Search

Your search keyword '"Sillén, U."' showing total 274 results
Start Over Author "Sillén, U."
274 results on '"Sillén, U."'

33. Management of Long-gap Esophagus: Experience with End-to-End Anastomosis Under Maximal Tension

Author

Hagberg, S., Rubenson, A., Sillén, U., Werkmäster, K., Hecker, W. Ch., editor, Stauffer, U. G., editor, Prévot, J., editor, Wurnig, P., editor, Spitz, L., editor, Rickham, P. P., editor, Bill, A. H., editor, Boix-Ochoa, J., editor, Ferguson, C. C., editor, Ghandi, R. K., editor, Gans, S. L., editor, Haller, J. A., editor, Kasai, M., editor, Knutrud, O., editor, Lister, J., editor, Myers, N. A., editor, Soave, F., editor, Angerpointner, T. A., editor, and Wurnig, Peter, editor

Published
1986
Full Text
View/download PDF

40. Lower urinary tract function in childhood; normal development and common functional disturbances

Author

Nevéus, Tryggve, Sillén, U, Nevéus, Tryggve, and Sillén, U

Abstract

This review aims to provide researchers and clinicians involved with the adult lower urinary tract with background knowledge regarding the early development of bladder function and its most common disturbances in childhood. Bladder development begins in weeks 4-6 and the detrusor muscle is formed during weeks 9-12 of gestation. Higher CNS centres are involved in micturition at birth, and the infant usually wakes up, at least briefly, to void. Voiding during the first years of life is often incomplete, owing to detrusor-sphincter dyscoordination, but this disappears when bladder control is attained. Approximately 5-10% of 7-year-old children suffer from daytime incontinence and/or nocturnal enuresis, and a few per cent of them will not outgrow it. Daytime incontinence in childhood is usually attributable to detrusor overactivity, although it is unclear to what extent it is the detrusor or the micturition reflex per se that is overactive. Enuresis - nocturnal incontinence - is caused by either nocturnal polyuria and/or nocturnal detrusor overactivity, in both cases combined with high arousal thresholds. Bladder problems in childhood constitute a risk factor for the development or persistence of bladder problems in adulthood.

Published
2013
Full Text
View/download PDF

41. Self-reported sleeping problems as a risk factor for subsequent muscular pain. Self-reported sleeping problems as a risk factor for subsequent muscular pain. Results from a one-year follow-up of the Malmö Shoulder Neck Study cohort:Results from a one-year follow-up of the Malmö Shoulder Neck Study cohort

Author

Canivet, C, Östergren, PO, Choi, B, Nilsson, P, Sillén, U, Moghadassi, M, Karasek, Robert Allen, Isacsson, SO, Canivet, C, Östergren, PO, Choi, B, Nilsson, P, Sillén, U, Moghadassi, M, Karasek, Robert Allen, and Isacsson, SO

Published
2008

43. Enuresis, sleep and desmopressin treatment.

Author

Nevéus, T., Bader, G., Sillén, U., Nevéus, T, and Sillén, U

Subjects
DESMOPRESSIN, TREATMENT of enuresis, JUVENILE diseases, POLYSOMNOGRAPHY, COMPARATIVE studies, DRUGS, ENURESIS, KIDNEY diseases, RESEARCH methodology, MEDICAL cooperation, RESEARCH, SLEEP, URINATION, EVALUATION research, TREATMENT effectiveness, PHARMACODYNAMICS, THERAPEUTICS
Abstract

Aim: To detect effects of desmopressin on sleep in enuretic children and to look for polysomnographical differences between responders and non-responders to desmopressin treatment.Methods: Twenty-one children with primary nocturnal enuresis were examined polysomnographically before treatment. All but one of the children then received treatment with desmopressin in standard dosage, and the response was documented. Seven of the children underwent a second polysomnographic registration while on treatment.Results: The time interval (+/- 1 SD) between sleep onset and the enuretic episode was 92 +/- 67 min without medication and 372 +/- 157 min when desmopressin was given (p = 0.003). Standard polysomnographic variables were not affected by the drug. Ten children were desmopressin responders and 10 were non-responders. The total sleep time was 455 +/- 56 min in the former and 408 +/- 31 min in the latter group (p = 0.04). The responders spent 27.4 +/- 5.5% of their total sleep time in rapid eye movement sleep, compared with 18.2 +/- 6.5% in the non-responder group (p = 0.004).Conclusion: Desmopressin has no major effects on sleep as such but does delay bladder emptying. Enuretic children responding to desmopressin treatment have more rapid eye movement sleep than therapy-resistant children. [ABSTRACT FROM AUTHOR]

Published
2002
Full Text
View/download PDF

Catalog

Books, media, physical & digital resources
See catalog results