45 results on '"Stuelten, Christina H."'
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2. GPCR Screening Reveals that the Metabolite Receptor HCAR3 Regulates Epithelial Proliferation, Migration, and Cellular Respiration
3. Smurf2 Regulates Inflammation and Collagen Processing in Cutaneous Wound Healing through Transforming Growth Factor-β/Smad3 Signaling
4. Cell motility in cancer invasion and metastasis: insights from simple model organisms
5. GPCR Screening Reveals that the Metabolite Receptor HCAR3 Regulates Epithelial Proliferation, Migration, and Cellular Respiration
6. Induced Chromosomal Aneuploidy Results in Global and Consistent Deregulation of the Transcriptome of Cancer Cells
7. TGF-β-induced alternative splicing of TAK1 promotes EMT and drug resistance
8. A GPCR screening in human keratinocytes identifies that the metabolite receptor HCAR3 controls epithelial proliferation, migration, and cellular respiration.
9. Data from Biological Responses to TGF-β in the Mammary Epithelium Show a Complex Dependency on Smad3 Gene Dosage with Important Implications for Tumor Progression
10. Supplementary Tables 1-2, Figures 1-2 from Biological Responses to TGF-β in the Mammary Epithelium Show a Complex Dependency on Smad3 Gene Dosage with Important Implications for Tumor Progression
11. Supplementary Figures S1 & S2, Table S1 from Bone Sialoprotein Mediates the Tumor Cell–Targeted Prometastatic Activity of Transforming Growth Factor β in a Mouse Model of Breast Cancer
12. Supplementary Figure Legends from Bone Sialoprotein Mediates the Tumor Cell–Targeted Prometastatic Activity of Transforming Growth Factor β in a Mouse Model of Breast Cancer
13. Segmentation and Cell Tracking of Breast Cancer Cells
14. Frontiers of Cancer Research: The Metastasis Challenge
15. Functional Interactions Between MMPs and TGF-β in Normal and Tumor Tissue
16. Transforming Growth Factor-β: An Agent of Change in the Tumor Microenvironment
17. Segmentation and Cell Tracking of Breast Cancer Cells
18. A novel approach for the generation of genetically modified mammary epithelial cell cultures yields new insights into TGFβ signaling in the mammary gland
19. Smad4-expression is decreased in breast cancer tissues: a retrospective study
20. Lysophosphatidic acid regulates the motility of MCF10CA1a breast cancer cell sheets via two opposing signaling pathways
21. Using the Dot Assay to Analyze Migration of Cell Sheets
22. Moving in and Out: Dispersion of Cells in Self-Generated Gradients
23. How Little Is Too Much? How Transient Tumor-Stromal Crosstalk Can Control Tumor Progression
24. Improving the design of the agarose spot assay for eukaryotic cell chemotaxis
25. SDF-1α Mediates Wound-Promoted Tumor Growth in a Syngeneic Orthotopic Mouse Model of Breast Cancer
26. Real-Time Motion Analysis Reveals Cell Directionality as an Indicator of Breast Cancer Progression
27. Biological Responses to TGF-β in the Mammary Epithelium Show a Complex Dependency on Smad3 Gene Dosage with Important Implications for Tumor Progression
28. Paracrine interactions between primary human macrophages and human fibroblasts enhance murine mammary gland humanization in vivo
29. miR-31 in cancer: Location matters
30. Abstract 3372: Analysis of putative tumor stem cell markers in the NCI-60 tumor cell line panel
31. Transient Tumor-Fibroblast Interactions Increase Tumor Cell Malignancy by a TGF-β Mediated Mechanism in a Mouse Xenograft Model of Breast Cancer
32. Complex Display of Putative Tumor Stem Cell Markers in the NCI60 Tumor Cell Line Panel
33. Acute Wounds Accelerate Tumorigenesis by a T Cell–Dependent Mechanism
34. Lentiviral reporter constructs for fluorescence tracking of the temporospatial pattern of Smad3 signaling
35. Bone Sialoprotein Mediates the Tumor Cell–Targeted Prometastatic Activity of Transforming Growth Factor β in a Mouse Model of Breast Cancer
36. Smad4-expression is decreased in breast cancer tissues: a retrospective study
37. Extracellular Matrix Proteoglycans Control the Fate of Bone Marrow Stromal Cells
38. Breast cancer cells induce stromal fibroblasts to express MMP-9 via secretion of TNF-α and TGF-β
39. Smad-Binding Defective Mutant of Transforming Growth Factor β Type I Receptor Enhances Tumorigenesis but Suppresses Metastasis of Breast Cancer Cell Lines
40. Extracellular Matrix Proteoglycans Control the Fate of Bone Marrow Stromal Cells.
41. A GPCR screening in human keratinocytes identifies that the metabolite receptor HCAR3 controls epithelial proliferation, migration, and cellular respiration.
42. Transient tumor-fibroblast interactions increase tumor cell malignancy by a TGF-Beta mediated mechanism in a mouse xenograft model of breast cancer.
43. Bone sialoprotein mediates the tumor cell-targeted prometastatic activity of transforming growth factor beta in a mouse model of breast cancer.
44. Breast cancer cells induce stromal fibroblasts to express MMP-9 via secretion of TNF-alpha and TGF-beta.
45. Smad-binding defective mutant of transforming growth factor beta type I receptor enhances tumorigenesis but suppresses metastasis of breast cancer cell lines.
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