29 results on '"Smolgovsky, Sasha"'
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2. Deletion of MyD88 in T Cells Improves Antitumor Activity in Melanoma
3. Impaired T cell IRE1[alpha]/XBP1 signaling directs inflammation in experimental heart failure with preserved ejection fraction
4. Mechanistic and therapeutic distinctions between cardiosphere-derived cell and mesenchymal stem cell extracellular vesicle non-coding RNA
5. Fibroblasts and immune cells: at the crossroad of organ inflammation and fibrosis.
6. Impaired T cell IRE1α-XBP1 signaling directs inflammation in experimental Heart Failure with Preserved Ejection Fraction
7. T-Cell MyD88 Is a Novel Regulator of Cardiac Fibrosis Through Modulation of T-Cell Activation
8. Abstract P3064: Cytotoxic T-cells Drive Doxorubicin Induced Cardiac Fibrosis And Systolic Dysfunction
9. Abstract P3078: The Stimulator Of Interferon Genes (STING) Modulates Adverse Cardiac Remodeling Induced By Cardiac Pressure Overload: New Role Of Endothelial Cell Sting In Systolic Dysfunction
10. Abstract P3077: Withdrawal Of High Fat Diet And L-NAME Dampens T Cell Activation And Ameliorates Pathology In Experimental Heart Failure With Preserved Ejection Fraction (HFPEF) Through Modulation Of The T Cell IRE1a-XBP1 Axis
11. 354-OR: Attenuation of Obesity-Induced Vascular Inflammation by microRNA-485 in Endothelial Cells
12. Abstract P3063: Downregulation Of The T Cell Unfolded Protein Response Precedes T Cell Cardiac Infiltration In Experimental Heart Failure With Preserved Ejection Fraction
13. Abstract P3062: T-cell Myd88 Regulates Cardiac Fibrosis In Heart Failure By Modulating T-cell Survival And Pro-inflammatory Signaling
14. Proinflammatory T Cells with Downregulated Unfolded Protein Response Genes Contribute to Experimental Heart Failure with Preserved Ejection Fraction (HFpEF)
15. Sialomucin CD43 Plays a Deleterious Role in the Development of Experimental Heart Failure Induced by Pressure Overload by Modulating Cardiac Inflammation and Fibrosis
16. Abstract 116: T Cells Contribute To Diastolic Dysfunction And Cardiac Hypertrophy In A Preclinical Model Of Heart Failure With Preserved Ejection Fraction
17. Abstract 104: Deletion Of Myd88 In T-Cells Worsens Pathology In A Mouse Model Of Non-Ischemic Heart Failure Through Enhanced T-Cell Survival And Effector Function
18. Endothelial STING controls Tcell transmigration in an IFN-I dependent manner
19. Gut Dysbiosis and Barrier Disruption Are Associated with Diastolic Dysfunction in a Novel Mouse Model of Heart Failure with Preserved Ejection Fraction
20. Combined Risk Factors Induce T Cell‐Mediated Diastolic Dysfunction in a Novel Mouse Model of Heart Failure with Preserved Ejection Fraction (HFpEF)
21. Stimulator of Interferon Genes (STING) regulates Re‐endothelialization Following Vascular Injury
22. Adding insult to injury - Inflammation at the heart of cardiac fibrosis
23. Gut dysbiosis induced by cardiac pressure overload enhances adverse cardiac remodeling in a T cell-dependent manner
24. MicroRNA composition delineates cardiosphere-derived cell and mesenchymal stem cell extracellular vesicles
25. Stimulator of Interferon Genes (STING) Regulates Endothelial Healing and Reendothelialization Following Angioplasty‐Mediated Vascular Injury
26. Gut dysbiosis induced by cardiac pressure overload enhances adverse cardiac remodeling in a T cell-dependent manner.
27. Deletion of MyD88 in T-cells Improves Anti-Tumor Activity in Melanoma
28. Dephosphorylation of the RNA‐ Binding Protein Tristetraprolin by PP2A in LPS‐Stimulated Macrophages
29. Endothelial STING controls T cell transmigration in an IFNI-dependent manner.
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