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2. Diagnosis and management of children and adult craniopharyngiomas: A French Endocrine Society/French Society for Paediatric Endocrinology & Diabetes Consensus Statement

Author

Cuny, Thomas, Reynaud, Rachel, Raverot, Gérald, Coutant, Régis, Chanson, Philippe, Kariyawasam, Dulanjalee, Poitou, Christine, Thomas-Teinturier, Cécile, Baussart, Bertrand, Samara-Boustani, Dinane, Feuvret, Loïc, Villanueva, Carine, Villa, Chiara, Bouillet, Benjamin, Tauber, Maïthé, Espiard, Stéphanie, Castets, Sarah, Beckers, Albert, Amsellem, Jessica, Vantyghem, Marie-Christine, Delemer, Brigitte, Chevalier, Nicolas, Brue, Thierry, André, Nicolas, Kerlan, Véronique, Graillon, Thomas, Raingeard, Isabelle, Alapetite, Claire, Raverot, Véronique, Salenave, Sylvie, Boulin, Anne, Appay, Romain, Dalmas, Florian, Fodil, Sarah, Coppin, Lucie, Buffet, Camille, Thuillier, Philippe, Castinetti, Frédéric, Vogin, Guillaume, Cazabat, Laure, Kuhn, Emmanuelle, Haissaguerre, Magalie, Reznik, Yves, Goichot, Bernard, Bachelot, Anne, Kamenicky, Peter, Decoudier, Bénédicte, Planchon, Charlotte, Micoulaud-Franchi, Jean-Arthur, Romanet, Pauline, Jacobi, David, Faucher, Pauline, Carette, Claire, Bihan, Hélène, Drui, Delphine, Rossignol, Sylvie, Gonin, Lucile, Sokol, Emmanuelle, Wiard, Laurent, Courtillot, Carine, Nicolino, Marc, Grunenwald, Solange, Chabre, Olivier, Christin-Maître, Sophie, Desailloud, Rachel, Maiter, Dominique, Guignat, Laurence, Brac de la Perrière, Aude, Salva, Philippe, Scavarda, Didier, Bonneville, Fabrice, Caron, Philippe, Vasiljevic, Alexandre, Leclerq, Delphine, Cortet, Christine, Gaillard, Stephan, Albarel, Frédérique, Clément, Karine, Jouanneau, Emmanuel, Dufour, Henry, Barat, Pascal, and Gatta-Cherifi, Blandine

Published
2025
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4. Imprinting disorders

Author

Eggermann, Thomas, Monk, David, de Nanclares, Guiomar Perez, Kagami, Masayo, Giabicani, Eloïse, Riccio, Andrea, Tümer, Zeynep, Kalish, Jennifer M., Tauber, Maithé, Duis, Jessica, Weksberg, Rosanna, Maher, Eamonn R., Begemann, Matthias, and Elbracht, Miriam

Published
2023
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5. First step towards a consensus strategy for multi-locus diagnostic testing of imprinting disorders

Author

Mackay, Deborah, Bliek, Jet, Kagami, Masayo, Tenorio-Castano, Jair, Pereda, Arrate, Brioude, Frédéric, Netchine, Irène, Papingi, Dzhoy, de Franco, Elisa, Lever, Margaret, Sillibourne, Julie, Lombardi, Paola, Gaston, Véronique, Tauber, Maithé, Diene, Gwenaelle, Bieth, Eric, Fernandez, Luis, Nevado, Julian, Tümer, Zeynep, Riccio, Andrea, Maher, Eamonn R., Beygo, Jasmin, Tannorella, Pierpaola, Russo, Silvia, de Nanclares, Guiomar Perez, Temple, I. Karen, Ogata, Tsutomu, Lapunzina, Pablo, and Eggermann, Thomas

Published
2022
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6. Hypothalamic syndrome

Author

Müller, Hermann L., Tauber, Maithé, Lawson, Elizabeth A., Özyurt, Jale, Bison, Brigitte, Martinez-Barbera, Juan-Pedro, Puget, Stephanie, Merchant, Thomas E., and van Santen, Hanneke M.

Published
2022
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10. Turner syndrome: French National Diagnosis and Care Protocol (NDCP; National Diagnosis and Care Protocol)

Author

Fiot, Elodie, Alauze, Bertille, Donadille, Bruno, Samara-Boustani, Dinane, Houang, Muriel, De Filippo, Gianpaolo, Bachelot, Anne, Delcour, Clemence, Beyler, Constance, Bois, Emilie, Bourrat, Emmanuelle, Bui Quoc, Emmanuel, Bourcigaux, Nathalie, Chaussain, Catherine, Cohen, Ariel, Cohen-Solal, Martine, Da Costa, Sabrina, Dossier, Claire, Ederhy, Stephane, Elmaleh, Monique, Iserin, Laurence, Lengliné, Hélène, Poujol-Robert, Armelle, Roulot, Dominique, Viala, Jerome, Albarel, Frederique, Bismuth, Elise, Bernard, Valérie, Bouvattier, Claire, Brac, Aude, Bretones, Patricia, Chabbert-Buffet, Nathalie, Chanson, Philippe, Coutant, Regis, de Warren, Marguerite, Demaret, Béatrice, Duranteau, Lise, Eustache, Florence, Gautheret, Lydie, Gelwane, Georges, Gourbesville, Claire, Grynberg, Mickaël, Gueniche, Karinne, Jorgensen, Carina, Kerlan, Veronique, Lebrun, Charlotte, Lefevre, Christine, Lorenzini, Françoise, Manouvrier, Sylvie, Pienkowski, Catherine, Reynaud, Rachel, Reznik, Yves, Siffroi, Jean-Pierre, Tabet, Anne-Claude, Tauber, Maithé, Vautier, Vanessa, Tauveron, Igor, Wambre, Sebastien, Zenaty, Delphine, Netchine, Irène, Polak, Michel, Touraine, Philippe, Carel, Jean-Claude, Christin-Maitre, Sophie, and Léger, Juliane

Published
2022
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17. Differential DNA methylation in iPSC-derived dopaminergic neurons: a step forward on the role of SNORD116microdeletion in the pathophysiology of addictive behavior in Prader-Willi syndrome

Author

Salles, Juliette, Eddiry, Sanaa, Amri, Saber, Galindo, Mélissa, Lacassagne, Emmanuelle, George, Simon, Mialhe, Xavier, Lhuillier, Émeline, Franchitto, Nicolas, Jeanneteau, Freddy, Gennero, Isabelle, Salles, Jean-Pierre, and Tauber, Maithé

Abstract

Introduction: A microdeletion including the SNORD116gene (SNORD116MD) has been shown to drive the Prader-Willi syndrome (PWS) features. PWS is a neurodevelopmental disorder clinically characterized by endocrine impairment, intellectual disability and psychiatric symptoms such as a lack of emotional regulation, impulsivity, and intense temper tantrums with outbursts. In addition, this syndrome is associated with a nutritional trajectory characterized by addiction-like behavior around food in adulthood. PWS is related to the genetic loss of expression of a minimal region that plays a potential role in epigenetic regulation. Nevertheless, the role of the SNORD116MD in DNA methylation, as well as the impact of the oxytocin (OXT) on it, have never been investigated in human neurons. Methods: We studied the methylation marks in induced pluripotent stem-derived dopaminergic neurons carrying a SNORD116MD in comparison with those from an age-matched adult healthy control. We also performed identical neuron differentiation in the presence of OXT. We performed a genome-wide DNA methylation analysis from the iPSC-derived dopaminergic neurons by reduced-representation bisulfite sequencing. In addition, we performed RNA sequencing analysis in these iPSC-derived dopaminergic neurons differentiated with or without OXT. Results: The analysis revealed that 153,826 cytosines were differentially methylated between SNORD116MD neurons and control neurons. Among the differentially methylated genes, we determined a list of genes also differentially expressed. Enrichment analysis of this list encompassed the dopaminergic system with COMTand SLC6A3. COMTdisplayed hypermethylation and under-expression in SNORD116MD, and SLC6A3displayed hypomethylation and over-expression in SNORD116MD. RT-qPCR confirmed significant over-expression of SLC6A3in SNORD116 MDneurons. Moreover, the expression of this gene was significantly decreased in the case of OXT adjunction during the differentiation. Conclusion: SNORD116MD dopaminergic neurons displayed differential methylation and expression in the COMTand SLC6A3genes, which are related to dopaminergic clearance.

Published
2024
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21. Copy number variation analysis in adults with catatonia confirms haploinsufficiency of SHANK3 as a predisposing factor

Author

Breckpot, Jeroen, Vercruyssen, Marieke, Weyts, Eddy, Vandevoort, Sean, D'Haenens, Greet, Van Buggenhout, Griet, Leempoels, Lore, Brischoux-Boucher, Elise, Van Maldergem, Lionel, Renieri, Alessandra, Mencarelli, Maria Antonietta, D'Angelo, Carla, Mericq, Veronica, Hoffer, Mariette J., Tauber, Maithé, Molinas, Catherine, Castiglioni, Claudia, Brison, Nathalie, Vermeesch, Joris R., Danckaerts, Marina, Sienaert, Pascal, Devriendt, Koenraad, and Vogels, Annick

Published
2016
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29. Obesity, Overweight, and Pituitary Stalk Interruption Syndrome in Children and Young Adults

Author

Nannette, Gaëlle, primary, Bar, Céline, additional, Diene, Gwenaëlle, additional, Pienkowski, Catherine, additional, Oliver-Petit, Isabelle, additional, Jouret, Béatrice, additional, Cartault, Audrey, additional, Porquet-Bordes, Valérie, additional, Salles, Jean-Pierre, additional, Grunenwald, Solange, additional, Edouard, Thomas, additional, Molinas, Catherine, additional, and Tauber, Maithé, additional

Published
2022
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30. LEOPARD syndrome-associated SHP2 mutation confers leanness and protection from diet-induced obesity

Author

Tajan, Mylène, Batut, Aurélie, Cadoudal, Thomas, Deleruyelle, Simon, Le Gonidec, Sophie, Laurent, Céline Saint, Vomscheid, Maëlle, Wanecq, Estelle, Tréguer, Karine, De Rocca Serra-Nédélec, Audrey, Vinel, Claire, Marques, Marie-Adeline, Pozzo, Joffrey, Kunduzova, Oksana, Salles, Jean-Pierre, Tauber, Maithé, Raynal, Patrick, Cavé, Hélène, Edouard, Thomas, Valet, Philippe, and Yart, Armelle

Published
2014
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32. Amygdala hyperactivation relates to eating behavior: a potential indicator of food addiction in Prader-Willi syndrome

Author

Strelnikov, Kuzma, Debladis, Jimmy, Salles, Juliette, Valette, Marion, Cortadellas, Julie, Tauber, Maithé, Barone, Pascal, Centre Hospitalier Universitaire de Toulouse (CHU Toulouse), Centre de recherche cerveau et cognition (CERCO UMR5549), Université Toulouse III - Paul Sabatier (UT3), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Centre National de la Recherche Scientifique (CNRS)-Toulouse Mind & Brain Institut (TMBI), Université Toulouse - Jean Jaurès (UT2J)-Université Toulouse III - Paul Sabatier (UT3), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Université Toulouse - Jean Jaurès (UT2J)-Université Toulouse III - Paul Sabatier (UT3), Université Fédérale Toulouse Midi-Pyrénées, Institut Toulousain des Maladies Infectieuses et Inflammatoires (Infinity), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), Centre de Référence du Syndrome de Prader-Willi, Pôle Enfants [CHU Toulouse], Centre Hospitalier Universitaire de Toulouse (CHU Toulouse)-Centre Hospitalier Universitaire de Toulouse (CHU Toulouse), and BARONE, Pascal

Subjects
[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiology, [SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiology, addiction, amygdala, Prader-Willi syndrome, eating behavior
Abstract

Prader-Willi syndrome (PWS) is a rare genetic neurodevelopmental disorder involving nutritional, endocrine /metabolic, emotional and behavior dimensions. There is evidence for impaired hypothalamic development and function in PWS, involving oxytocin and ghrelin, which can account for the typical PWS phenotype. Hyperphagia with addiction-like behavior is one of the common features of PWS and is a consequence of the hypothalamic dysfunction. In this study, we hypothesized that brain regions associated with compulsive eating behavior would be abnormally activated by food-related odors in PWS, as these can stimulate the appetite and induce hunger-related behavior.MethodsWe used a classic olfactory discrimination test to verify that olfaction was normal in patients with PWS. In an fMRI scanner, we presented two odors, a tulip and a caramel odor, which have a different hedonic valence and a different capacity to arouse hunger-related behavior.ResultsThere was a five-fold higher activation in the right amygdala for the caramel odor compared with the tulip odor in patients with PWS (n=14). No such hyperactivation was found in age-matched controls (n=11). Cluster analysis of clinical hyperphagia scores in patients with PWS revealed a link with the right amygdala hyperactivation.ConclusionsOur study provides evidence for functional alteration of the right amygdala in PWS, which is part of the brain reward network involved in food addiction. This finding may relate to dysfunction of the ghrelin and oxytocin systems in PWS, as these are involved in addictive behavior, appetite, and olfactory bulb regulation.

Published
2022
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34. Hypothalamic syndrome

Author

Endocrinologie patientenzorg, Brain, Child Health, Cancer, Müller, Hermann L., Tauber, Maithé, Lawson, Elizabeth A., Özyurt, Jale, Bison, Brigitte, Martinez-Barbera, Juan Pedro, Puget, Stephanie, Merchant, Thomas E., van Santen, Hanneke M., Endocrinologie patientenzorg, Brain, Child Health, Cancer, Müller, Hermann L., Tauber, Maithé, Lawson, Elizabeth A., Özyurt, Jale, Bison, Brigitte, Martinez-Barbera, Juan Pedro, Puget, Stephanie, Merchant, Thomas E., and van Santen, Hanneke M.

Published
2022

36. Yearly Height Gain Is Dependent on the Truly Received Dose of Growth Hormone and the Duration of Periods of Poor Adherence: Practical Lessons From the French Easypod (TM) Connect Multicenter Observational Study

Author

Coutant, Regis, Nicolino, Marc, Cammas, Benoit, de Buyst, Valerie, Tauber, Maithé, Hamel, Jean-François, HAL UR1, Admin, Centre Hospitalier Universitaire d'Angers (CHU Angers), PRES Université Nantes Angers Le Mans (UNAM), Hospices Civils de Lyon (HCL), Hôpital Femme Mère Enfant [CHU - HCL] (HFME), Merck [Darmstadt], Institut Toulousain des Maladies Infectieuses et Inflammatoires (Infinity), Université Toulouse III - Paul Sabatier (UT3), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), Centre Hospitalier Universitaire de Toulouse (CHU Toulouse), Institut de recherche en santé, environnement et travail (Irset), Université d'Angers (UA)-Université de Rennes (UR)-École des Hautes Études en Santé Publique [EHESP] (EHESP)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Structure Fédérative de Recherche en Biologie et Santé de Rennes ( Biosit : Biologie - Santé - Innovation Technologique ), École des Hautes Études en Santé Publique [EHESP] (EHESP), Merck Serono SAS, Lyon, France, Merck KGaA, and Université de Toulouse (UT)-Université de Toulouse (UT)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS)

Subjects
Male, growth hormone deficiency, Adolescent, Hormone Replacement Therapy, Endocrinology, Diabetes and Metabolism, [SDV]Life Sciences [q-bio], Diseases of the endocrine glands. Clinical endocrinology, Medication Adherence, electronic device, small for gestational age, Endocrinology, children, Humans, adherence, Child, Growth Disorders, recombinant human growth hormone, Retrospective Studies, Original Research, height gain, Human Growth Hormone, RC648-665, Body Height, [SDV] Life Sciences [q-bio], Treatment Outcome, Growth Hormone, Female
Abstract

ObjectiveTo study the impact of the true mean daily dose and the true mean number of injections per week on the yearly height gain in short children treated with recombinant human growth hormone (rhGH).Design and Methods220 children from the French Easypod™ Connect Observational Study (ECOS) used the Easypod™ electronic device to record rhGH injections. The mean daily rhGH dose (the sum of the doses truly received divided by the number of days) and mean number of injections per week (the number of injections truly performed divided by the number of weeks) were calculated. Linear mixed models were used to study the impact of short (3-month) and long (1-year) variations in rhGH administration on the yearly height change [as a standard deviation score (SDS)], with time on treatment as a covariate. For each patient, several periods of 3 or 12 months were considered and designated as poorly adherence or fully adherence. We studied the impact of each of period on the height change.ResultsAt treatment initiation, the mean ± SD age was 9.8 ± 3.7 years (females: 47%, prepubertal: 86%) and the mean height was -2.28 ± 0.92 SDS. The mean treatment duration was 3.2 ± 1.1 years (685.2 patient years). 122 patients were GH-deficient, 79 were small for gestational age, and 19 had Turner syndrome. When treatment was computed over 12-month periods, receiving a mean daily dose 5 injections/week), whereas maintaining a mean daily dose >0.03 mg/kg.d with5 injections/week). For 3-month periods, changes in the daily rhGH dose or the number of injections per week over such short period did not influence the yearly height gain SDS.ConclusionThe 12-month treatment model showed that when poor adherence leads to a low true daily GH dose, the yearly height gain is low. The 3-month treatment model showed that poor adherence for short periods (

Published
2022
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40. Obesity, Overweight, and Pituitary Stalk Interruption Syndrome in Children and Young Adults.

Author

Nannette, Gaëlle, Bar, Céline, Diene, Gwenaëlle, Pienkowski, Catherine, Oliver-Petit, Isabelle, Jouret, Béatrice, Cartault, Audrey, Porquet-Bordes, Valérie, Salles, Jean-Pierre, Grunenwald, Solange, Edouard, Thomas, Molinas, Catherine, and Tauber, Maithé

Subjects
OBESITY, YOUNG adults, PHENOTYPES
Abstract

Context: Pituitary stalk interruption syndrome (PSIS) is rare in the pediatric population. It combines ectopic posterior pituitary stalk interruption and anterior pituitary hypoplasia with hormonal deficiencies. The phenotype is highly heterogeneous and obesity/overweight seems to be underreported in the literature. Objective: To identify patients with PSIS and obesity or overweight, describe their phenotype, and compare them with patients with PSIS without overweight/obesity. Methods: Sixty-nine children and young adults with PSIS in a Toulouse cohort from 1984 to 2019 were studied. We identified 25 obese or overweight patients (OB-OW group), and 44 were nonobese/overweight (NO group). Then the groups were compared. Results: All cases were sporadic. The sex ratio was 1.6. The main reason for consultation in both groups was growth retardation (61% in OB-OW group, 77% in NO group). History of neonatal hypoglycemia was more common in the OB-OW than in the NO group (57% vs 14%, P=.0008), along with extrapituitary malformations (64% vs 20%, P<0001). The incidence of caesarean section was higher in the OB-OW group (52%) than in the NO group (23%), although not significant (P= .07). Conclusion: Patients with PSIS who are obese/overweight display interesting phenotypic differences that suggest hypothalamic defects. Studies are needed that include additional information on hormonal levels, particularly regarding oxytocin and ghrelin. [ABSTRACT FROM AUTHOR]

Published
2023
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43. Hyponatremia in Children and Adults with Prader–Willi Syndrome: A Survey Involving Seven Countries

Author

Coupaye, Muriel, primary, Pellikaan, Karlijn, additional, Goldstone, Anthony P., additional, Crinò, Antonino, additional, Grugni, Graziano, additional, Markovic, Tania P., additional, Høybye, Charlotte, additional, Caixàs, Assumpta, additional, Mosbah, Helena, additional, De Graaff, Laura C. G., additional, Tauber, Maithé, additional, and Poitou, Christine, additional

Published
2021
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44. Growth restriction and genomic imprinting-overlapping phenotypes support the concept of an imprinting network

Author

Eggermann, Thomas, Davies, Justin H., Tauber, Maithé, van den Akker, Erica, Hokken-Koelega, Anita, Johansson, Gudmundur, Netchine, Irène, Eggermann, Thomas, Davies, Justin H., Tauber, Maithé, van den Akker, Erica, Hokken-Koelega, Anita, Johansson, Gudmundur, and Netchine, Irène

Abstract

Intrauterine and postnatal growth disturbances are major clinical features of imprinting disorders, a molecularly defined group of congenital syndromes caused by molecular alterations affecting parentally imprinted genes. These genes are expressed monoallelically and in a parent-of-origin manner, and they have an impact on human growth and development. In fact, several genes with an exclusive expression from the paternal allele have been shown to promote foetal growth, whereas maternally expressed genes suppress it. The evolution of this correlation might be explained by the different interests of the maternal and paternal genomes, aiming for the conservation of maternal resources for multiple offspring versus extracting maximal maternal resources. Since not all imprinted genes in higher mammals show the same imprinting pattern in different species, the findings from animal models are not always transferable to human. Therefore, human imprinting disorders might serve as models to understand the complex regulation and interaction of imprinted loci. This knowledge is a prerequisite for the development of precise diagnostic tools and therapeutic strategies for patients affected by imprinting disorders. In this review we will specifically overview the current knowledge on imprinting disorders associated with growth retardation, and its increasing relevance in a personalised medicine direction and the need for a multidisciplinary therapeutic approach.

Published
2021

45. Hyponatremia in children and adults with Prader–Willi Syndrome:A survey involving seven countries

Author

Coupaye, Muriel, Pellikaan, Karlijn, Goldstone, Anthony P., Crinò, Antonino, Grugni, Graziano, Markovic, Tania P., Høybye, Charlotte, Caixàs, Assumpta, Mosbah, Helena, De Graaff, Laura C.G., Tauber, Maithé, Poitou, Christine, Coupaye, Muriel, Pellikaan, Karlijn, Goldstone, Anthony P., Crinò, Antonino, Grugni, Graziano, Markovic, Tania P., Høybye, Charlotte, Caixàs, Assumpta, Mosbah, Helena, De Graaff, Laura C.G., Tauber, Maithé, and Poitou, Christine

Abstract

In Prader–Willi syndrome (PWS), conditions that are associated with hyponatremia are common, such as excessive fluid intake (EFI), desmopressin use and syndrome of inappropriate antidiuretic hormone (SIADH) caused by psychotropic medication. However, the prevalence of hyponatremia in PWS has rarely been reported. Our aim was to describe the prevalence and severity of hyponatremia in PWS. In October 2020, we performed a retrospective study based on the medical records of a large cohort of children and adults with PWS from seven countries. Among 1326 patients (68% adults), 34 (2.6%) had at least one episode of mild or moderate hyponatremia (125 ≤ Na < 135 mmol/L). The causes of non-severe hyponatremia were often multi-factorial, including psychotropic medication in 32%, EFI in 24% and hyperglycemia in 12%. No obvious cause was found in 29%. Seven (0.5%) adults experienced severe hyponatremia (Na < 125 mmol/L). Among these, five recovered completely, but two died. The causes of severe hyponatremia were desmopressin treatment for nocturnal enuresis (n = 2), EFI (n = 2), adrenal insufficiency (n = 1), diuretic treatment (n = 1) and unknown (n = 1). In conclusion, severe hyponatremia was very rare but potentially fatal in PWS. Desmopressin treatment for nocturnal enuresis should be avoided. Enquiring about EFI and monitoring serum sodium should be included in the routine follow-ups of patients with PWS.

Published
2021

47. New candidate loci identified by array-CGH in a cohort of 100 children presenting with syndromic obesity

Author

Vuillaume, Marie-Laure, Naudion, Sophie, Banneau, Guillaume, Diene, Gwenaelle, Cartault, Audrey, Cailley, Dorothée, Bouron, Julie, Toutain, Jérôme, Bourrouillou, Georges, Vigouroux, Adeline, Bouneau, Laurence, Nacka, Fabienne, Kieffer, Isabelle, Arveiler, Benoit, Knoll-Gellida, Anja, Babin, Patrick J., Bieth, Eric, Jouret, Béatrice, Julia, Sophie, Sarda, Pierre, Geneviève, David, Faivre, Laurence, Lacombe, Didier, Barat, Pascal, Tauber, Maithé, Delrue, Marie-Ange, and Rooryck, Caroline

Published
2014
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49. SHP2 drives inflammation-triggered insulin resistance by reshaping tissue macrophage populations

Author

Paccoud, Romain, primary, Saint-Laurent, Céline, additional, Piccolo, Enzo, additional, Tajan, Mylène, additional, Dortignac, Alizée, additional, Pereira, Ophélie, additional, Le Gonidec, Sophie, additional, Baba, Inès, additional, Gélineau, Adélaïde, additional, Askia, Haoussa, additional, Branchereau, Maxime, additional, Charpentier, Julie, additional, Personnaz, Jean, additional, Branka, Sophie, additional, Auriau, Johanna, additional, Deleruyelle, Simon, additional, Canouil, Mickaël, additional, Beton, Nicolas, additional, Salles, Jean-Pierre, additional, Tauber, Maithé, additional, Weill, Jacques, additional, Froguel, Philippe, additional, Neel, Benjamin G., additional, Araki, Toshiyuki, additional, Heymes, Christophe, additional, Burcelin, Rémy, additional, Castan, Isabelle, additional, Valet, Philippe, additional, Dray, Cédric, additional, Gautier, Emmanuel L., additional, Edouard, Thomas, additional, Pradère, Jean-Philippe, additional, and Yart, Armelle, additional

Published
2021
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