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1. Blood donor exposome and impact of common drugs on red blood cell metabolism.

8. Elevated sphingosine-1-phosphate promotes sickling and sickle cell disease progression

9. Erythrocyte adenosine A2B receptor prevents cognitive and auditory dysfunction by promoting hypoxic and metabolic reprogramming

10. Erythrocyte Metabolic Reprogramming by Sphingosine 1-Phosphate in Chronic Kidney Disease and Therapies

11. Maternal erythrocyte ENT1–mediated AMPK activation counteracts placental hypoxia and supports fetal growth

14. Blood Donor Exposome and Impact of Common Drugs on Red Blood Cell Metabolism

17. Adenosine A2B Receptor Controls Erythroid Lineage Commitment in Stress Erythropoiesis By Promoting Metabolic Reprogramming

18. Elevated ecto-5′-nucleotidase: a missing pathogenic factor and new therapeutic target for sickle cell disease

20. Structural and Functional Insight of Sphingosine 1-Phosphate-Mediated Pathogenic Metabolic Reprogramming in Sickle Cell Disease

21. Hypoxia modulates the purine salvage pathway and decreases red blood cell and supernatant levels of hypoxanthine during refrigerated storage

22. Erythrocyte purinergic signaling components underlie hypoxia adaptation

23. Elevated Adenosine Induces Placental DNA Hypomethylation Independent of A2B Receptor Signaling in Preeclampsia

24. Erythrocytes retain hypoxic adenosine response for faster acclimatization upon re-ascent

27. Structural and Functional Insight of Sphingosine 1-Phosphate-Mediated Pathogenic Metabolic Reprogramming in Sickle Cell Disease

28. Hypoxia-independent up-regulation of placental HIF-1α gene expression contributes to the pathogenesis of preeclampsia

29. AltitudeOmics: Red Blood Cell metabolic adaptation to high altitude hypoxia

30. AltitudeOmics: Red Blood Cell Metabolic Adaptation to High Altitude Hypoxia

31. Beneficial Role of Erythrocyte Adenosine A2B Receptor–Mediated AMP-Activated Protein Kinase Activation in High-Altitude Hypoxia

32. Hypoxia-mediated impaired erythrocyte Lands’ Cycle is pathogenic for sickle cell disease

33. Sphingosine-1-phosphate promotes erythrocyte glycolysis and oxygen release for adaptation to high-altitude hypoxia

35. Metabolomic Profiling Identifies Hypoxia-Induced Imbalanced Lands' Cycle Promotes Sickling and Disease Progression

37. Response to Letter Regarding Article, “Elevated Placental Adenosine Signaling Contributes to the Pathogenesis of Preeclampsia”

39. Elevated Placental Adenosine Signaling Contributes to the Pathogenesis of Preeclampsia

43. Erythrocyte Adenosine A2B Receptor-Mediated AMP-Activated Protein Kinase Prevents Hypoxia-Induced Tissue Injury in High Altitude By Inducing 2,3-Bisphosphoglycerate

44. Elevated Adenosine Induces Placental DNA Hypomethylation Independent of A2B Receptor Signaling in Preeclampsia.

46. Adenosine Is A Common Factor Regulating Erythrocyte 2,3-Bisphosphate Induction In Normal Individuals At High Altitude and In Patients With Sickle Cell Disease

47. Abstract 275: Tissue Transglutaminase Stabilizes Placental AT1 Receptor and Contributes to Pathophysiology in Preeclampsia

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