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1. Can the clinical sign “head-turning sign” and simple questions in “Neucop-Q” predict amyloid β pathology?

2. Alterations of striatal phosphodiesterase 10 A and their association with recurrence rate in bipolar I disorder

3. A novel plasma p-tau181 assay as a specific biomarker of tau pathology in Alzheimer's disease

6. Association of protein distribution and gene expression revealed by positron emission tomography and postmortem gene expression in the dopaminergic system of the human brain

7. Imaging α-synuclein pathologies in animal models and patients with Parkinson’s and related diseases

9. Investigating neural dysfunction with abnormal protein deposition in Alzheimer’s disease through magnetic resonance spectroscopic imaging, plasma biomarkers, and positron emission tomography

12. Performance of plasma Aβ42/40, measured using a fully automated immunoassay, across a broad patient population in identifying amyloid status

13. Increased glutamate and glutamine levels and their relationship to astrocytes and dopaminergic transmissions in the brains of adults with autism

14. Bayesian Semiparametric Modeling of Response Mechanism for Nonignorable Missing Data

15. Semiparametric estimation of heterogeneous treatment effects under the nonignorable assignment condition

16. Two pathways differentially linking tau depositions, oxidative stress, and neuronal loss to apathetic phenotypes in progressive supranuclear palsy

18. Identification and Estimation of Heterogeneous Treatment Effects under Non-compliance or Non-ignorable assignment

19. An optimized reference tissue method for quantification of tau protein depositions in diverse neurodegenerative disorders by PET with 18F-PM-PBB3 (18F-APN-1607)

21. PET-based classification of corticobasal syndrome

24. In Vivo Assessment of Astrocyte Reactivity in Patients with Progressive Supranuclear Palsy

26. Contributors

30. High-Contrast In Vivo Imaging of Tau Pathologies in Alzheimer’s and Non-Alzheimer’s Disease Tauopathies

32. In vivo PET classification of tau pathologies in patients with frontotemporal dementia

35. Development and Comparison of a Novel Mid‐Region Directed p‐Tau 181 Assay with Tau Positron Emission Tomography in Alzheimer’s Disease

36. In vivo characterization of tau accumulation in patients with frontotemporal dementia: A PET study with 18F‐florzolotau

37. The aftermath of boxing: Longitudinal changes of cerebral retention patterns of 18F‐florzolotau (18F‐APN‐1607) in the former professional boxers

38. Involvement of differential tau pathologies in late‐onset bipolar disorder assessed by PET with 18F‐florzolotau

39. In vivoassessment of astrocyte reactivity in patients with progressive supranuclear palsy

40. Development and Performance Assessment of a Novel Plasma p-Tau181 Assay Reflecting Tau Tangle Pathology in Alzheimer's Disease

42. Altered Brain Energy Metabolism Related to Astrocytes in Alzheimer's Disease.

43. Correlation of 18F-PM-PBB3 (18F-florzolotau) Tau PET Imaging with Postmortem Neuropathological Findings in A Case with Progressive Supranuclear Palsy (P9-11.015)

44. Investigating Neural Dysfunction with Abnormal Protein Deposition in Alzheimer’s Disease Through Magnetic Resonance Spectroscopic Imaging, Plasma Biomarkers, and Positron Emission Tomography

45. Altered brain energy metabolism related to astrocytes in Alzheimer′s disease

48. Estimating Density Ratio of Marginals to Joint: Applications to Causal Inference

50. Pharmacokinetic and pharmacodynamic assessment of histamine H3 receptor occupancy by enerisant: a human PET study with a novel H3 binding ligand, [11C]TASP457

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