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13 results on '"Smevik, Hanne"'

1. Accelerated Aging after Traumatic Brain Injury: An ENIGMA Multi‐Cohort Mega‐Analysis

Author

Dennis, Emily L, primary, Vervoordt, Samantha, additional, Adamson, Maheen M, additional, Houshang, Amiri, additional, Bigler, Erin D, additional, Caeyenberghs, Karen, additional, Cole, James H, additional, Dams‐O'Connor, Kristen, additional, Deutscher, Evelyn M, additional, Dobryakova, Ekaterina, additional, Genova, Helen M, additional, Grafman, Jordan H, additional, Håberg, Asta K, additional, Hellstrøm, Torgeir, additional, Irimia, Andrei, additional, Koliatsos, Vassilis E, additional, Lindsey, Hannah M, additional, Livny, Abigail, additional, Menon, David K, additional, Merkley, Tricia L, additional, Mohamed, Abdalla Z, additional, Mondello, Stefania, additional, Monti, Martin M, additional, Newcombe, Virginia FJ, additional, Newsome, Mary R, additional, Ponsford, Jennie, additional, Rabinowitz, Amanda, additional, Smevik, Hanne, additional, Spitz, Gershon, additional, Venkatesan, Umesh M, additional, Westlye, Lars T, additional, Zafonte, Ross, additional, Thompson, Paul M, additional, Wilde, Elisabeth A, additional, Olsen, Alexander, additional, and Hillary, Frank G, additional

Published
2024
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2. Metacognitive therapy for people experiencing persistent post-concussion symptoms following mild traumatic brain injury: a preliminary multiple case-series study

Author

Rauwenhoff, Johanne, primary, Hagen, Roger, additional, Karaliute, Migle, additional, Hjemdal, Odin, additional, Kennair, Leif Edward Ottesen, additional, Solem, Stian, additional, Asarnow, Robert F., additional, Einarsen, Cathrine, additional, Halvorsen, Joar, additional, Paoli, Stephanie, additional, Saksvik, Simen, additional, Smevik, Hanne, additional, Storvig, Gøril, additional, Wells, Adrian, additional, Skandsen, Toril, additional, and Olsen, Alexander, additional

Published
2024
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3. Accelerated Aging after Traumatic Brain Injury: an ENIGMA Multi-Cohort Mega-Analysis

Author

Dennis, Emily L, primary, Vervoordt, Samantha, additional, Adamson, Maheen M, additional, Amiri, Houshang, additional, Bigler, Erin, additional, Caeyenberghs, Karen, additional, Cole, James H, additional, Dams-O'Connor, Kristen, additional, Deutscher, Evelyn M, additional, Dobryakova, Ekaterina, additional, Genova, Helen M, additional, Grafman, Jordan H, additional, Haberg, Asta K, additional, Hellstrom, Torgeir, additional, Irimia, Andrei, additional, Koliatsos, Vassilis E, additional, Lindsey, Hannah M, additional, Livny, Abigail, additional, Menon, David K, additional, Merkley, Tricia L, additional, Mohamed, Abdalla Z, additional, Mondello, Stefania, additional, Monti, Martin M, additional, Newcombe, Virginia FJ, additional, Newsome, Mary R, additional, Ponsford, Jennie, additional, Smevik, Hanne, additional, Spitz, Gershon, additional, Westlye, Lars T, additional, Zafonte, Ross, additional, Thompson, Paul M, additional, Wilde, Elisabeth A, additional, Olsen, Alexander, additional, and Hillary, Frank G, additional

Published
2023
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4. Poorer sleep health is associated with altered brain activation during cognitive control processing in healthy adults

Author

Smevik, Hanne, primary, Habli, Sarah, additional, Saksvik, Simen Berg, additional, Kliem, Elisabeth, additional, Evensmoen, Hallvard Røe, additional, Conde, Virginia, additional, Petroni, Agustin, additional, Asarnow, Robert F, additional, Dennis, Emily L, additional, Eikenes, Live, additional, Kallestad, Håvard, additional, Sand, Trond, additional, Thompson, Paul M, additional, Saksvik-Lehouillier, Ingvild, additional, Håberg, Asta Kristine, additional, and Olsen, Alexander, additional

Published
2023
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5. Brain age prediction in subjects with sleep deprivation based on the ENIGMA-Sleep data

Author

Hoffstaedter, Felix, Antonopoulos, Georgios, Bajaj, Sahil, Bi, Hanwen, Chu, Congying, Cross, Nathan, Deantoni, Michele, Vu, Thien, Grova, Christophe, Eickhoff, Simon, Elmenhorst, Eva-Maria, Elvsåshagen, Torbjørn, Holst, Sebastian, Jahanshad, Neda, Jegou, Aude, Jones, Richard, Landolt, Hans-Peter, Lekander, Mats, Mohamed, Abdalla, Mortazavi, Nasrin, Nilsonne, Gustav, Olsen, Alexander, Patil, Kaustubh, Peigneux, Philippe, Pomares, Florence, Poudel, Govinda, Raimondo, Federico, Smevik, Hanne, Spiegelhalder, Kai, Tamm, Sandra, Thomopoulos, Sophia, Thompson, Paul, Vandewalle, Gilles, Voldsbekk, Irene, Åkerstedt, Torbjörn, Tahmasian, Masoud, and Elmenhorst, David

Subjects
Sleep Medicine, ENIGMA-Sleep, Neuroscience and Neurobiology, Medicine and Health Sciences, Medical Specialties, Life Sciences, Social and Behavioral Sciences, sleep deprivation
Abstract

Sleep is essential for the maintenance of human physical health (Grandner et al., 2012; Reid et al., 2006), cognitive performance (Ferrie et al., 2011; Leng et al., 2017), and mental health (Freeman et al., 2017; Joao et al., 2018). Experimental sleep deprivation and different degrees of sleep restriction permit the characterization of human sleep behaviors and brain's responses to inadequate sleep (Durmer and Dinges, 2005; Elmenhorst et al., 2017; Elmenhorst et al., 2018; Van Dongen et al., 2003). Sleep disruption is known to impact not only brain functions but also affects brain anatomy; earlier studies found that sleep deprivation is associated with lower volumes of gray matter across various brain regions (Åkerstedt et al., 2020; Liu et al., 2014; Long et al., 2020; Sun et al., 2020), however with inconsistent localization of effects. Other studies reported alterations in white matter microstructure (Elvsåshagen et al., 2015; Voldsbekk et al., 2021), increased ventricles size, and cognitive decline (Lo et al., 2014). In addition, one-night sleep deprivation was found to increase the Aβ burden in the right hippocampus and thalamus of healthy controls (Shokri-Kojori 2018), suggesting an interrupted clearance pathway due to sleep deprivation. These prior studies suggest that sleep deprivation may impact anatomical features of the brain, but there is a need for large-scale replications and extensions of previous work. Increased brain age compared to chronological age is a risk factor for several neurodegenerative and psychiatric disorders (Wyss-Coray, 2016). Brain-age prediction is a widely studied topic that aims to estimate the trajectory of brain aging (Franke et al., 2019), and its association with pathological conditions. MRI-derived features used to predict individuals’ brain age have proven to constitute a proxy for overall health (Habes et al., 2016, Koutsouleris et al., 2014, Cole et al., 2018). Sleep patterns also change with aging. In particular, older adults tend to sleep less, and present a more fragmented sleep with lower slow-wave sleep. They also present a reduced sleep rebound following sleep deprivation, report less sleepiness under acute sleep deprivation conditions, and report a smaller increase in lapses of attention after sleep deprivation [23]. However, little is known on whether brain-age models reflect the impact of sleep deprivation on the brain structure - i.e., the difference between predicted age and real age (so-called brain-age delta) using MRI data. We hypothesized that the morphological alterations associated with sleep deprivation would lead to an increased brain-delta score.

Published
2022
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6. Poorer Sleep Health is Associated With Altered Brain Activation During Cognitive Control Processing in Healthy Adults

Author

Smevik, Hanne, primary, Habli, Sarah, additional, Saksvik, Simen Berg, additional, Kliem, Elisabeth, additional, Evensmoen, Hallvard Røe, additional, Conde, Virginia, additional, Petroni, Agustin, additional, Asarnow, Robert F., additional, Dennis, Emily L., additional, Eikenes, Live, additional, Kallestad, Håvard, additional, Sand, Trond, additional, Thompson, Paul M., additional, Saksvik-Lehouillier, Ingvild, additional, Håberg, Asta Kristine, additional, and Olsen, Alexander, additional

Published
2022
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8. Poor sleep quality is associated with greater negative consequences for cognitive control function and psychological health after mild traumatic brain injury than after orthopedic injury.

Author

Saksvik, Simen Berg, primary, Smevik, Hanne, additional, Stenberg, Jonas, additional, Follestad, Turid, additional, Vik, Anne, additional, Håberg, Asta, additional, Asarnow, Robert F., additional, Kallestad, Håvard, additional, Skandsen, Toril, additional, and Olsen, Alexander, additional

Published
2021
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9. ENIGMA-Sleep: Challenges, opportunities, and the road map

Author

Tahmasian, Masoud, Aleman, André, Andreassen, Ole A, Arab, Zahra, Baillet, Marion, Benedetti, Francesco, Bresser, Tom, Bright, Joanna, Chee, Michael W L, Chylinski, Daphne, Cheng, Wei, Deantoni, Michele, Dresler, Martin, Eickhoff, Simon B, Eickhoff, Claudia R, Elvsåshagen, Torbjørn, Feng, Jianfeng, Foster-Dingley, Jessica C, Ganjgahi, Habib, Grabe, Hans J, Groenewold, Nynke A, Ho, Tiffany C, Bong Hong, Seung, Houenou, Josselin, Irungu, Benson, Jahanshad, Neda, Khazaie, Habibolah, Kim, Hosung, Koshmanova, Ekaterina, Kocevska, Desi, Kochunov, Peter, Lakbila-Kamal, Oti, Leerssen, Jeanne, Li, Meng, Luik, Annemarie I, Muto, Vincenzo, Narbutas, Justinas, Nilsonne, Gustav, O'Callaghan, Victoria S, Olsen, Alexander, Osorio, Ricardo S, Poletti, Sara, Poudel, Govinda, Reesen, Joyce E, Reneman, Liesbeth, Reyt, Mathilde, Riemann, Dieter, Rosenzweig, Ivana, Rostampour, Masoumeh, Saberi, Amin, Schiel, Julian, Schmidt, Christina, Schrantee, Anouk, Sciberras, Emma, Silk, Tim J, Sim, Kang, Smevik, Hanne, Soares, Jair C, Spiegelhalder, Kai, Stein, Dan J, Talwar, Puneet, Tamm, Sandra, Teresi, Giana L, Valk, Sofie L, Van Someren, Eus, Vandewalle, Gilles, Van Egroo, Maxime, Völzke, Henry, Walter, Martin, Wassing, Rick, Weber, Frederik D, Weihs, Antoine, Westlye, Lars Tjelta, Wright, Margaret J, Wu, Mon-Ju, Zak, Nathalia, Zarei, Mojtaba, Tahmasian, Masoud, Aleman, André, Andreassen, Ole A, Arab, Zahra, Baillet, Marion, Benedetti, Francesco, Bresser, Tom, Bright, Joanna, Chee, Michael W L, Chylinski, Daphne, Cheng, Wei, Deantoni, Michele, Dresler, Martin, Eickhoff, Simon B, Eickhoff, Claudia R, Elvsåshagen, Torbjørn, Feng, Jianfeng, Foster-Dingley, Jessica C, Ganjgahi, Habib, Grabe, Hans J, Groenewold, Nynke A, Ho, Tiffany C, Bong Hong, Seung, Houenou, Josselin, Irungu, Benson, Jahanshad, Neda, Khazaie, Habibolah, Kim, Hosung, Koshmanova, Ekaterina, Kocevska, Desi, Kochunov, Peter, Lakbila-Kamal, Oti, Leerssen, Jeanne, Li, Meng, Luik, Annemarie I, Muto, Vincenzo, Narbutas, Justinas, Nilsonne, Gustav, O'Callaghan, Victoria S, Olsen, Alexander, Osorio, Ricardo S, Poletti, Sara, Poudel, Govinda, Reesen, Joyce E, Reneman, Liesbeth, Reyt, Mathilde, Riemann, Dieter, Rosenzweig, Ivana, Rostampour, Masoumeh, Saberi, Amin, Schiel, Julian, Schmidt, Christina, Schrantee, Anouk, Sciberras, Emma, Silk, Tim J, Sim, Kang, Smevik, Hanne, Soares, Jair C, Spiegelhalder, Kai, Stein, Dan J, Talwar, Puneet, Tamm, Sandra, Teresi, Giana L, Valk, Sofie L, Van Someren, Eus, Vandewalle, Gilles, Van Egroo, Maxime, Völzke, Henry, Walter, Martin, Wassing, Rick, Weber, Frederik D, Weihs, Antoine, Westlye, Lars Tjelta, Wright, Margaret J, Wu, Mon-Ju, Zak, Nathalia, and Zarei, Mojtaba

Abstract

Neuroimaging and genetics studies have advanced our understanding of the neurobiology of sleep and its disorders. However, individual studies usually have limitations to identifying consistent and reproducible effects, including modest sample sizes, heterogeneous clinical characteristics and varied methodologies. These issues call for a large-scale multi-centre effort in sleep research, in order to increase the number of samples, and harmonize the methods of data collection, preprocessing and analysis using pre-registered well-established protocols. The Enhancing NeuroImaging Genetics through Meta-Analysis (ENIGMA) consortium provides a powerful collaborative framework for combining datasets across individual sites. Recently, we have launched the ENIGMA-Sleep working group with the collaboration of several institutes from 15 countries to perform large-scale worldwide neuroimaging and genetics studies for better understanding the neurobiology of impaired sleep quality in population-based healthy individuals, the neural consequences of sleep deprivation, pathophysiology of sleep disorders, as well as neural correlates of sleep disturbances across various neuropsychiatric disorders. In this introductory review, we describe the details of our currently available datasets and our ongoing projects in the ENIGMA-Sleep group, and discuss both the potential challenges and opportunities of a collaborative initiative in sleep medicine.

Published
2021

10. ENIGMA‐Sleep: Challenges, opportunities, and the road map

Author

Tahmasian, Masoud, primary, Aleman, André, additional, Andreassen, Ole A., additional, Arab, Zahra, additional, Baillet, Marion, additional, Benedetti, Francesco, additional, Bresser, Tom, additional, Bright, Joanna, additional, Chee, Michael W.L., additional, Chylinski, Daphne, additional, Cheng, Wei, additional, Deantoni, Michele, additional, Dresler, Martin, additional, Eickhoff, Simon B., additional, Eickhoff, Claudia R., additional, Elvsåshagen, Torbjørn, additional, Feng, Jianfeng, additional, Foster‐Dingley, Jessica C., additional, Ganjgahi, Habib, additional, Grabe, Hans J., additional, Groenewold, Nynke A., additional, Ho, Tiffany C., additional, Bong Hong, Seung, additional, Houenou, Josselin, additional, Irungu, Benson, additional, Jahanshad, Neda, additional, Khazaie, Habibolah, additional, Kim, Hosung, additional, Koshmanova, Ekaterina, additional, Kocevska, Desi, additional, Kochunov, Peter, additional, Lakbila‐Kamal, Oti, additional, Leerssen, Jeanne, additional, Li, Meng, additional, Luik, Annemarie I., additional, Muto, Vincenzo, additional, Narbutas, Justinas, additional, Nilsonne, Gustav, additional, O’Callaghan, Victoria S., additional, Olsen, Alexander, additional, Osorio, Ricardo S., additional, Poletti, Sara, additional, Poudel, Govinda, additional, Reesen, Joyce E., additional, Reneman, Liesbeth, additional, Reyt, Mathilde, additional, Riemann, Dieter, additional, Rosenzweig, Ivana, additional, Rostampour, Masoumeh, additional, Saberi, Amin, additional, Schiel, Julian, additional, Schmidt, Christina, additional, Schrantee, Anouk, additional, Sciberras, Emma, additional, Silk, Tim J., additional, Sim, Kang, additional, Smevik, Hanne, additional, Soares, Jair C., additional, Spiegelhalder, Kai, additional, Stein, Dan J., additional, Talwar, Puneet, additional, Tamm, Sandra, additional, Teresi, Giana l., additional, Valk, Sofie L., additional, Someren, Eus, additional, Vandewalle, Gilles, additional, Van Egroo, Maxime, additional, Völzke, Henry, additional, Walter, Martin, additional, Wassing, Rick, additional, Weber, Frederik D., additional, Weihs, Antoine, additional, Westlye, Lars Tjelta, additional, Wright, Margaret J., additional, Wu, Mon‐Ju, additional, Zak, Nathalia, additional, and Zarei, Mojtaba, additional

Published
2021
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11. Poor sleep quality is associated with greater negative consequences for cognitive control function and psychological health after mild traumatic brain injury than after orthopedic injury

Author

Saksvik, Simen, primary, Smevik, Hanne, additional, Stenberg, Jonas, additional, Follestad, Truid, additional, Vik, Anne, additional, Håberg, Asta, additional, Asarnow, Robert F., additional, Kallestad, Håvard, additional, Skandsen, Toril, additional, and Olsen, Alexander, additional

Published
2020
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12. Low dose oxytocin delivered intranasally with Breath Powered device affects social-cognitive behavior: a randomized 4-way crossover trial with nasal cavity dimension assessment

Author

Quintana, Daniel, Westlye, Lars Tjelta, Rustan, Øyvind, Tesli, Natalia, Poppy, Claire, Smevik, Hanne, Tesli, Martin Steen, Røine, Marianne, Mahmoud, Ramy, Smerud, Knut Terje, Djupesland, Per G., and Andreassen, Ole Andreas

Abstract

Despite the promise of intranasal oxytocin (OT) for modulating social behavior, recent work has provided mixed results. This may relate to suboptimal drug deposition achieved with conventional nasal sprays, inter-individual differences in nasal physiology and a poor understanding of how intranasal OT is delivered to the brain in humans. Delivering OT using a novel ‘Breath Powered’ nasal device previously shown to enhance deposition in intranasal sites targeted for nose-to-brain transport, we evaluated dose-dependent effects on social cognition, compared response with intravenous (IV) administration of OT, and assessed nasal cavity dimensions using acoustic rhinometry. We adopted a randomized, double-blind, double-dummy, crossover design, with 16 healthy male adults completing four single-dose treatments (intranasal 8 IU (international units) or 24 IU OT, 1 IU OT IV and placebo). The primary outcome was social cognition measured by emotional ratings of facial images. Secondary outcomes included the pharmacokinetics of OT, vasopressin and cortisol in blood and the association between nasal cavity dimensions and emotional ratings. Despite the fact that all the treatments produced similar plasma OT increases compared with placebo, there was a main effect of treatment on anger ratings of emotionally ambiguous faces. Pairwise comparisons revealed decreased ratings after 8 IU OT in comparison to both placebo and 24 IU OT. In addition, there was an inverse relationship between nasal valve dimensions and anger ratings of ambiguous faces after 8-IU OT treatment. These findings provide support for a direct nose-to-brain effect, independent of blood absorption, of low-dose OT delivered from a Breath Powered device.

Published
2015

13. Metacognitive Therapy for People Experiencing Persistent Post-Concussion Symptoms Following Mild Traumatic Brain Injury: A Preliminary Multiple Case-Series Study.

Author

Rauwenhoff JCC, Hagen R, Karaliute M, Hjemdal O, Kennair LEO, Solem S, Asarnow RF, Einarsen C, Halvorsen JØ, Paoli S, Saksvik SB, Smevik H, Storvig G, Wells A, Skandsen T, and Olsen A

Abstract

After mild traumatic brain injury (mTBI), a subgroup of individuals experience persistent post-concussion symptoms (PPCS) that include headaches, cognitive difficulties, and fatigue. The aim of this preliminary study was to investigate possible effects associated with metacognitive therapy (MCT) on PPCS, maladaptive coping strategies, and positive and negative metacognitive beliefs following mTBI. A pre-post design supplemented with single-case A-B replication series to assess potential MCT mechanisms was used. Of the nine participants who received MCT, all experienced a decrease in PPCS, which constituted a reliable improvement for eight participants. For eight participants (we could calculate effect sizes for eight out of nine participants), moderate to very large decreases in maladaptive coping styles and positive and negative metacognitive beliefs were observed. However, based on visual analyses, participants 6, 8, and 9 show a downward baseline trend regarding MCT mechanisms that may have persisted into the intervention phase. No adverse events were reported. In conclusion, MCT was associated with improvements in PPCS and unhelpful psychological mechanisms, but caution is required in interpreting this association. Future research using formal single-case replication on symptom measures and randomized controlled trials appears to be justified., (© The Author(s) 2024. Published by Mary Ann Liebert, Inc.)

Published
2024
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