40 results on '"Shemshedini, Lirim"'
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2. MDM2 regulates the stability of AR, AR-V7, and TM4SF3 proteins in prostate cancer
3. Dual-Acting Peptides Target EZH2 and AR: A New Paradigm for Effective Treatment of Castration-Resistant Prostate Cancer.
4. The Transmembrane Protein TM4SF3 Interacts with AR and AR-V7 and is Recruited to AR Target Genes
5. Peptides disrupting TM4SF3 interaction with AR or AR-V7 block prostate cancer cell proliferation
6. Dual-Acting Peptides Target EZH2 and AR: A New Paradigm for Effective Treatment of Castration-Resistant Prostate Cancer
7. Dual-Acting Peptides Target EZH2 and AR: A New Paradigm for Effective Treatment of Castration-Resistant Prostate Cancer.
8. TM4SF3 and AR: A Nuclear Complex that Stabilizes Both Proteins
9. Resistance to Juvenile Hormone and an Insect Growth Regulator in Drosophila is Associated with an Altered Cytosolic Juvenile Hormone-Binding Protein
10. COP9 Subunits 4 and 5 Target Soluble Guanylyl Cyclase α1 and p53 in Prostate Cancer Cells
11. Makorin RING finger protein 1 (MKRN1) has negative and positive effects on RNA polymerase II-dependent transcription
12. c-Jun targets amino terminus of androgen receptor in regulating androgen-responsive transcription
13. Use of Reporter Genes to Study Promoters of the Androgen Receptor
14. Soluble Guanylyl Cyclase α1 and p53 Cytoplasmic Sequestration and Down-Regulation in Prostate Cancer
15. Ligand-free RAR can interact with the RNA polymerase II subunit hsRPB7 and repress transcription
16. Abstract 2072: TM4SF3 interaction with AR or AR-V7 is important for the survival of prostate cancer cells
17. Androgen up-regulation of Twist1 gene expression is mediated by ETV1
18. c-Fos dimerization with c-Jun represses c-Jun enhancement of androgen receptor transactivation
19. Identification of domains of c-Jun mediating androgen receptor transactivation
20. ETV1 Is a Novel Androgen Receptor-Regulated Gene that Mediates Prostate Cancer Cell Invasion
21. Ligand-Activated Retinoic Acid Receptor Inhibits AP-1 Transactivation by Disruptingc-Jun/c-Fos Dimerization
22. Peptide B targets soluble guanylyl cyclase α1 and kills prostate cancer cells
23. RNase L Suppresses Androgen Receptor Signaling, Cell Migration and Matrix Metalloproteinase Activity in Prostate Cancer Cells
24. Zinc Finger 280B Regulates sGCα1 and p53 in Prostate Cancer Cells
25. A Peptide against Soluble Guanylyl Cyclase α1: A New Approach to Treating Prostate Cancer
26. Abstract 215: Soluble guanylyl cyclase α1 and p53 cytoplasmic sequestration: A novel mechanism for p53 down-regulation in prostate cancer
27. Expression of a hyperactive androgen receptor leads to androgen-independent growth of prostate cancer cells
28. c-Jun Has Multiple Enhancing Activities in the Novel Cross Talk between the Androgen Receptor and Ets Variant Gene 1 in Prostate Cancer
29. SUMO-3 Enhances Androgen Receptor Transcriptional Activity through a Sumoylation-independent Mechanism in Prostate Cancer Cells
30. Glycogen Synthase Kinase-3β Is Involved in the Phosphorylation and Suppression of Androgen Receptor Activity
31. c-Jun Potentiates the Functional Interaction between the Amino and Carboxyl Termini of the Androgen Receptor
32. p53 Represses Androgen-induced Transactivation of Prostate-specific Antigen by Disrupting hAR Amino- to Carboxyl-terminal Interaction
33. Ligand-Activated Retinoic Acid Receptor Inhibits AP-1 Transactivation by Disrupting c-Jun/c-Fos Dimerization
34. c-Jun Can Mediate Androgen Receptor-induced Transactivation
35. pLEF, a novel vector for expression of glutathione S-transferase fusion proteins in mammalian cells
36. Juvenile hormone binding proteins in larval fat body nuclei of Drosophila melanogaster
37. A high affinity, high molecular weight juvenile hormone binding protein in the hemolymph of Drosophila melanogaster
38. SUMO-3 Enhances Androgen Receptor Transcriptional Activity through a Sumoylation-independent Mechanism in Prostate Cancer Cells.
39. Use of reporter genes to study promoters of the androgen receptor.
40. Glycogen synthase kinase-3 beta is involved in the phosphorylation and suppression of androgen receptor activity.
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