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11 results on '"Stockbauer, Anna"'

1. Tau accumulation is associated with dopamine deficiency in vivo in four-repeat tauopathies

Author

Ferschmann, Christian, Messerschmidt, Konstantin, Gnörich, Johannes, Barthel, Henryk, Marek, Ken, Palleis, Carla, Katzdobler, Sabrina, Stockbauer, Anna, Fietzek, Urban, Finze, Anika, Biechele, Gloria, Rumpf, Jost-Julian, Saur, Dorothee, Schroeter, Matthias L., Rullmann, Michael, Beyer, Leonie, Eckenweber, Florian, Wall, Stephan, Schildan, Andreas, Patt, Marianne, Stephens, Andrew, Classen, Joseph, Bartenstein, Peter, Seibyl, John, Franzmeier, Nicolai, Levin, Johannes, Höglinger, Günter U., Sabri, Osama, Brendel, Matthias, and Scheifele, Maximilian

Published
2024
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2. Metabolic network alterations as a supportive biomarker in dementia with Lewy bodies with preserved dopamine transmission

Author

Stockbauer, Anna, Beyer, Leonie, Huber, Maria, Kreuzer, Annika, Palleis, Carla, Katzdobler, Sabrina, Rauchmann, Boris-Stephan, Morbelli, Silvia, Chincarini, Andrea, Bruffaerts, Rose, Vandenberghe, Rik, Kramberger, Milica G., Trost, Maja, Garibotto, Valentina, Nicastro, Nicolas, Lathuilière, Aurélien, Lemstra, Afina W., van Berckel, Bart N. M., Pilotto, Andrea, Padovani, Alessandro, Ochoa-Figueroa, Miguel A., Davidsson, Anette, Camacho, Valle, Peira, Enrico, Bauckneht, Matteo, Pardini, Matteo, Sambuceti, Gianmario, Aarsland, Dag, Nobili, Flavio, Gross, Mattes, Vöglein, Jonathan, Perneczky, Robert, Pogarell, Oliver, Buerger, Katharina, Franzmeier, Nicolai, Danek, Adrian, Levin, Johannes, Höglinger, Günter U., Bartenstein, Peter, Cumming, Paul, Rominger, Axel, and Brendel, Matthias

Published
2024
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3. Subcortical tau is linked to hypoperfusion in connected cortical regions in 4-repeat tauopathies

Author

Roemer, Sebastian N, primary, Brendel, Matthias, additional, Gnoerich, Johannes, additional, Malpetti, Maura, additional, Zaganjori, Mirlind, additional, Quattrone, Andrea, additional, Gross, Mattes, additional, Steward, Anna, additional, Dewenter, Anna, additional, Wagner, Fabian, additional, Dehsarvi, Amir, additional, Ferschmann, Christian, additional, Wall, Stephan, additional, Palleis, Carla, additional, Rauchmann, Boris S, additional, Katzdobler, Sabrina, additional, Jaeck, Alexander, additional, Stockbauer, Anna, additional, Fietzek, Urban M, additional, Bernhardt, Alexander M, additional, Weidinger, Endy, additional, Zwergal, Andreas, additional, Stoecklein, Sophia, additional, Perneczky, Robert, additional, Barthel, Henryk, additional, Sabri, Osama, additional, Levin, Johannes, additional, Hoeglinger, Guenter U, additional, and Franzmeier, Nicolai, additional

Published
2024
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5. Metabolic network alterations as a supportive biomarker in dementia with Lewy bodies with preserved dopamine transmission

Author

Stockbauer, Anna, primary, Beyer, Leonie, additional, Huber, Maria, additional, Kreuzer, Annika, additional, Palleis, Carla, additional, Katzdobler, Sabrina, additional, Rauchmann, Boris-Stephan, additional, Morbelli, Silvia, additional, Chincarini, Andrea, additional, Bruffaerts, Rose, additional, Vandenberghe, Rik, additional, Kramberger, Milica G., additional, Trost, Maja, additional, Garibotto, Valentina, additional, Nicastro, Nicolas, additional, Lathuilière, Aurélien, additional, Lemstra, Afina W., additional, van Berckel, Bart N. M., additional, Pilotto, Andrea, additional, Padovani, Alessandro, additional, Ochoa-Figueroa, Miguel A., additional, Davidsson, Anette, additional, Camacho, Valle, additional, Peira, Enrico, additional, Bauckneht, Matteo, additional, Pardini, Matteo, additional, Sambuceti, Gianmario, additional, Aarsland, Dag, additional, Nobili, Flavio, additional, Gross, Mattes, additional, Vöglein, Jonathan, additional, Perneczky, Robert, additional, Pogarell, Oliver, additional, Buerger, Katharina, additional, Franzmeier, Nicolai, additional, Danek, Adrian, additional, Levin, Johannes, additional, Höglinger, Günter U., additional, Bartenstein, Peter, additional, Cumming, Paul, additional, Rominger, Axel, additional, and Brendel, Matthias, additional

Published
2023
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6. Detection Gap of Right-Asymmetric Neuronal Degeneration by CERAD Test Battery in Alzheimer's Disease

Author

Kreuzer, Annika, Sauerbeck, Julia, Scheifele, Maximilian, Stockbauer, Anna, Schönecker, Sonja, Prix, Catharina, Wlasich, Elisabeth, Loosli, Sandra V, M Kazmierczak, Philipp, Unterrainer, Marcus, Catak, Cihan, Janowitz, Daniel, Pogarell, Oliver, Palleis, Carla, Perneczky, Robert, Albert, Nathalie L, Bartenstein, Peter, Danek, Adrian, Buerger, Katharina, Levin, Johannes, Zwergal, Andreas, Rominger, Axel, Brendel, Matthias, and Beyer, Leonie

Subjects
hippocampal atrophy, 610 Medicine & health, ddc:610, FDG-PET, cognitive performance, Alzheimer’s disease, asymmetry, Neuroscience, Original Research
Abstract

Objectives: Asymmetric disease characteristics on neuroimaging are common in structural and functional imaging of neurodegenerative diseases, particularly in Alzheimer's disease (AD). However, a standardized clinical evaluation of asymmetric neuronal degeneration and its impact on clinical findings has only sporadically been investigated for F-18-fluorodeoxyglucose positron emission tomography (F-18-FDG-PET). This study aimed to evaluate the impact of lateralized neuronal degeneration on the detection of AD by detailed clinical testing. Furthermore, we compared associations between clinical evaluation and lateralized neuronal degeneration between FDG-PET hypometabolism and hippocampal atrophy. Finally, we investigated if specific subtests show associations with lateralized neuronal degeneration. Methods: One-hundred and forty-six patients with a clinical diagnosis of AD (age 71 �� 8) were investigated by FDG-PET and the "Consortium to Establish a Registry for Alzheimer's disease" (CERAD) test battery. For assessment of neuronal degeneration, FDG-PET hypometabolism in brain regions typically affected in AD were graded by visual (3D-surface projections) and semiquantitative analysis. Asymmetry of the hippocampus (left-right) in magnetic resonance tomography (MRI) was rated visually by the Scheltens scale. Measures of asymmetry were calculated to quantify lateralized neuronal degeneration and asymmetry scores were subsequently correlated with CERAD. Results: Asymmetry with left-dominant neuronal degeneration to FDG-PET was an independent predictor of cognitive impairment (visual: �� = -0.288, p < 0.001; semiquantitative: �� = -0.451, p < 0.001) when controlled for age, gender, years of education and total burden of neuronal degeneration, whereas hippocampal asymmetry to MRI was not (�� = -0.034; p = 0.731). Direct comparison of CERAD-PET associations in cases with right- and left-lateralized neuronal degeneration estimated a detection gap of 2.7 years for right-lateralized cases. Left-hemispheric neuronal degeneration was significantly associated with the total CERAD score and multiple subscores, whereas only MMSE (semiquantitative: �� = 0.429, p < 0.001) and constructional praxis (semiquantitative: �� = 0.292, p = 0.008) showed significant associations with right-hemispheric neuronal degeneration. Conclusions: Asymmetry of deteriorated cerebral glucose metabolism has a significant impact on the coupling between neuronal degeneration and cognitive function. Right dominant neuronal degeneration shows a delayed detection by global CERAD testing and requires evaluation of specific subdomains of cognitive testing.

Published
2021
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7. Detection Gap of Right-Asymmetric Neuronal Degeneration by CERAD Test Battery in Alzheimer’s Disease

Author

Kreuzer, Annika, primary, Sauerbeck, Julia, additional, Scheifele, Maximilian, additional, Stockbauer, Anna, additional, Schönecker, Sonja, additional, Prix, Catharina, additional, Wlasich, Elisabeth, additional, Loosli, Sandra V., additional, M. Kazmierczak, Philipp, additional, Unterrainer, Marcus, additional, Catak, Cihan, additional, Janowitz, Daniel, additional, Pogarell, Oliver, additional, Palleis, Carla, additional, Perneczky, Robert, additional, Albert, Nathalie L., additional, Bartenstein, Peter, additional, Danek, Adrian, additional, Buerger, Katharina, additional, Levin, Johannes, additional, Zwergal, Andreas, additional, Rominger, Axel, additional, Brendel, Matthias, additional, and Beyer, Leonie, additional

Published
2021
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11. Application errors associated with topical treatment of scabies: an observational study.

Author

Nemecek R, Stockbauer A, Lexa M, Poeppl W, and Mooseder G

Subjects
Administration, Topical, Adolescent, Austria, Humans, Ivermectin administration & dosage, Male, Prospective Studies, Young Adult, Insecticides administration & dosage, Medication Errors, Permethrin administration & dosage, Scabies drug therapy
Abstract

Background: In recent years, there has been a significant increase in scabies infestations throughout German-speaking countries. Given the high frequency of treatment failures, the question arises as to whether topical permethrin treatment is always performed correctly., Patients and Methods: Our department uses a fluorescent test cream to teach patients on how to correctly apply topical permethrin. In the context of a prospective observational study of 21 patients, we systematically assessed and analyzed potential application errors., Results: None of the participants succeeded in adequately applying the cream to the entire skin as previously instructed. The median number of regions left untreated was six (minimum: 2; maximum: 18), which included a median body surface area of 6 % (minimum: 2 %; maximum: 30 %). With regard to predilection sites of scabies, the ankles were left untreated in 62 % of cases, followed by the interdigital spaces (toes) (33 %) and the sacral region (24 %). All patients considered the pretreatment training to be very useful., Conclusions: The present findings clearly demonstrate potential shortcomings when it comes to the application of topical antiscabies treatment. This may provide a (potentially underestimated) explanation for the large number of reports on treatment failures in this regard, which falsely suggest potential treatment resistance., (© 2020 Deutsche Dermatologische Gesellschaft (DDG). Published by John Wiley & Sons Ltd.)

Published
2020
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