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1. The Eurofever Registry for autoinflammatory diseases: results of the first 15 months of enrolment

4. Fighting Post-COVID and ME/CFS – development of curative therapies

6. Fieber

7. 69P Osimertinib is selective against NSCLC cells and modulates the multidrug-resistant phenotype in patient-derived cell cultures and co-cultures of NSCLC cells and fibroblasts

8. Entwicklung eines bedarfs- und bedürfnisorientierten interdisziplinären Versorgungsmodells für Kinder, Jugendliche und junge Erwachsene mit ME/CFS im Forschungsprojekt 'MECFS Kids Bavaria'

15. Clinical and epidemiological picture of B pertussis and B parapertussis infections after introduction of acellular pertussis vaccines

25. An international registry on autoinflammatory diseases: the Eurofever experience

26. Validation of the Auto-Inflammatory Diseases Activity Index (AIDAI) for hereditary recurrent fever syndromes

29. A preliminary score for the assessment of disease activity in hereditary recurrent fevers: results from the AIDAI (Auto-Inflammatory Diseases Activity Index) Consensus Conference.

30. PReS-FINAL-2194: Evidence-based clinical classification criteria for periodic fevers

31. PReS-FINAL-2335: Preliminary analysis of 85 patients with mevalonate kinase deficiency from the eurofever registry

33. OR13-004 – Evidence-based clinical classification criteria for periodic fevers

34. PW02-007 - The Eurofever registry: 3 years of enrollment

35. OP0175 The eurofever registry for autoinflammatory disease: Update on enrollment after 2 years

40. Clinical and functional characterisation of a novel TNFRSF1A c.605T>A/V173D cleavage site mutation associated with tumour necrosis factor receptor-associated periodic fever syndrome (TRAPS), cardiovascular complications and excellent response to etanercept treatment

50. Molecular analysis of the MVK and TNFRSF1A genes in patients with a clinical presentation typical of the hyperimmunoglobulinemia D with periodic fever syndrome: a low-penetrance TNFRSF1A variant in a heterozygous MVK carrier possibly influences the phenotype of hyperimmunoglobulinemia D with periodic fever syndrome or vice versa.

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