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2. A phase 1 trial of itacitinib, a selective JAK1 inhibitor, in patients with acute graft-versus-host disease

10. Baricitinib with cyclosporine eliminates acute graft rejection in fully mismatched skin and heart transplant models

12. An “off-the-shelf” fratricide-resistant CAR-T for the treatment of T cell hematologic malignancies

15. Imatinib mesylate for plexiform neurofibromas in patients with neurofibromatosis type 1: a phase 2 trial

17. Combined Treatment of Baricitinib and Cyclosporin Prevents Solid Organ Graft Rejection in Mice

19. Normal hematopoiesis and neurofibromin-deficient myeloproliferative disease require Erk

22. A murine model of neurofibromatosis type 2 that accurately phenocopies human schwannoma formation

23. Baricitinib prevents GvHD by increasing Tregs via JAK3 and treats established GvHD by promoting intestinal tissue repair via EGFR

25. Multiple increased osteoclast functions in individuals with neurofibromatosis type 1

28. Modeling Sézary Syndrome for Immunophenotyping and Anti-Tumor Effect of Ucart and Long-Acting Interleukin-7 Combination Therapy

30. Modeling Sézary Syndrome for Immunophenotyping and Anti-Tumor Effect of Ucart and Long-Acting Interleukin-7 Combination Therapy

33. A proteasome-resistant fragment of NIK mediates oncogenic NF-κB signaling in schwannomas

36. An Off-the-Shelf™ Fratricide-Resistant CAR-T for the Treatment of T Cell Hematologic Malignancies

38. Nf1 haploinsufficiency alters myeloid lineage commitment and function, leading to deranged skeletal homeostasis

41. Nf1Haploinsufficiency Alters Myeloid Lineage Commitment and Function, Leading to Deranged Skeletal Homeostasis

42. Pak2 Regulates Hematopoietic Progenitor Cell Proliferation, Survival, and Differentiation

44. A murine model of neurofibromatosis type 2 that accurately phenocopies human schwannoma formation

45. The Beak Sign

46. Hyperactive Transforming Growth Factor-β1 Signaling Potentiates Skeletal Defects in a Neurofibromatosis Type 1 Mouse Model

47. Normal hematopoiesis and neurofibromin-deficient myeloproliferative disease require Erk

49. Identification of molecular compartments and genetic circuitry in the developing mammalian kidney

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