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Your search keyword '"Singh, Ajit S."' showing total 5 results
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5 results on '"Singh, Ajit S."'

1. High Temperature Resistant Thermosetting Resin Materials

Author

Singh, Ajit S., Das, Vishal, Chopra, Swati, Pandey, Arvind Kr., Namburi, Eswara Prasad, Mudali, U. Kamachi, Series Editor, Basu, Bikramjit, Editorial Board Member, Mishra, Suman K., Editorial Board Member, Prasad, N. Eswara, Editorial Board Member, Narayana Murty, S.V.S., Editorial Board Member, Singh, R.N., Editorial Board Member, Balamuralikrishnan, R., Editorial Board Member, Namburi, Eswara Prasad, editor, Wanhill, R. J. H., editor, and Setua, Dipak Kumar, editor

Published
2024
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5. The role of the glutamatergic system in posttraumatic stress disorder.

Author

Nair J and Singh Ajit S

Subjects
Animals, Calcium Channel Blockers therapeutic use, Corticotropin-Releasing Hormone metabolism, Glutamic Acid metabolism, Humans, Hypothalamo-Hypophyseal System metabolism, Hypothalamo-Hypophyseal System physiopathology, Lamotrigine, Pituitary-Adrenal System metabolism, Pituitary-Adrenal System physiopathology, Stress Disorders, Post-Traumatic drug therapy, Stress Disorders, Post-Traumatic metabolism, Stress, Physiological metabolism, Stress, Physiological physiopathology, Triazines therapeutic use, Corticotropin-Releasing Hormone physiology, Glutamic Acid physiology, Stress Disorders, Post-Traumatic physiopathology
Abstract

Antiglutamatergic agents, such as lamotrigine, have been used successfully for the treatment of posttraumatic stress disorder (PTSD). They could be potentially acting through the stabilization of the corticotropin-releasing factor (CRF) systems. Glutamate mediates CRF release in various brain regions involved in the pathophysiology of PTSD, antiglutamatergic agents could stabilize the CRF system and, thereby, improve the symptom complex of PTSD (reexperiencing, hyperarousal, and avoidance). The role of glutamate and CRF in PTSD and other anxiety disorders are still being elucidated. However, it is clear that the glutamatergic systems play a role in the pathophysiology of PTSD.

Published
2008
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