218 results on '"Strathdee, Douglas"'
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2. KRAS allelic imbalance drives tumour initiation yet suppresses metastasis in colorectal cancer in vivo
3. Author Correction: Epithelial TGFβ engages growth-factor signalling to circumvent apoptosis and drive intestinal tumourigenesis with aggressive features
4. ALDH1L2 regulation of formate, formyl-methionine, and ROS controls cancer cell migration and metastasis
5. Tafazzin regulates neutrophil maturation and inflammatory response
6. Multi-scale in vivo imaging of tumour development using a germline conditional triple-reporter system
7. Epithelial TGFβ engages growth-factor signalling to circumvent apoptosis and drive intestinal tumourigenesis with aggressive features
8. Translation of secretory mRNAs must be restrained during early oncogenesis to permit hepatocellular carcinoma initiation
9. The RAC1 Target NCKAP1 Plays a Crucial Role in the Progression of Braf;Pten-Driven Melanoma in Mice
10. Dynamic Cardiolipin Synthesis Is Required for CD8+ T Cell Immunity
11. The amino acid transporter SLC7A5 is required for efficient growth of KRAS-mutant colorectal cancer
12. RNA helicase EIF4A1-mediated translation is essential for the GC response
13. eIF4A1 is essential for reprogramming the translational landscape of Wnt-driven colorectal cancers
14. Oncogenic BRAF, unrestrained by TGFβ-receptor signalling, drives right-sided colonic tumorigenesis
15. RAC1B modulates intestinal tumourigenesis via modulation of WNT and EGFR signalling pathways
16. BRF1 accelerates prostate tumourigenesis and perturbs immune infiltration
17. Brf1 loss and not overexpression disrupts tissues homeostasis in the intestine, liver and pancreas
18. A MYC–GCN2–eIF2α negative feedback loop limits protein synthesis to prevent MYC-dependent apoptosis in colorectal cancer
19. RNA helicase EIF4A1-mediated translation is essential for the GC response.
20. Phenotypic Characterization of Female Carrier Mice Heterozygous for Tafazzin Deletion
21. KRAS allelic imbalance drives tumour initiation yet suppresses metastasis in colorectal cancer in vivo
22. The eIF4A2 negative regulator of mRNA translation promotes extracellular matrix deposition to accelerate hepatocellular carcinoma initiation
23. Abstract P1016: Single Cell Transcriptomic Analysis Of Tafazzin Knockout Mice For Pathway And Therapeutic Discovery In Barth Syndrome
24. Abstract P2171: Lentivirus TAZ Gene Therapy Restores Metabolic Functions In Taz Ko Mice Model Of Barth Syndrome
25. Single Cell Transcriptomic Analysis in a Mouse Model of Barth Syndrome Reveals Cell-Specific Alterations in Gene Expression and Intercellular Communication
26. AAV Gene Therapy Prevents and Reverses Heart Failure in a Murine Knockout Model of Barth Syndrome
27. Data from Rab11-FIP1C Is a Critical Negative Regulator in ErbB2-Mediated Mammary Tumor Progression
28. Supplemental Figure 2 In vitro role of FIP1C in regulating cell proliferation in ErbB2/neuNDL primary cells from Rab11-FIP1C Is a Critical Negative Regulator in ErbB2-Mediated Mammary Tumor Progression
29. Supplemental Figure 3 Characterization of FIP1C/ErbB2/neuNDL cell lines in regulating tumor growth using athymic NCr mice from Rab11-FIP1C Is a Critical Negative Regulator in ErbB2-Mediated Mammary Tumor Progression
30. Table S2 from CRISPR/Cas9-Mediated Trp53 and Brca2 Knockout to Generate Improved Murine Models of Ovarian High-Grade Serous Carcinoma
31. Data from CRISPR/Cas9-Mediated Trp53 and Brca2 Knockout to Generate Improved Murine Models of Ovarian High-Grade Serous Carcinoma
32. Supplemental Materials and Methods from Rab11-FIP1C Is a Critical Negative Regulator in ErbB2-Mediated Mammary Tumor Progression
33. Supplemental Figure Legends from Rab11-FIP1C Is a Critical Negative Regulator in ErbB2-Mediated Mammary Tumor Progression
34. Supplemental Figure 1 Schematics of the tetracycline inducible-FIP1C-MTB and inducible knock-out FIP1C mouse models from Rab11-FIP1C Is a Critical Negative Regulator in ErbB2-Mediated Mammary Tumor Progression
35. Supplemental Figure 5 FIP1C is not distribute at adjacent junctional complexes. Clinical relevance of FIP1C in Her2-positive breast cancer patients from Rab11-FIP1C Is a Critical Negative Regulator in ErbB2-Mediated Mammary Tumor Progression
36. Supplemental Figure 4 FIP1C is a negative modulator of Akt and MAPK activation in an ErbB2-driven model from Rab11-FIP1C Is a Critical Negative Regulator in ErbB2-Mediated Mammary Tumor Progression
37. Genetic modifiers modulate phenotypic expression of tafazzin deficiency in a mouse model of Barth syndrome
38. Phenotypic Characterization of Male Tafazzin-Knockout Mice at 3, 6, and 12 Months of Age
39. Design and development of a cell marking system in transgenic mice
40. Morphogenesis of extra-embryonic tissues directs the remodelling of the mouse embryo at implantation
41. Correction: BRF1 accelerates prostate tumourigenesis and perturbs immune infiltration
42. Glycan degradation promotes macroautophagy
43. A noninvasive iRFP713 p53 reporter reveals dynamic p53 activity in response to irradiation and liver regeneration in vivo
44. Corrigendum: Differential requirements for MDM2 E3 activity during embryogenesis and in adult mice
45. TT2013 meeting report: the Transgenic Technology meeting visits Asia for the first time
46. Distal Transgene Insertion Affects CpG Island Maintenance during Differentiation
47. The in vivo function of the p53 target gene TIGAR
48. STEF/TIAM2-mediated Rac1 activity at the nuclear envelope regulates the perinuclear actin cap
49. Differential requirements for MDM2 E3 activity during embryogenesis and in adult mice
50. Correction: BRF1 accelerates prostate tumourigenesis and perturbs immune infiltration
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