulino, A., and Martinotti, S.

Abstract

Tumors escape from immune surveillance by, among other mechanisms, the down- regulation of endothelial adhesion molecules, such as ICAM-1, and by unresponsiveness to inflammatory signals, a process mediated by angiogenic factors that is called endothelial cell anergy. Here we present the cell biological regulation of these processes. The angiogenic basic fibroblast growth factor (bFGF/FGF-2) was found to inhibit tumor necrosis factor-a (TNF-a)- induced elevation of ICAM-1, at transcriptional level. Furthermore, we found that bFGF inhibits the TNF-mediated activation of NF-?B by blocking phosphorylation and degradation of I?Ba. We also found that bFGF induces hyperphosphorylation of p38 MAPK on endothelial cells, whereas inhibition of such kinase abrogates the effect of bFGF on the TNF-mediated activation of NF-?B. Thus, we suggest that bFGF acts as an inhibitor of leukocyte adhesion in tumor vessels by decreasing the ICAM-1 expression through the sustained activation of p38-MAPK and via inhibition of NF-?B.

Published
2006
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