34 results on '"Smiljanovic, Biljana"'
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2. Intestinal Microbiota Reduction Followed by Fasting Discloses Microbial Triggering of Inflammation in Rheumatoid Arthritis
3. Synovial tissue transcriptomes of long-standing rheumatoid arthritis are dominated by activated macrophages that reflect microbial stimulation
4. Genomic stratification by expression of HLA-DRB4 alleles identifies differential innate and adaptive immune transcriptional patterns - A strategy to detect predictors of methotrexate response in early rheumatoid arthritis
5. Monocyte alterations in rheumatoid arthritis are dominated by preterm release from bone marrow and prominent triggering in the joint
6. 05.08 Increased turnover of monocytes in patients with rheumatoid arthritis identified by transcriptome and cytometric profiling
7. The multifaceted balance of TNF-α and type I/II interferon responses in SLE and RA: how monocytes manage the impact of cytokines
8. Transcriptomes of two cohorts of patients with axial spondyloarthritis. Comparing microarrays and RNAseq
9. Defining TNF-α- and LPS-induced gene signatures in monocytes to unravel the complexity of peripheral blood transcriptomes in health and disease
10. RA-, OA- and ND-Synovial Tissue: High Performance Chip Data Analysis (HPCDA) with RMA Quantile normalised Signals selecting significant and most predictive genes
11. Classification with Prediction Analysis for Microarrays (PAM) of 49 independent GeneChips validates HPCDA significant gene lists of 30 synovial tissue chips of RA, OA and ND
12. Peripheral blood mononuclear cells are hypomethylated in active rheumatoid arthritis and methylation correlates with disease activity
13. Monocytes transcriptom from sle and ra patients revelaed the disease-specific imprint of TNFα, IFNα and IFNγ
14. The type 1 interferon signature: facts, fads and fallacies
15. Advantage of new HPCDA parameters with higher DNA-chip number datasets
16. AB0122 TNF INHIBITION IN RHEUMATOID ARTHRITIS RESPONDERS TO CERTOLIZUMAB-PEGOL REDUCES LYMPHOCYTE RECRUITMENT FROM BLOOD WHILE MONOCYTE TURNOVER REMAINS INCREASED
17. FRI0360 ANALYSIS OF BLOOD MONOCYTE TRANSCRIPTOMES AND BONE MARROW SAMPLES OF PATIENTS WITH AXIAL SPONDYLOARTHRITIS REVEALS THEIR CHANGES RELATED TO ACTIVATION AND MYELOPOIESIS
18. Peripheral blood mononuclear cells are hypomethylated in active rheumatoid arthritis and methylation correlates with disease activity.
19. Monocyte alterations in rheumatoid arthritis are dominated by preterm release from bone marrow and prominent triggering in the joint
20. Bone marrow and blood monocytes from rheumatoid arthritis patients show premature transcriptomes and reveal incremental inflammatory activation from bone marrow to blood and joint
21. 05.08 Increased turnover of monocytes in patients with rheumatoid arthritis identified by transcriptome and cytometric profiling
22. Prediction of response to methotrexate treatment in RA using mRNA and miRNA biomarkers
23. Cell-Type Specific Type I Interferon Signatures in Autologous Stem Cell Transplanted Lupus Patients: Different Molecular Behavior Between CD4+ T Cells and Monocytes
24. Cell-Type Specific Type I Interferon Signatures in Systemic Lupus Erythematosus and Viral Infection: What Makes the Difference?
25. Transcriptional profiling of peripheral blood monocytes from SLE, RA and AS patients
26. Defining cytokine-induced gene expression profiles in monocytes to unravel complexity of monocytes transcriptomes in health and disease
27. Upregulation of Immunoproteasome Subunits in Myositis Indicates Active Inflammation with Involvement of Antigen Presenting Cells, CD8 T-Cells and IFNγ
28. A8.16 T helper lymphocytes and monocytes as biosensors of type I interferon responses in viral infection and autoimmunity
29. A1.18 From tissue- and cell-specific transcriptomes to candidate markers in rheumatoid arthritis
30. Cell-Specific Type I IFN Signatures in Autoimmunity and Viral Infection: What Makes the Difference?
31. A2.6 Cell-Specific Type I IFN Signatures in Autoimmunity and Viral Infection: What Makes the Difference?
32. A10.25 Synovial Tissue Transcriptome and Synovial Fluid Proteome have Stronger Discriminatory Power than Serum in Dissecting Inflammation in RA
33. A10.6 Dissecting Disease-Specific Differences in RA and OA by Transcriptome Analyses of Synovial Tissue, Blood and Bone Marrow Monocytes
34. Upregulation of Immunoproteasome Subunits in Myositis Indicates Active Inflammation with Involvement of Antigen Presenting Cells, CD8 T-Cells and IFNγ
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