18 results on '"Sides, Mark"'
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2. Neoadjuvant Radiation with Concurrent 5-FU Resulting in Complete Pathologic Response in Stage IIIB Squamous Cell Carcinoma of the Urethra
3. The Sarbanes-Oxley Act and fiduciary duties.
4. Overexpression of Sulf2 in idiopathic pulmonary fibrosis
5. The Epstein-Barr Virus Latent Membrane Protein 1 and Transforming Growth Factor-β1 Synergistically Induce Epithelial-Mesenchymal Transition in Lung Epithelial Cells
6. 5-FLUOROURACIL INDUCED CORONARY VASOSPASM AND NON-ISCHEMIC CARDIOMYOPATHY PRESENTING IN THE SAME PATIENT
7. Epstein - Barr virus latent membrane protein 1 suppresses reporter activity through modulation of promyelocytic leukemia protein-nuclear bodies
8. Arsenic trioxide inhibits EBV reactivation and promotes cell death in EBV-positive lymphoma cells
9. Arsenic trioxide inhibits EBV reactivation and promotes cell death in EBV-positive lymphoma cells.
10. Arsenic trioxide inhibits transforming growth factor-β1-induced fibroblast to myofibroblast differentiation in vitro and bleomycin induced lung fibrosis in vivo
11. Co-treatment with arsenic trioxide and ganciclovir reduces tumor volume in a murine xenograft model of nasopharyngeal carcinoma
12. Arsenic Trioxide Abrogates Transforming Growth Factor-beta1 Induced Fibroblast To Myofibroblast Differentiation
13. Epstein - Barr virus latent membrane protein 1 suppresses reporter activity through modulation of promyelocytic leukemia protein-nuclear bodies
14. Arsenic mediated disruption of promyelocytic leukemia protein nuclear bodies induces ganciclovir susceptibility in Epstein–Barr positive epithelial cells
15. Arsenic Trioxide Abrogates Transforming Growth Factor-Beta1 Induced Fibroblast To Myofibroblast Differentiation
16. Transforming Growth Factor-²1 Induces Sulf2 Expression In Type II Alveolar Epithelial Cells
17. The Epstein ‐ Barr virus Latent Membrane Protein 1 and Transforming Growth Factor beta1 synergistically induce Epithelial‐to‐Mesenchymal Transition in human alveolar epithelial cells
18. Arsenic trioxide inhibits transforming growth factor-β1-induced fibroblast to myofibroblast differentiation in vitro and bleomycin induced lung fibrosis in vivo.
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