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574 results on '"Tait B"'

1. Applied Phon Curve Algorithm for Improved Voice Recognition and Authentication

Author

Tait, B. L., Akan, Ozgur, Series editor, Bellavista, Paolo, Series editor, Cao, Jiannong, Series editor, Dressler, Falko, Series editor, Ferrari, Domenico, Series editor, Gerla, Mario, Series editor, Kobayashi, Hisashi, Series editor, Palazzo, Sergio, Series editor, Sahni, Sartaj, Series editor, Shen, Xuemin (Sherman), Series editor, Stan, Mircea, Series editor, Xiaohua, Jia, Series editor, Zomaya, Albert, Series editor, Coulson, Geoffrey, Series editor, Georgiadis, Christos K., editor, Jahankhani, Hamid, editor, Pimenidis, Elias, editor, Bashroush, Rabih, editor, and Al-Nemrat, Ameer, editor

Published
2012
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9. Personal Data Handling And Processing In Canada

Author

Tait, B. Douglas

Subjects
Privacy, Right of -- Laws, regulations and rules, Data security -- Laws, regulations and rules, Personal information -- Laws, regulations and rules, Government regulation, Data security issue, Business, international
Abstract

Legitimate processing of PII Legitimate processing - grounds Does the law require that the holding of PII be legitimised on specific grounds, for example to meet the owner's legal obligations [...]

Published
2021

16. Biobanks of blood from donors and recipients of blood products

Author

Reesink, H. W., Engelfriet, C. P., Hyland, C. A., Coghlan, P., Tait, B., Wsolak, M., Keller, A. J., Henn, G., Mayr, W. R., Thomas, I., Osselaer, J.-C., Lambermont, M., Beaten, M., Wendel, S., Qiu, Y., Georgsen, J., Krusius, T., Mäki, T., Andreu, G., Morel, P., Lefrère, J.-J., Rebulla, P., Giovanelli, S., Butti, B., Lecchi, L., Mozzi, F., van Hilten, J. A., Zwaginga, J. J., Flanagan, P., Flesland, Ø., Brojer, E., Łętowska, M., Åkerblom, O., Norda, R., Prowse, C., Dow, B., Jarvis, L., Davidson, F., Kleinman, S., Bianco, C., Stramer, S. L., Dodd, R. Y., and Busch, M. P.

Published
2008

38. Associations between HLA and antibodies to collagen in rheumatoid arthritis

Author

Rowley, M.J., Tait, B., Doran, T., Emery, P., and Mackay, I.R.

Subjects
HLA histocompatibility antigens -- Physiological aspects, Rheumatoid arthritis -- Physiological aspects, Rheumatoid arthritis -- Causes of, Health
Abstract

Rheumatoid arthritis (RA) is a disease with a strong association to collagen antibodies. These associations have also been made with other joint and inflammatory diseases. Previous research has revealed that a response to type II collagen may contribute to the pathogenesis of RA. Histocompatibility antigens and HLA-typed patients were studied to extend previous work on this association and to determine if there is a specific gene involved in the pathogenesis of RA. The possible immunogenetic association of RA was studied by examining the antibodies to collagen. The association between RA and collagen antibodies is believed to be disease specific; however the importance of these antibodies needs to be clarified. There were no significant associations between HLA antigens and high antibody titers; however, in previous studies, antibody titers to collagen in patients who were DR4 positive were higher than those who were DR4 negative. There were significant associations made between HLA antigens and high antibody titers to collagen in the 105 patients studied. An association between RA and HLA-DR4 is well established and investigators believe that the gene DR4 may be susceptible to, or closely linked to RA. Clinical subsets of patients with RA may be associated with different combinations of HLA antigens and further research is needed to determine the gene that is activated in rheumatoid arthritis. (Consumer Summary produced by Reliance Medical Information, Inc.)

Published
1990

42. Antigen Society #26 Report (DR3, DR7, DQw2): Part 1

Author

Cambon-Thomsen, A., Calot, M., Sommer, E., Ohayon, E., Goeken, N., Kaplan, C., Mattiuz, P. L., Menicucci, A., Cross, R., Tait, B., Buc, M., Jeannet, M., Irle, C., Mayer, S., Tongio, M. M., Contu, L., Purpura, M., Nikaein, A., Mervart, H., Sullivan, K., Schweizer, R., Hansen, J. A., Du Toit, E., Hammond, M. G., and Dupont, Bo, editor

Published
1989
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44. Cyber Insurance: A Complicated Necessity

Author

Tait, B Douglas

Subjects
Data security -- Laws, regulations and rules, Computer insurance -- Laws, regulations and rules, Cyberterrorism -- Laws, regulations and rules, Insurance policies -- Laws, regulations and rules, Government regulation, Data security issue, Business, international
Abstract

Whether an organization suffers a cyber incident is no longer a question of 'if' but 'when' and as such, cyber security is a risk to be managed not solved. The [...]

Published
2018

45. Manipulated Data - The Hack You May Not Notice

Author

Tait, B Douglas

Subjects
Computer hackers -- Laws, regulations and rules, Personal information -- Laws, regulations and rules, Cyberterrorism -- Laws, regulations and rules, Government regulation, Hacker, Business, international
Abstract

Hacks, leaks and cyber attacks dominated the news in 2017. However, while these events were generally driven by the hackers intent to gather information either for their own use or [...]

Published
2018

49. Vox sanguinis / Biobanks of blood from donors and recipients of blood products

Author

Reesink, H. W., Engelfriet, C. P., Hyland, C. A., Coghlan, P., Tait, B., Wsolak, M., Keller, A. J., Henn, G., Mayr, Wolfgang, Thomas, I., Osselaer, J. C., Lambermont, M., Beaten, M., Amorim, L., Qiu, Y., Georgsen, J., Krusius, T., Maki, T., Andreu, G., Morel, P., Lefrere, J. J., Rebulla, P., Giovanelli, S., Butti, B., Lecchi, L., Mozzi, F., van Hilten, J. A., Zwaginga, J. J., Flanagan, P., Flesland, O., Brojer, E., Letowska, M., Akerblom, O., Norda, R., Prowse, C., Dow, B., Jarvis, L., Davidson, F., Kleinman, S., Bianco, C., Stramer, S. L., Dodd, R. Y., and Busch, M. P.

Published
2008

50. Tumour necrosis factor-{alpha} single nucleotide polymorphisms are not independent of HLA class I in UK Caucasians with adult onset idiopathic inflammatory myopathies

Author

Marriage, Fiona, Chinoy, H., Salway, F., John, S., Fertig, N., Tait, B. D., Oddis, C. V., Ollier, W. E R, and Cooper, R. G.

Subjects
Male, Adult, Risk, Genotype, Neuroscience(all), TNF, Single-nucleotide polymorphism, Polymyositis, Polymorphism, Single Nucleotide, Linkage Disequilibrium, Antibodies, Dermatomyositis, methods, Rheumatology, Gene Frequency, medicine, Humans, Pharmacology (medical), Genetic Predisposition to Disease, Myositis, Alleles, HLA-DQB1, business.industry, Tumor Necrosis Factor-alpha, Haplotype, Histocompatibility Antigens Class I, or, Middle Aged, medicine.disease, Genotype frequency, Cross-Sectional Studies, Haplotypes, Case-Control Studies, Immunology, Rna, Female, HLA, TNF-α, business, polymorphisms
Abstract

Objective. To investigate haplotype tagging single nucleotide polymorphisms (SNPs) in the tumour necrosis factor alpha (TNF-alpha) gene, in UK Caucasian idiopathic inflammatory myopathy (IIM) patients. Methods. A cross-sectional, case-control study of four TNF-alpha SNPs was undertaken, comparing cases of polymyositis (PM) (n = 121), dermatomyositis (DM) (n = 109) and myositis overlapping with other connective tissue diseases (CTD-overlap) (n = 73) with normal subjects (n = 177). Subgroup analyses were undertaken after stratifying for myositis specific/associated antibodies. Results. The TNF-308A allele demonstrated a strong association with each myositis disease subgroup vs controls [PM, odds ratio (OR) 2.8, 95% confidence interval 1.9-4.3; DM, OR 2.5, 1.6-3.8; CTD-overlap, OR 3.3, 2.1-5.1]. The TNF-308GA/AA genotype frequency was significantly increased vs controls (PM, OR 3.7, 2.1-6.3; DM, OR 3.2, 1.8-5.5; CTD-overlap, OR 5.0, 2.6-9.6) suggesting a dominant model. The association was strongest in patients possessing anti-aminoacyl transfer RNA synthetase (anti-synthetase) (OR 5.1, 3.3-8.0) or -PM-Scl (OR 5.0, 2.7-8.9) antibodies. The -1031T allele was also a significant risk factor in DM (OR 2.2, 1.4-3.6), anti-synthetase (OR 2.9, 1.6-5.3) and -PM-Scl (OR 5.6, 1.9-6.4) antibody positive patients. The TNF-308A association was lost after adjusting for HLA-B*08, but remained independent of HLA-DQB1*02 (both are alleles forming part of the common ancestral haplotype). The HLA-B*08/TNF-308A/DRB1*03/DQA1*05/DQB1*02 haplotype was a risk factor in all myositis subgroups vs controls (OR 3.0, 1.8-5.3). Conclusions. TNF-308A and -1031T alleles are significant risk factors in the IIMs. In the IIMs, the TNF-308A allele is part of the common ancestral haplotype, but is not independent of HLA-B*08 DA - 20070822IS - 1462-0324 (Print)LA - engPT - Journal ArticleSB - AIMSB - IM

Published
2007

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