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351 results on '"Tejasvi, Trilokraj"'

4. Transethnic analysis of psoriasis susceptibility in South Asians and Europeans enhances fine mapping in the MHC and genome wide

Author

Stuart, Philip E., Tsoi, Lam C., Nair, Rajan P., Ghosh, Manju, Kabra, Madhulika, Shaiq, Pakeeza A., Raja, Ghazala K., Qamar, Raheel, Thelma, B.K., Patrick, Matthew T., Parihar, Anita, Singh, Sonam, Khandpur, Sujay, Kumar, Uma, Wittig, Michael, Degenhardt, Frauke, Tejasvi, Trilokraj, Voorhees, John J., Weidinger, Stephan, Franke, Andre, Abecasis, Goncalo R., Sharma, Vinod K., and Elder, James T.

Published
2022
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6. GATA-3 is a proto-oncogene in T-cell lymphoproliferative neoplasms

Author

Geng, Xiangrong, Wang, Chenguang, Gao, Xin, Chowdhury, Pinki, Weiss, Jonathan, Villegas, José A., Saed, Badeia, Perera, Thilini, Hu, Ying, Reneau, John, Sverdlov, Maria, Wolfe, Ashley, Brown, Noah, Harms, Paul, Bailey, Nathanael G., Inamdar, Kedar, Hristov, Alexandra C., Tejasvi, Trilokraj, Montes, Jaime, Barrionuevo, Carlos, Taxa, Luis, Casavilca, Sandro, de Pádua Covas Lage, J. Luís Alberto, Culler, Hebert Fabrício, Pereira, Juliana, Runge, John S., Qin, Tingting, Tsoi, Lam C., Hong, Hanna S., Zhang, Li, Lyssiotis, Costas A., Ohe, Rintaro, Toubai, Tomomi, Zevallos-Morales, Alejandro, Murga-Zamalloa, Carlos, and Wilcox, Ryan A.

Published
2022
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7. Direct to Patient Synchronous Teledermatology During COVID-19 Pandemic.

Author

Vaishampayan, Neil, Trupiano, Nicole, Goldberg, Rebecca, Zhang, Haihan, Tang, Michael, Chopra, Ahab, Castanedo-Tardan, Mari Paz, Renati, Sruthi, and Tejasvi, Trilokraj

Subjects
COVID-19 pandemic, MEDICAL care, ASIANS, MEDICAL offices, ODDS ratio
Abstract

Background: During the COVID-19 pandemic, teledermatology became a popular mode of health care delivery. Thus, deciphering which diagnoses are best suited for synchronous video visits is important to guide providers on appropriate patient care. Methods: We conducted a retrospective study of 1,647 submitted synchronous video visits from September 1, 2020 to March 31, 2021 at a single, large academic institution. Results: Video visits' follow-up rate was significantly associated with diagnosis subtype (p < 0.001). Compared with patients with skin lesions and nonskin dermatologic conditions, patients with a rash had higher odds of being recommended to have their follow-up visit as a video visit (odds ratio [OR] = 0.222, p < 0.001; OR = 0.296, p < 0.001). Patients with a rash had lower odds of being recommended to have their follow-up visit as an in-person office visit when compared with skin lesions (OR = 9.679, p < 0.001), nonskin dermatologic conditions (OR = 4.055, p < 0.001), and other skin dermatologic conditions (OR = 2.23, p < 0.01). Demographically, employed, middle-aged patients with private insurance made up the majority of video visit usage. African American patients were less likely to utilize a video visit compared with Asian patients (OR = 2.06, p < 0.038). Conclusions: Certain dermatologic diagnoses, most notably rashes, are more conducive to video visit management. Rashes made up 86% of new patient video visits, were more likely to have video visit follow-up if needed and were more likely to not require further follow-up indicating that the management of rashes from initial diagnosis to completion in care is suitable for video visit management. [ABSTRACT FROM AUTHOR]

Published
2024
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8. Comparing the quality of ChatGPT- and physician-generated responses to patients' dermatology questions in the electronic medical record.

Author

Reynolds, Kelly, Nadelman, Daniel, Durgin, Joseph, Ansah-Addo, Stephen, Cole, Daniel, Fayne, Rachel, Harrell, Jane, Ratycz, Madison, Runge, Mason, Shepard-Hayes, Amanda, Wenzel, Daniel, and Tejasvi, Trilokraj

Subjects
ELECTRONIC health records, GENERATIVE artificial intelligence, NATURAL language processing, PATIENT portals, CHATGPT
Abstract

Background ChatGPT is a free artificial intelligence (AI)-based natural language processing tool that generates complex responses to inputs from users. Objectives To determine whether ChatGPT is able to generate high-quality responses to patient-submitted questions in the patient portal. Methods Patient-submitted questions and the corresponding responses from their dermatology physician were extracted from the electronic medical record for analysis. The questions were input into ChatGPT (version 3.5) and the outputs extracted for analysis, with manual removal of verbiage pertaining to ChatGPT's inability to provide medical advice. Ten blinded reviewers (seven physicians and three nonphysicians) rated and selected their preference in terms of 'overall quality', 'readability', 'accuracy', 'thoroughness' and 'level of empathy' of the physician- and ChatGPT-generated responses. Results Thirty-one messages and responses were analysed. Physician-generated responses were vastly preferred over the ChatGPT -responses by the physician and nonphysician reviewers and received significantly higher ratings for 'readability' and 'level of empathy'. Conclusions The results of this study suggest that physician-generated responses to patients' portal messages are still preferred over ChatGPT, but generative AI tools may be helpful in generating the first drafts of responses and providing information on education resources for patients. [ABSTRACT FROM AUTHOR]

Published
2024
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14. Identification of 15 new psoriasis susceptibility loci highlights the role of innate immunity

Author

Tsoi, Lam C, Spain, Sarah L, Knight, Jo, Ellinghaus, Eva, Stuart, Philip E, Capon, Francesca, Ding, Jun, Li, Yanming, Tejasvi, Trilokraj, Gudjonsson, Johann E, Kang, Hyun M, Allen, Michael H, McManus, Ross, Novelli, Giuseppe, Samuelsson, Lena, Schalkwijk, Joost, Ståhle, Mona, Burden, A David, Smith, Catherine H, Cork, Michael J, Estivill, Xavier, Bowcock, Anne M, Krueger, Gerald G, Weger, Wolfgang, Worthington, Jane, Tazi-Ahnini, Rachid, Nestle, Frank O, Hayday, Adrian, Hoffmann, Per, Winkelmann, Juliane, Wijmenga, Cisca, Langford, Cordelia, Edkins, Sarah, Andrews, Robert, Blackburn, Hannah, Strange, Amy, Band, Gavin, Pearson, Richard D, Vukcevic, Damjan, Spencer, Chris CA, Deloukas, Panos, Mrowietz, Ulrich, Schreiber, Stefan, Weidinger, Stephan, Koks, Sulev, Kingo, Külli, Esko, Tonu, Metspalu, Andres, Lim, Henry W, Voorhees, John J, Weichenthal, Michael, Wichmann, H Erich, Chandran, Vinod, Rosen, Cheryl F, Rahman, Proton, Gladman, Dafna D, Griffiths, Christopher EM, Reis, Andre, Kere, Juha, Nair, Rajan P, Franke, Andre, Barker, Jonathan NWN, Abecasis, Goncalo R, Elder, James T, and Trembath, Richard C

Subjects
Genetics, Autoimmune Disease, Human Genome, Psoriasis, Aetiology, 2.1 Biological and endogenous factors, Skin, CARD Signaling Adaptor Proteins, Core Binding Factor Alpha 3 Subunit, DEAD Box Protein 58, DEAD-box RNA Helicases, GTPase-Activating Proteins, Genetic Loci, Genetic Predisposition to Disease, Genome-Wide Association Study, Guanylate Cyclase, Humans, Immunity, Innate, Membrane Proteins, Oligonucleotide Array Sequence Analysis, Polymorphism, Single Nucleotide, Receptors, Immunologic, STAT3 Transcription Factor, T-Lymphocytes, White People, Collaborative Association Study of Psoriasis, Genetic Analysis of Psoriasis Consortium, Psoriasis Association Genetics Extension, Wellcome Trust Case Control Consortium 2, Biological Sciences, Medical and Health Sciences, Developmental Biology
Abstract

To gain further insight into the genetic architecture of psoriasis, we conducted a meta-analysis of 3 genome-wide association studies (GWAS) and 2 independent data sets genotyped on the Immunochip, including 10,588 cases and 22,806 controls. We identified 15 new susceptibility loci, increasing to 36 the number associated with psoriasis in European individuals. We also identified, using conditional analyses, five independent signals within previously known loci. The newly identified loci shared with other autoimmune diseases include candidate genes with roles in regulating T-cell function (such as RUNX3, TAGAP and STAT3). Notably, they included candidate genes whose products are involved in innate host defense, including interferon-mediated antiviral responses (DDX58), macrophage activation (ZC3H12C) and nuclear factor (NF)-κB signaling (CARD14 and CARM1). These results portend a better understanding of shared and distinctive genetic determinants of immune-mediated inflammatory disorders and emphasize the importance of the skin in innate and acquired host defense.

Published
2012

15. Retrospective pharmacogenetic study of psoriasis highlights the role of KLK7 in TNF signaling

Author

Zhang, Haihan, primary, Patrick, Matthew T, additional, Tejasvi, Trilokraj, additional, Sarkar, Mrinal K, additional, Wasikowski, Rachael, additional, Stuart, Philip E, additional, Li, Qinmengge, additional, Xing, Xianying, additional, Voorhees, John J, additional, Ward, Nicole L, additional, He, Kevin, additional, Zhou, Xiang, additional, Gudjonsson, Johann E, additional, Nair, Rajan P, additional, Elder, James T, additional, and Tsoi, Lam C, additional

Published
2023
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17. Genome-wide scan reveals association of psoriasis with IL-23 and NF-κB pathways

Author

Nair, Rajan P, Duffin, Kristina Callis, Helms, Cynthia, Ding, Jun, Stuart, Philip E, Goldgar, David, Gudjonsson, Johann E, Li, Yun, Tejasvi, Trilokraj, Feng, Bing-Jian, Ruether, Andreas, Schreiber, Stefan, Weichenthal, Michael, Gladman, Dafna, Rahman, Proton, Schrodi, Steven J, Prahalad, Sampath, Guthery, Stephen L, Fischer, Judith, Liao, Wilson, Kwok, Pui-Yan, Menter, Alan, Lathrop, G Mark, Wise, Carol A, Begovich, Ann B, Voorhees, John J, Elder, James T, Krueger, Gerald G, Bowcock, Anne M, and Abecasis, Gonçalo R

Subjects
Genetics, Human Genome, Psoriasis, Autoimmune Disease, Aetiology, 2.1 Biological and endogenous factors, Skin, Adult, Case-Control Studies, DNA-Binding Proteins, Female, Gene Frequency, Genetic Predisposition to Disease, Genome-Wide Association Study, HLA-C Antigens, Humans, Interleukin-12 Subunit p40, Interleukin-13, Interleukin-23, Interleukin-4, Intracellular Signaling Peptides and Proteins, Male, Middle Aged, NF-kappa B, Nuclear Proteins, Polymorphism, Single Nucleotide, Signal Transduction, Tumor Necrosis Factor alpha-Induced Protein 3, Young Adult, Collaborative Association Study of Psoriasis, Biological Sciences, Medical and Health Sciences, Developmental Biology
Abstract

Psoriasis is a common immune-mediated disorder that affects the skin, nails and joints. To identify psoriasis susceptibility loci, we genotyped 438,670 SNPs in 1,409 psoriasis cases and 1,436 controls of European ancestry. We followed up 21 promising SNPs in 5,048 psoriasis cases and 5,041 controls. Our results provide strong support for the association of at least seven genetic loci and psoriasis (each with combined P < 5 x 10(-8)). Loci with confirmed association include HLA-C, three genes involved in IL-23 signaling (IL23A, IL23R, IL12B), two genes that act downstream of TNF-alpha and regulate NF-kappaB signaling (TNIP1, TNFAIP3) and two genes involved in the modulation of Th2 immune responses (IL4, IL13). Although the proteins encoded in these loci are known to interact biologically, we found no evidence for epistasis between associated SNPs. Our results expand the catalog of genetic loci implicated in psoriasis susceptibility and suggest priority targets for study in other auto-immune disorders.

Published
2009

18. Genome-wide scan reveals association of psoriasis with IL-23 and NF-kappaB pathways.

Author

Nair, Rajan P, Duffin, Kristina Callis, Helms, Cynthia, Ding, Jun, Stuart, Philip E, Goldgar, David, Gudjonsson, Johann E, Li, Yun, Tejasvi, Trilokraj, Feng, Bing-Jian, Ruether, Andreas, Schreiber, Stefan, Weichenthal, Michael, Gladman, Dafna, Rahman, Proton, Schrodi, Steven J, Prahalad, Sampath, Guthery, Stephen L, Fischer, Judith, Liao, Wilson, Kwok, Pui-Yan, Menter, Alan, Lathrop, G Mark, Wise, Carol A, Begovich, Ann B, Voorhees, John J, Elder, James T, Krueger, Gerald G, Bowcock, Anne M, Abecasis, Gonçalo R, and Collaborative Association Study of Psoriasis

Subjects
Collaborative Association Study of Psoriasis, Humans, Psoriasis, Genetic Predisposition to Disease, Intracellular Signaling Peptides and Proteins, DNA-Binding Proteins, NF-kappa B, Nuclear Proteins, Interleukin-4, Interleukin-13, HLA-C Antigens, Case-Control Studies, Signal Transduction, Gene Frequency, Polymorphism, Single Nucleotide, Adult, Middle Aged, Female, Male, Interleukin-23, Interleukin-12 Subunit p40, Genome-Wide Association Study, Young Adult, Tumor Necrosis Factor alpha-Induced Protein 3, Genetics, Autoimmune Disease, Clinical Research, 2.1 Biological and endogenous factors, Inflammatory and immune system, Skin, Developmental Biology, Biological Sciences, Medical and Health Sciences
Abstract

Psoriasis is a common immune-mediated disorder that affects the skin, nails and joints. To identify psoriasis susceptibility loci, we genotyped 438,670 SNPs in 1,409 psoriasis cases and 1,436 controls of European ancestry. We followed up 21 promising SNPs in 5,048 psoriasis cases and 5,041 controls. Our results provide strong support for the association of at least seven genetic loci and psoriasis (each with combined P < 5 x 10(-8)). Loci with confirmed association include HLA-C, three genes involved in IL-23 signaling (IL23A, IL23R, IL12B), two genes that act downstream of TNF-alpha and regulate NF-kappaB signaling (TNIP1, TNFAIP3) and two genes involved in the modulation of Th2 immune responses (IL4, IL13). Although the proteins encoded in these loci are known to interact biologically, we found no evidence for epistasis between associated SNPs. Our results expand the catalog of genetic loci implicated in psoriasis susceptibility and suggest priority targets for study in other auto-immune disorders.

Published
2009

19. CD30 expression in cutaneous B‐cell lymphomas

Author

Afolayan‐Oloye, Olabisi, primary, Zhao, Lili, additional, Tejasvi, Trilokraj, additional, Chan, May P., additional, Harms, Paul W., additional, Fullen, Douglas R., additional, Wilcox, Ryan A., additional, and Hristov, Alexandra C., additional

Published
2023
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23. Structural variation of centromeric endogenous retroviruses in human populations and their impact on cutaneous T-cell lymphoma, Sézary syndrome, and HIV infection

Author

Kaplan, Mark H., Kaminski, Mark, Estes, Judith M., Gitlin, Scott D., Zahn, Joseph, Elder, James T., Tejasvi, Trilokraj, Gensterblum, Elizabeth, Sawalha, Amr H., McGowan, Joseph Patrick, Dosik, Michael H., Direskeneli, Haner, Direskeneli, Guher Saruhan, Adebamowo, Sally N., Adebamowo, Clement A., Sajadi, Mohammad, and Contreras-Galindo, Rafael

Published
2019
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25. Exome-Guided Proteomic Analysis Identifies Early Biomarkers for the Progression from Psoriasis to Psoriatic Arthritis

Author

Feng, Bing-Jian, primary, Carroll, Courtney, additional, Tejasvi, Trilokraj, additional, Tsoi, Lam, additional, Nair, Rajan, additional, Goldgar, David, additional, Callis Duffin, Kristina, additional, Orbai, Ana-Maria, additional, Stuart, Philip, additional, Elder, James, additional, Walsh, Jessica A., additional, and Krueger, Gerald, additional

Published
2023
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27. Genome-wide Association Analysis of Psoriatic Arthritis and Cutaneous Psoriasis Reveals Differences in Their Genetic Architecture

Author

Stuart, Philip E., Nair, Rajan P., Tsoi, Lam C., Tejasvi, Trilokraj, Das, Sayantan, Kang, Hyun Min, Ellinghaus, Eva, Chandran, Vinod, Callis-Duffin, Kristina, Ike, Robert, Li, Yanming, Wen, Xiaoquan, Enerbäck, Charlotta, Gudjonsson, Johann E., Kõks, Sulev, Kingo, Külli, Esko, Tõnu, Mrowietz, Ulrich, Reis, Andre, Wichmann, H. Erich, Gieger, Christian, Hoffmann, Per, Nöthen, Markus M., Winkelmann, Juliane, Kunz, Manfred, Moreta, Elvia G., Mease, Philip J., Ritchlin, Christopher T., Bowcock, Anne M., Krueger, Gerald G., Lim, Henry W., Weidinger, Stephan, Weichenthal, Michael, Voorhees, John J., Rahman, Proton, Gregersen, Peter K., Franke, Andre, Gladman, Dafna D., Abecasis, Gonçalo R., and Elder, James T.

Published
2015
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31. Fine Mapping Major Histocompatibility Complex Associations in Psoriasis and Its Clinical Subtypes

Author

Okada, Yukinori, Han, Buhm, Tsoi, Lam C., Stuart, Philip E., Ellinghaus, Eva, Tejasvi, Trilokraj, Chandran, Vinod, Pellett, Fawnda, Pollock, Remy, Bowcock, Anne M., Krueger, Gerald G., Weichenthal, Michael, Voorhees, John J., Rahman, Proton, Gregersen, Peter K., Franke, Andre, Nair, Rajan P., Abecasis, Gonçalo R., Gladman, Dafna D., Elder, James T., de Bakker, Paul I.W., and Raychaudhuri, Soumya

Published
2014
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33. Genetic signature to provide robust risk assessment of psoriatic arthritis development in psoriasis patients

Author

Patrick, Matthew T., Stuart, Philip E., Raja, Kalpana, Gudjonsson, Johann E., Tejasvi, Trilokraj, Yang, Jingjing, Chandran, Vinod, Das, Sayantan, Callis-Duffin, Kristina, Ellinghaus, Eva, Enerbäck, Charlotta, Esko, Tõnu, Franke, Andre, Kang, Hyun M., Krueger, Gerald G., Lim, Henry W., Rahman, Proton, Rosen, Cheryl F., Weidinger, Stephan, Weichenthal, Michael, Wen, Xiaoquan, Voorhees, John J., Abecasis, Gonçalo R., Gladman, Dafna D., Nair, Rajan P., Elder, James T., and Tsoi, Lam C.

Published
2018
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38. Exome-wide association study reveals novel psoriasis susceptibility locus at TNFSF15 and rare protective alleles in genes contributing to type I IFN signalling

Author

Dand, Nick, Mucha, Sören, Tsoi, Lam C, Mahil, Satveer K, Stuart, Philip E, Arnold, Andreas, Baurecht, Hansjörg, Burden, A David, Callis Duffin, Kristina, Chandran, Vinod, Curtis, Charles J, Das, Sayantan, Ellinghaus, David, Ellinghaus, Eva, Enerback, Charlotta, Esko, Tõnu, Gladman, Dafna D, Griffiths, Christopher E M, Gudjonsson, Johann E, Hoffman, Per, Homuth, Georg, Hüffmeier, Ulrike, Krueger, Gerald G, Laudes, Matthias, Lee, Sang Hyuck, Lieb, Wolfgang, Lim, Henry W, Löhr, Sabine, Mrowietz, Ulrich, Müller-Nurayid, Martina, Nöthen, Markus, Peters, Annette, Rahman, Proton, Reis, André, Reynolds, Nick J, Rodriguez, Elke, Schmidt, Carsten O, Spain, Sarah L, Strauch, Konstantin, Tejasvi, Trilokraj, Voorhees, John J, Warren, Richard B, Weichenthal, Michael, Weidinger, Stephan, Zawistowski, Matthew, Nair, Rajan P, Capon, Francesca, Smith, Catherine H, Trembath, Richard C, Abecasis, Goncalo R, Elder, James T, Franke, Andre, Simpson, Michael A, and Barker, Jonathan N

Published
2017
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40. Combined Analysis of Genome-wide Association Studies for Crohn Disease and Psoriasis Identifies Seven Shared Susceptibility Loci

Author

Ellinghaus, David, Ellinghaus, Eva, Nair, Rajan P., Stuart, Philip E., Esko, Tõnu, Metspalu, Andres, Debrus, Sophie, Raelson, John V., Tejasvi, Trilokraj, Belouchi, Majid, West, Sarah L., Barker, Jonathan N., Kõks, Sulev, Kingo, Külli, Balschun, Tobias, Palmieri, Orazio, Annese, Vito, Gieger, Christian, Wichmann, H. Erich, Kabesch, Michael, Trembath, Richard C., Mathew, Christopher G., Abecasis, Gonçalo R., Weidinger, Stephan, Nikolaus, Susanna, Schreiber, Stefan, Elder, James T., Weichenthal, Michael, Nothnagel, Michael, and Franke, Andre

Published
2012
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41. Genome-Wide Meta-Analysis of Psoriatic Arthritis Identifies Susceptibility Locus at REL

Author

Ellinghaus, Eva, Stuart, Philip E., Ellinghaus, David, Nair, Rajan P., Debrus, Sophie, Raelson, John V., Belouchi, Majid, Tejasvi, Trilokraj, Li, Yanming, Tsoi, Lam C., Onken, Anna T., Esko, Tonu, Metspalu, Andres, Rahman, Proton, Gladman, Dafna D., Bowcock, Anne M., Helms, Cynthia, Krueger, Gerald G., Koks, Sulev, Kingo, Külli, Gieger, Christian, Erich Wichmann, H., Mrowietz, Ulrich, Weidinger, Stephan, Schreiber, Stefan, Abecasis, Gonçalo R., Elder, James T., Weichenthal, Michael, and Franke, Andre

Published
2012
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43. Meta-Analysis Confirms the LCE3C_LCE3B Deletion as a Risk Factor for Psoriasis in Several Ethnic Groups and Finds Interaction with HLA-Cw6

Author

Riveira-Munoz, Eva, He, Su-Min, Escaramís, Georgia, Stuart, Philip E., Hüffmeier, Ulrike, Lee, Catherine, Kirby, Brian, Oka, Akira, Giardina, Emiliano, Liao, Wilson, Bergboer, Judith, Kainu, Kati, de Cid, Rafael, Munkhbat, Batmunkh, Zeeuwen, Patrick L.J.M., Armour, John A.L., Poon, Annie, Mabuchi, Tomotaka, Ozawa, Akira, Zawirska, Agnieszka, Burden, A. David, Barker, Jonathan N., Capon, Francesca, Traupe, Heiko, Sun, Liang-Dan, Cui, Yong, Yin, Xian-Yong, Chen, Gang, Lim, Henry W., Nair, Rajan P., Voorhees, John J., Tejasvi, Trilokraj, Pujol, Ramón, Munkhtuvshin, Namid, Fischer, Judith, Kere, Juha, Schalkwijk, Joost, Bowcock, Anne, Kwok, Pui-Yan, Novelli, Giuseppe, Inoko, Hidetoshi, Ryan, Anthony W., Trembath, Richard C., Reis, André, Zhang, Xue-Jun, Elder, James T., and Estivill, Xavier

Published
2011
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45. Two cases of challenging cutaneous lymphoid infiltrates presenting in the context of COVID-19 vaccination: A reactive lymphomatoid papulosis-like eruption and a bona fide lymphoma.

Author

Bresler, Scott C., Menge, Tyler D., Tejasvi, Trilokraj, Carty, Shannon A., and Hristov, Alexandra C.

Subjects
COVID-19 vaccines, COVID-19, LYMPHOMAS, BREAKTHROUGH infections, LYMPHOPROLIFERATIVE disorders, B cells, MONOCLONAL antibodies
Abstract

COVID-19 infection and vaccination may be associated with a wide variety of cutaneous and immune manifestations. Here, we describe two patients who presented with monoclonal cutaneous T-cell infiltrates that showed cytologic and immunophenotypic features concerning for lymphoma shortly following COVID-19 vaccination. In one case, the eruption completely resolved. The second patient showed initial resolution, but her disease recurred and progressed following a breakthrough SARSCoV-2 infection. These cases suggest that immune stimulation following exposure to SARS-Cov-2 protein(s) in vaccine or infection may facilitate the development of a lymphoma or lymphoproliferative disorder in susceptible individuals. Moreover, they show that separating these cases from pseudolymphomatous reactive conditions is often challenging and requires close clinical correlation. [ABSTRACT FROM AUTHOR]

Published
2023
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