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2. Accelerate Clinical Trials in Charcot-Marie-Tooth Disease (ACT-CMT): A Protocol to Address Clinical Trial Readiness in CMT1A

3. Reply: The p.Ser107Leu in BICD2 is a mutation ‘hot spot’ causing distal spinal muscular atrophy

4. Association Between Body Mass Index and Disability in Children With Charcot-Marie-Tooth Disease

5. Phenotypic and molecular insights into spinal muscular atrophy due to mutations in BICD2

7. Reliability of the Charcot‐Marie‐Tooth functional outcome measure

9. Electrophysiologic features of SYT2 mutations causing a treatable neuromuscular syndrome

10. Recruiting for an International Rare Disease Clinical Trial Readiness Study during the COVID‐19 pandemic: Challenges and solutions.

11. Biallelic mutations in SORDcause a common and potentially treatable hereditary neuropathy with implications for diabetes

12. Electrophysiological features of SYT2 mutations; a novel and treatable neuromuscular syndrome

13. Synaptotagmin 2 Mutations Cause an Autosomal-Dominant Form of Lambert-Eaton Myasthenic Syndrome and Nonprogressive Motor Neuropathy

14. Synaptotagmin 2 Mutations Cause an Autosomal-Dominant Form of Lambert-Eaton Myasthenic Syndrome and Nonprogressive Motor Neuropathy

15. Mutations in BICD2 Cause Dominant Congenital Spinal Muscular Atrophy and Hereditary Spastic Paraplegia

16. Electrophysiologic features ofSYT2mutations causing a treatable neuromuscular syndrome

17. Reply: The p.Ser107Leu inBICD2is a mutation ‘hot spot’ causing distal spinal muscular atrophy

18. Phenotypic and molecular insights into spinal muscular atrophy due to mutations in BICD2

20. Author Correction: Biallelic mutations in SORDcause a common and potentially treatable hereditary neuropathy with implications for diabetes

21. Genotype and phenotype spectrum of Charcot-Marie-Tooth disease due to mutations in SORD.

22. Author Correction: Biallelic mutations in SORD cause a common and potentially treatable hereditary neuropathy with implications for diabetes.

23. Biallelic mutations in SORD cause a common and potentially treatable hereditary neuropathy with implications for diabetes.

24. Electrophysiologic features of SYT2 mutations causing a treatable neuromuscular syndrome.

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