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1. The Partial Support of the Left Ventricular Assist Device Shifts the Systemic Cardiac Output Curve Upward in Proportion to the Effective Left Ventricular Ejection Fraction in Pressure-Volume Loop

2. Persistent high fever and systemic inflammation induced by percutaneous coronary intervention-related periaortitis

3. Total Mechanical Unloading Minimizes Metabolic Demand of Left Ventricle and Dramatically Reduces Infarct Size in Myocardial Infarction.

5. Overexpression of TFAM or twinkle increases mtDNA copy number and facilitates cardioprotection associated with limited mitochondrial oxidative stress.

6. Prediction of hemodynamics under left ventricular assist device

7. Intravenous electrical vagal nerve stimulation prior to coronary reperfusion in a canine ischemia-reperfusion model markedly reduces infarct size and prevents subsequent heart failure

8. Significance of troponin I level as a marker of disease activity in the management of acute necrotizing eosinophilic myocarditis with normal peripheral eosinophil count: a case report

9. An Arrhythmogenic Large Coronary Artery Aneurysm

10. Left Ventricular Mechanical Unloading by Total Support of Impella in Myocardial Infarction Reduces Infarct Size, Preserves Left Ventricular Function, and Prevents Subsequent Heart Failure in Dogs

11. Abstract 13813: Intravascular Vagal Nerve Stimulation in Acute Myocardial Infarction Markedly Reduced Infarct Size and Improved Cardiac Function in the Long Term

12. Abstract 14058: Transvascular Total Left Ventricular Unloading for Acute Myocardial Infarction Strikingly Reduced Myocardial Oxygen Consumption, Limited the Infarct Size and Prevented Heart Failure in the long term

13. The Akt-mTOR axis is a pivotal regulator of eccentric hypertrophy during volume overload

14. Chronic Heart Failure Blunts the Low Pressure Baroreflex Induced Biphasic Change of Sympathetic Nerve Activity

15. Neural Unloading using Intravenous Vagal Nerve Stimulation for Acute Myocardial Infarction (AMI) Improves Cardiac Function and Prevents Heart Failure in the Long Term

16. Abstract 17082: Carotid Body Denervation Improves Autonomic Balance and Prevents the Worsening of Heart Failure in Rats After Large Myocardial Infarction

17. Changes in vascular properties, not ventricular properties, predominantly contribute to baroreflex regulation of arterial pressure

18. Afferent vagal nerve stimulation resets baroreflex neural arc and inhibits sympathetic nerve activity

19. Prediction of the impact of venoarterial extracorporeal membrane oxygenation on hemodynamics

20. Vagally mediated low pressure baroreflex evokes biphasic changes in sympathetic nerve activity and resets the arterial baroreflex (1169.12)

21. Carotid body denervation prevents pulmonary edema, attenuates left ventricular remodeling and restores normal autonomic balance in chronic heart failure after myocardial infarction (709.5)

22. Transvenous vagal nerve stimulation (VNS) in acute myocardial infarction (AMI) markedly reduces the infarction size and improves chronic cardiac function (LB670)

23. Combination therapy of vagal nerve stimulation and left ventricular assist device maximally reduces the infarct size in ischemia reperfusion model (1080.4)

24. The Neuro-Mechanical Unloading for Acute Myocardial Infarction Markedly Reduces the Infarct Size and Prevents Heart Failure in the Long Term

25. Risk factors for coronary artery calcification in Japanese patients

26. Afferent Vagal Nerve Stimulation Resets the Baroreflex Neural Arc and Inhibits Sympathetic Nerve Activity

27. The Increase of Mitochondrial DNA Copy Number Attenuates Eccentric Cardiac Remodeling In Volume Overload Model

28. Quantitative Prediction of Impact of Left Ventricular Assist Device (LVAD) on Hemodynamics

29. Tolvaptan Accelerates Diuresis without Contraction of Stressed Blood Volume

30. Transvascular Left Ventricular Unloading in the Acute Phase of Myocardial Infarction Markedly Reduces Infarct Size and Prevents Future Heart Failure

31. Renal afferent nerve stimulation induces baroreflex resetting through the activation of sympathorenal axis without compromising arterial pressure buffering function

32. INTRAVENOUS VAGAL NERVE STIMULATION IN ACUTE MYOCARDIAL INFARCTION (AMI) MARKEDLY IMPROVES CARDIAC FUNCTION AND PREVENTS CHRONIC HEART FAILURE

34. Intravenous Vagal Nerve Stimulation in Acute Myocardial Infarction (AMI) Strikingly Reduces Infarction Size and Improves Chronic Cardiac Failure

36. Concomitant Vagal Nerve Stimulation (VNS) with Totally Left Ventricular Unloading Markedly Reduces the Infarct Size in Ischemia Reperfusion (IR) Model

37. Prediction of hemodynamics under left ventricular assist device.

38. Low Pressure Baroreflex Evokes Biphasic Change in Sympathetic Nerve Activity through Vagal Nerve

41. Afferent vagal nerve stimulation induced sympathoinhibition may in part attribute to the beneficial impact of vagal nerve stimulation on heart failure

42. Total unloading of the Left Ventricle by circulatory Assist Device (LVAD) strikingly reduces the infract size in ischemia-reperfusion injury

43. Prediction of hemodynamic impact of the venoarterial Extracorporeal Membrane Oxygenation (ECMO)

44. Quantitative Prediction of the Hemodynamic Impact of Left Ventricular (LV) Assist Device (LVAD)

47. Persistent high fever and systemic inflammation induced by percutaneous coronary intervention-related periaortitis

48. Prediction of the impact of venoarterial extracorporeal membrane oxygenation on hemodynamics.

49. Changes in vascular properties, not ventricular properties, predominantly contribute to baroreflex regulation of arterial pressure.

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