185 results on '"Shelkovnikova, Tatyana"'
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2. C9orf72 poly-PR forms anisotropic condensates causative of nuclear TDP-43 pathology
3. Stress-induced TDP-43 nuclear condensation causes splicing loss of function and STMN2 depletion
4. The master energy homeostasis regulator PGC-1α exhibits an mRNA nuclear export function
5. The Batten disease protein CLN3 is important for stress granules dynamics and translational activity
6. Deep learning modeling of rare noncoding genetic variants in human motor neurons definesCCDC146as a therapeutic target for ALS
7. C9orf72 poly-PR condensation induces nuclear TDP-43 pathology and is inhibited by RNA in an optogenetic cell model
8. TDP-43 in nuclear condensates: where, how, and why.
9. TDP-43 is a Master Regulator of Paraspeckle Condensation
10. Toxic gain of function from mutant FUS protein is crucial to trigger cell autonomous motor neuron loss
11. Long non-coding RNA Neat1 regulates adaptive behavioural response to stress in mice
12. Frameshift peptides alter the properties of truncated FUS proteins in ALS-FUS
13. ALS-linked FUS mutations confer loss and gain of function in the nucleus by promoting excessive formation of dysfunctional paraspeckles
14. Model of Artificial Neural Network of Biological Type and Simulation of the Network Operation
15. Assessment of radiological techniques application possibility for non-invasive diagnostics of latent inflammatory processes in myocardium in patients with atrial fibrillation
16. Oligonucleotide-Recognizing Topoisomerase Inhibitors (OTIs): Precision Gene Editors for Neurodegenerative Diseases?
17. A toolkit for the identification of NEAT1_2/paraspeckle modulators
18. Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis
19. AtypicalTDP‐43 protein expression in anALSpedigree carrying a p.Y374Xtruncation mutation inTARDBP
20. MRI POTENTIAL FOR PREDICTION OF THE ANTIHYPERTENSIVE EFFECT OF RFA IN PATIENTS WITH TREATMENT-RESISTANT HYPERTENSION
21. MRI IN THE ASSESSMENT OF CEREBRAL INJURY AND CEREBROPROTECTIVE EFFECTS OF RENAL DENERVATION IN RESISTANT ARTERIAL HYPERTENSION
22. NEPHROPROTECTIVE EFFECT OF RD ACCORDING TO 5-YEAR FOLLOW-UP
23. ALS-linked cytoplasmic FUS assemblies are compositionally different from physiological stress granules and sequester hnRNPA3, a novel modifier of FUS toxicity
24. Processing and Recognition of Two-Coordinate Reference Images in a Scanning Probe Microscopy
25. Atypical TDP‐43 protein expression in an ALS pedigree carrying a p.Y374X truncation mutation in TARDBP.
26. Connecting the “dots”: RNP granule network in health and disease
27. Dimebon Slows Progression of Proteinopathy in γ-Synuclein Transgenic Mice
28. Multistep process of FUS aggregation in the cell cytoplasm involves RNA-dependent and RNA-independent mechanisms
29. Compromised paraspeckle formation as a pathogenic factor in FUSopathies
30. Compositional analysis of ALS-linked stress granule-like structures reveals factors and cellular pathways dysregulated by mutant FUS under stress
31. Additional file 1 of Frameshift peptides alter the properties of truncated FUS proteins in ALS-FUS
32. Long non-coding RNA NEAT1_1 ameliorates TDP-43 toxicity in in vivo models of TDP-43 proteinopathy
33. Stress granules regulate paraspeckles: RNP granule continuum at work
34. Antiviral Immune Response as a Trigger of FUS Proteinopathy in Amyotrophic Lateral Sclerosis
35. Additional file 1: of ALS-linked FUS mutations confer loss and gain of function in the nucleus by promoting excessive formation of dysfunctional paraspeckles
36. Additional file 3: of Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis
37. Additional file 6: of Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis
38. Additional file 4: of Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis
39. Additional file 1: of Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis
40. Additional file 5: of Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis
41. Additional file 2: of Protective paraspeckle hyper-assembly downstream of TDP-43 loss of function in amyotrophic lateral sclerosis
42. Stress granules regulate stress-induced paraspeckle assembly
43. Long non-coding RNA Neat1 regulates adaptive behavioural response to stress in mice
44. Calcium-responsive transactivator (CREST) toxicity is rescued by loss of PBP1/ATXN2 function in a novel yeast proteinopathy model and in transgenic flies
45. Antiviral Immune Response as a Trigger of FUS Proteinopathy in Amyotrophic Lateral Sclerosis
46. NEAT1 and paraspeckles in neurodegenerative diseases: A missing lnc found?
47. Chronically stressed or stress-preconditioned neurons fail to maintain stress granule assembly
48. Magnetic resonance imaging syndromes in patients with suspected myocarditis and theyr' relations to histological data
49. Calcium-responsive transactivator (CREST) toxicity is rescued by loss of PBP1/ATXN2 function in a novel yeast proteinopathy model and in transgenic flies
50. Modulation of p-eIF2α cellular levels and stress granule assembly/disassembly by trehalose
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