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466 results on '"Singh, Abhay K."'

8. Supplementary Figure S5 from Development of a [89Zr]Zr-labeled Human Antibody using a Novel Phage-displayed Human scFv Library

Author

Singh, Abhay K., primary, Lewis, Calvin D., primary, Boas, Cristian A.W.V., primary, Diebolder, Philipp, primary, Jethva, Prashant N., primary, Rhee, Aaron, primary, Song, Jong Hee, primary, Goo, Young Ah, primary, Li, Shunqian, primary, Nickels, Michael L., primary, Liu, Yongjian, primary, Rogers, Buck E., primary, Kapoor, Vaishali, primary, and Hallahan, Dennis E., primary

Published
2024
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9. Supplementary Table S3 from Development of a [89Zr]Zr-labeled Human Antibody using a Novel Phage-displayed Human scFv Library

Author

Singh, Abhay K., primary, Lewis, Calvin D., primary, Boas, Cristian A.W.V., primary, Diebolder, Philipp, primary, Jethva, Prashant N., primary, Rhee, Aaron, primary, Song, Jong Hee, primary, Goo, Young Ah, primary, Li, Shunqian, primary, Nickels, Michael L., primary, Liu, Yongjian, primary, Rogers, Buck E., primary, Kapoor, Vaishali, primary, and Hallahan, Dennis E., primary

Published
2024
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10. Data from Development of a [89Zr]Zr-labeled Human Antibody using a Novel Phage-displayed Human scFv Library

Author

Singh, Abhay K., primary, Lewis, Calvin D., primary, Boas, Cristian A.W.V., primary, Diebolder, Philipp, primary, Jethva, Prashant N., primary, Rhee, Aaron, primary, Song, Jong Hee, primary, Goo, Young Ah, primary, Li, Shunqian, primary, Nickels, Michael L., primary, Liu, Yongjian, primary, Rogers, Buck E., primary, Kapoor, Vaishali, primary, and Hallahan, Dennis E., primary

Published
2024
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13. Essentially Critically Compressible Modules

Author

Singh, Abhay K.

Subjects
Mathematics - Commutative Algebra, Mathematics - Rings and Algebras, 16D50, 16D70, 16D80
Abstract

The main purpose of this paper is to introduce the concept of essentially critically compressible modules. We call an R-module M essentially critically compressible module if it is essentially compressible and additionally it cannot be imbedded in any of its factor module., Comment: 4 pages

Published
2012

14. Generalization of Strongly Clean Rings

Author

Singh, Abhay K.

Subjects
Mathematics - Rings and Algebras, Mathematics - Commutative Algebra, 16D70, 16P70, 16E50
Abstract

In this paper, strongly clean ring defined by W. K. Nicholson in 1999 has been generalized to n-strongly clean, {\Sigma}-strongly clean and with the help of example it has been shown that there exists a ring, which is n-strongly clean and {\Sigma}-strongly clean but not strongly clean. It has been shown that for a commutative ring R formal power series R[(x)] of R is n-strongly clean if and only if R is n- strongly clean. We also discussed the structure of homomorphic image of n- strongly clean and direct product of n- strongly clean rings. It has also been shown that for any commutative ring R, the polynomial ring R (x) is not {\Sigma}-strongly clean ring., Comment: 7 pages

Published
2012

15. Compressible Modules

Author

Singh, Abhay K.

Subjects
Mathematics - Commutative Algebra, Mathematics - Rings and Algebras, 16D50, 16D70, 16D80
Abstract

The main purpose of this paper is to study under what condition compressible modules are critically compressible. A sufficient condition for the injective hull of a critically compressible module to be critically compressible is also provided. Furthermore we prove sufficient conditions for a critically compressible module to be continuous. In addition, some characterization of critically compressible modules in terms of CS modules, nonsingular modules and cyclic modules are also provided

Published
2012

16. Development of a [89Zr]Zr-labeled Human Antibody using a Novel Phage-displayed Human scFv Library

Author

Singh, Abhay K., primary, Lewis, Calvin D., additional, Boas, Cristian A.W.V., additional, Diebolder, Philipp, additional, Jethva, Prashant N., additional, Rhee, Aaron, additional, Song, Jong Hee, additional, Goo, Young Ah, additional, Li, Shunqian, additional, Nickels, Michael L., additional, Liu, Yongjian, additional, Rogers, Buck E., additional, Kapoor, Vaishali, additional, and Hallahan, Dennis E., additional

Published
2024
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44. Plasma glial fibrillary acidic protein in autosomal dominant Alzheimer's disease: Associations with Aβ‐PET, neurodegeneration, and cognition.

Author

Chatterjee, Pratishtha, Vermunt, Lisa, Gordon, Brian A., Pedrini, Steve, Boonkamp, Lynn, Armstrong, Nicola J., Xiong, Chengjie, Singh, Abhay K., Li, Yan, Sohrabi, Hamid R., Taddei, Kevin, Molloy, Mark, Benzinger, Tammie L. S., Morris, John C., Karch, Celeste, Berman, Sarah, Chhatwal, Jasmeer, Cruchaga, Carlos, Graff‐Radford, Neill R., and Day, Gregory S

Abstract

Background: Glial fibrillary acidic protein (GFAP) is a promising candidate blood‐based biomarker for Alzheimer's disease (AD) diagnosis and prognostication. The timing of its disease‐associated changes, its clinical correlates, and biofluid‐type dependency will influence its clinical utility. Methods: We evaluated plasma, serum, and cerebrospinal fluid (CSF) GFAP in families with autosomal dominant AD (ADAD), leveraging the predictable age at symptom onset to determine changes by stage of disease. Results: Plasma GFAP elevations appear a decade before expected symptom onset, after amyloid beta (Aβ) accumulation and prior to neurodegeneration and cognitive decline. Plasma GFAP distinguished Aβ‐positive from Aβ‐negative ADAD participants and showed a stronger relationship with Aβ load in asymptomatic than symptomatic ADAD. Higher plasma GFAP was associated with the degree and rate of neurodegeneration and cognitive impairment. Serum GFAP showed similar relationships, but these were less pronounced for CSF GFAP. Conclusion: Our findings support a role for plasma GFAP as a clinical biomarker of Aβ‐related astrocyte reactivity that is associated with cognitive decline and neurodegeneration. Highlights: Plasma glial fibrillary acidic protein (GFAP) elevations appear a decade before expected symptom onset in autosomal dominant Alzheimer's disease (ADAD).Plasma GFAP was associated to amyloid positivity in asymptomatic ADAD.Plasma GFAP increased with clinical severity and predicted disease progression.Plasma and serum GFAP carried similar information in ADAD, while cerebrospinal fluid GFAP did not. [ABSTRACT FROM AUTHOR]

Published
2023
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50. Plasma glial fibrillary acidic protein in autosomal dominant Alzheimer's disease

Author

Chatterjee, Pratishtha, Vermunt, Lisa, Taddei, Kevin, Molloy, Mark, Benzinger, Tammie L S, Morris, John C, Karch, Celeste, Berman, Sarah, Chhatwal, Jasmeer, Cruchaga, Carlos, Graff-Radford, Neill R, Day, Gregory S, Gordon, Brian A, Farlow, Martin, Fox, Nick, Goate, Alison, Hassenstab, Jason, Lee, Jaehyun, Levin, Johannes, McDade, Eric, Mori, Hiroshi, Perrin, Richard, Sanchez-Valle, Raquel, Pedrini, Steve, Schofield, Peter R, Levey, Allan, Jucker, Mathias, Masters, Colin L, Fagan, Anne M, Bateman, Randall J, Martins, Ralph N, Teunissen, Charlotte, Network, and the Dominantly Inherited Alzheimer, Boonkamp, Lynn, Armstrong, Nicola J, Xiong, Chengjie, Singh, Abhay K, Li, Yan, Sohrabi, Hamid R, Laboratory Medicine, Amsterdam Neuroscience - Neurodegeneration, and Amsterdam Neuroscience - Neuroinfection & -inflammation

Subjects
Psychiatry and Mental health, Cellular and Molecular Neuroscience, Developmental Neuroscience, Epidemiology, Health Policy, Neurology (clinical), ddc:610, Geriatrics and Gerontology
Abstract

Background: Glial fibrillary acidic protein (GFAP) is a promising candidate blood-based biomarker for Alzheimer's disease (AD) diagnosis and prognostication. The timing of its disease-associated changes, its clinical correlates, and biofluid-type dependency will influence its clinical utility. Methods: We evaluated plasma, serum, and cerebrospinal fluid (CSF) GFAP in families with autosomal dominant AD (ADAD), leveraging the predictable age at symptom onset to determine changes by stage of disease. Results: Plasma GFAP elevations appear a decade before expected symptom onset, after amyloid beta (Aβ) accumulation and prior to neurodegeneration and cognitive decline. Plasma GFAP distinguished Aβ-positive from Aβ-negative ADAD participants and showed a stronger relationship with Aβ load in asymptomatic than symptomatic ADAD. Higher plasma GFAP was associated with the degree and rate of neurodegeneration and cognitive impairment. Serum GFAP showed similar relationships, but these were less pronounced for CSF GFAP. Conclusion: Our findings support a role for plasma GFAP as a clinical biomarker of Aβ-related astrocyte reactivity that is associated with cognitive decline and neurodegeneration. Highlights: Plasma glial fibrillary acidic protein (GFAP) elevations appear a decade before expected symptom onset in autosomal dominant Alzheimer's disease (ADAD). Plasma GFAP was associated to amyloid positivity in asymptomatic ADAD. Plasma GFAP increased with clinical severity and predicted disease progression. Plasma and serum GFAP carried similar information in ADAD, while cerebrospinal fluid GFAP did not.

Published
2022
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