28 results on '"Sheldon, Kely L."'
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2. Physiologically based pharmacokinetic modelling to predict intragastric rifabutin concentrations in the treatment of Helicobacter pylori infection
3. Tubulin Tail Sequences and Post-translational Modifications Regulate Closure of Mitochondrial Voltage-dependent Anion Channel (VDAC)
4. S1597 Modeling Gastric Luminal Rifabutin Concentrations: RHB-105 (Rifabutin 50 mg Q8H) Provides More Favorable Exposure for Helicobacter pylori Eradication Than Generic Rifabutin 150 mg BID or 300 mg QD
5. Voltage-dependent Anion Channels Modulate Mitochondrial Metabolism in Cancer Cells: REGULATION BY FREE TUBULIN AND ERASTIN
6. Tubulin Binding Blocks Mitochondrial Voltage-Dependent Anion Channel and Regulates Respiration
7. Tu1078: LOW-DOSE RIFABUTIN TRIPLE THERAPY (RHB-105) MAINTAINS HIGH HELICOBACTER PYLORI ERADICATION RATES AND SHOWS FAVORABLE SAFETY AND EFFICACY IN SUBJECTS WITH DIABETES MELLITUS
8. Tu1077: PHYSIOLOGICALLY-BASED PHARMACOKINETIC MODEL TO ESTIMATE INTRAGASTRIC RIFABUTIN CONCENTRATIONS FOLLOWING RIFABUTIN 50 MG Q8H (RHB-105) OR 150 MG QD: MORE FAVORABLE EXPOSURE FOR HELICOBACTER PYLORI ERADICATON WITH Q8H DOSING
9. Role of Subplasmalemmal Mitochondria in Angiotensin II–Mediated Contraction
10. Pitfalls of Physician-Directed Treatment of Helicobacter pylori: Results from Two Phase 3 Clinical Trials and Real-World Prescribing Data
11. Helicobacter pylori eradication by low‐dose rifabutin triple therapy (RHB‐105) is unaffected by high body mass index
12. S1417 RHB-105 Q8H Rifabutin Dosing Provides Favorable Exposure for Helicobacter pylori Eradication
13. S1451 Pitfalls of Physician-Directed Treatment of H. pylori Infection: Results From Two Phase 3 Clinical Trials and Real-World Prescribing Data
14. Sa1381 PHYSIOLOGICALLY-BASED PHARMACOKINETIC MODELING CONFIRMS RIFABUTIN, AMOXICILLIN, AND OMEPRAZOLE BIOEQUIVALENCE BETWEEN Q8H AND TID DOSING FOR LOW-DOSE RIFABUTIN TRIPLE THERAPY (RHB-105) FOR THE ERADICATION OF HELICOBACTER PYLORI IN ADULT PATIENTS
15. S1342 Efficacy of H. pylori Eradication by Low-Dose Rifabutin Triple Therapy (RHB-105) Is Unaffected by BMI: Post-hoc Analysis From Two Phase 3 Trials
16. Abstract 3044: The ability of tubulin to close mitochondrial VDAC pores depends on beta tubulin isotype
17. Antagonists of tubulin–VDAC interaction induce oxidative stress and mitochondrial dysfunction
18. Antagonists of the Inhibitory Effect of Free Tubulin on VDAC Induce Oxidative Stress and Mitochondrial Dysfunction
19. Current Insights into the Molecular Mechanisms of VDAC-Tubulin Interaction
20. Novel Mechanism of Mitochondrial Respiration Control through Competition between Hexokinase-2 and Tubulin for VDAC Binding
21. VDAC Phosphorylation in Control of Mitochondrial Respiration
22. VDAC Regulation by Tubulin and its Physiological Implications
23. Voltage Dependent Anion Channel-3 (VDAC3) is the Major Isoform Contributing to Mitochondrial Metabolism in HepG2 Cells and is Regulated by Free Tubulin and Erastin
24. Phosphorylation of Voltage-Dependent Anion Channel by Serine/Threonine Kinases Governs Its Interaction with Tubulin
25. Membrane Lipid Composition Regulates Tubulin-VDAC Interaction
26. VDAC Phosphorylation Regulates Interaction with Tubulin
27. Governing Respiration: Tubulin's C-Terminus Interaction with VDAC
28. Physiologically Based Pharmacokinetic Modeling and Simulation to Support a Change in the FDA‐Labeled Dosing Frequency of RHB‐105 Low‐Dose Rifabutin Triple Therapy for Helicobacter pylori Eradication.
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