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4. Genetic landscape of ultra-stable chronic lymphocytic leukemia patients

5. Evidence for X(3872) in Pb-Pb Collisions and Studies of its Prompt Production at root s(NN)=5.02 TeV

11. Évaluation échographique de l’atteinte cardiaque dans le syndrome de Sneddon sans anticorps antiphospholipide et potentielle association avec la sévérité de l’atteinte neurologique : une étude de cohorte rétrospective de 61 patients

18. Refined karyotype-based prognostic stratification of chronic lymphocytic leukemia with a low-and very-low-risk genetic profile

19. Refined karyotype-based prognostic stratification of chronic lymphocytic leukemia with a low- and very-low-risk genetic profile

20. Chromosome aberrations detected by conventional karyotyping using novel mitogens in chronic lymphocytic leukemia: Clinical and biologic correlations

21. Aortic flow alterations in dilated and hypertrophic cardiomyopathy: New insight from quantitative flow MRI

23. Chlorambucil plus rituximab with or without maintenance rituximab as first-line treatment for elderly chronic lymphocytic leukemia patients

25. Stress tests sur le système bancaire et les organismes d’assurance en France

26. Domino Effects when Banks Hoard Liquidity: The French network

28. Behind the scenes of non-nodal MCL: downmodulation of genes involved in actin cytoskeleton organization, cell projection, cell adhesion, tumour invasion, TP53 pathway and mutated status of immunoglobulin heavy chain genes

29. TP53 mutations in adult acute lymphoblastic leukemia (ALL) are relatively frequent in molecularly negative case of both B- and T-lineage and correlate with poor response to induction therapy

32. PP.03.16

46. TP53 mutations are frequent in adult acute lymphoblastic leukemia cases negative for recurrent fusion genes and correlate with poor response to induction therapy

47. NOTCH1 mutations in +12 chronic lymphocytic leukemia (CLL) confer an unfavorable prognosis, induce a distinctive transcriptional profiling and refine the intermediate prognosis of +12 CLL

48. Somatically acquired JAK1 mutations in adult acute lymphoblastic leukemia

49. TP53 mutations are frequent in adult acute lymphoblastic leukemia cases negative for recurrent fusion genes and correlate with poor response to induction therapy

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