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3. Spatial and single-cell colocalisation analysis reveals MDK-mediated immunosuppressive environment with regulatory T cells in colorectal carcinogenesis

4. Spatial and single-cell transcriptomics decipher the cellular environment containing HLA-G+ cancer cells and SPP1+ macrophages in colorectal cancer

14. GIMLET: Identifying Biological Modulators in Context-Specific Gene Regulation Using Local Energy Statistics

15. Tumor Subclonal Progression Model for Cancer Hallmark Acquisition

21. FXYD3 functionally demarcates an ancestral breast cancer stem cell subpopulation with features of drug-tolerant persisters

23. Hypoxia activates SREBP2 through Golgi disassembly in bone marrow‐derived monocytes for enhanced tumor growth

24. Acidic extracellular pH drives accumulation of N1-acetylspermidine and recruitment of protumor neutrophils

26. ARHGAP10, which encodes Rho GTPase-activating protein 10, is a novel gene for schizophrenia risk

27. Integrated multiomics analysis of hepatoblastoma unravels its heterogeneity and provides novel druggable targets

35. ATR inhibition controls aggressive prostate tumors deficient in Y-linked histone demethylase KDM5D

36. Convergent genomic diversity and novel BCAA metabolism in intrahepatic cholangiocarcinoma

37. The list of PRC2 target genes and the result of microarray analysis from PRC2-Mediated Transcriptomic Alterations at the Embryonic Stage Govern Tumorigenesis and Clinical Outcome in MYCN-Driven Neuroblastoma

39. Supplementary Materials and Methods from Pathogenic Epigenetic Consequences of Genetic Alterations in IDH-Wild-Type Diffuse Astrocytic Gliomas

41. Supplementary Figures from Integrated Molecular Characterization of the Lethal Pediatric Cancer Pancreatoblastoma

42. Supplementary Tables from Integrated Molecular Characterization of the Lethal Pediatric Cancer Pancreatoblastoma

43. Supplementary Figure Legends from Pathogenic Epigenetic Consequences of Genetic Alterations in IDH-Wild-Type Diffuse Astrocytic Gliomas

44. Supplementary Tables from Pathogenic Epigenetic Consequences of Genetic Alterations in IDH-Wild-Type Diffuse Astrocytic Gliomas

45. Data from ER Stress Signaling Promotes the Survival of Cancer “Persister Cells” Tolerant to EGFR Tyrosine Kinase Inhibitors

47. Supplementary Methods from ER Stress Signaling Promotes the Survival of Cancer “Persister Cells” Tolerant to EGFR Tyrosine Kinase Inhibitors

48. Supplementary Table S1 from ER Stress Signaling Promotes the Survival of Cancer “Persister Cells” Tolerant to EGFR Tyrosine Kinase Inhibitors

49. Data from Pathogenic Epigenetic Consequences of Genetic Alterations in IDH-Wild-Type Diffuse Astrocytic Gliomas

50. Supplementary Data from Meflin-Positive Cancer-Associated Fibroblasts Inhibit Pancreatic Carcinogenesis

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